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Crosstalk between Hypoxia and Extracellular Matrix in the Tumor Microenvironment in Breast Cancer

Even though breast cancer is the most diagnosed cancer among women, treatments are not always successful in preventing its progression. Recent studies suggest that hypoxia and the extracellular matrix (ECM) are important in altering cell metabolism and tumor metastasis. Therefore, the aim of this re...

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Autores principales: Dekker, Yasmin, Le Dévédec, Sylvia E., Danen, Erik H. J., Liu, Qiuyu
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2022
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9498429/
https://www.ncbi.nlm.nih.gov/pubmed/36140753
http://dx.doi.org/10.3390/genes13091585
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author Dekker, Yasmin
Le Dévédec, Sylvia E.
Danen, Erik H. J.
Liu, Qiuyu
author_facet Dekker, Yasmin
Le Dévédec, Sylvia E.
Danen, Erik H. J.
Liu, Qiuyu
author_sort Dekker, Yasmin
collection PubMed
description Even though breast cancer is the most diagnosed cancer among women, treatments are not always successful in preventing its progression. Recent studies suggest that hypoxia and the extracellular matrix (ECM) are important in altering cell metabolism and tumor metastasis. Therefore, the aim of this review is to study the crosstalk between hypoxia and the ECM and to assess their impact on breast cancer progression. The findings indicate that hypoxic signaling engages multiple mechanisms that directly contribute to ECM remodeling, ultimately increasing breast cancer aggressiveness. Second, hypoxia and the ECM cooperate to alter different aspects of cell metabolism. They mutually enhance aerobic glycolysis through upregulation of glucose transport, glycolytic enzymes, and by regulating intracellular pH. Both alter lipid and amino acid metabolism by stimulating lipid and amino acid uptake and synthesis, thereby providing the tumor with additional energy for growth and metastasis. Third, YAP/TAZ signaling is not merely regulated by the tumor microenvironment and cell metabolism, but it also regulates it primarily through its target c-Myc. Taken together, this review provides a better understanding of the crosstalk between hypoxia and the ECM in breast cancer. Additionally, it points to a role for the YAP/TAZ mechanotransduction pathway as an important link between hypoxia and the ECM in the tumor microenvironment, driving breast cancer progression.
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spelling pubmed-94984292022-09-23 Crosstalk between Hypoxia and Extracellular Matrix in the Tumor Microenvironment in Breast Cancer Dekker, Yasmin Le Dévédec, Sylvia E. Danen, Erik H. J. Liu, Qiuyu Genes (Basel) Review Even though breast cancer is the most diagnosed cancer among women, treatments are not always successful in preventing its progression. Recent studies suggest that hypoxia and the extracellular matrix (ECM) are important in altering cell metabolism and tumor metastasis. Therefore, the aim of this review is to study the crosstalk between hypoxia and the ECM and to assess their impact on breast cancer progression. The findings indicate that hypoxic signaling engages multiple mechanisms that directly contribute to ECM remodeling, ultimately increasing breast cancer aggressiveness. Second, hypoxia and the ECM cooperate to alter different aspects of cell metabolism. They mutually enhance aerobic glycolysis through upregulation of glucose transport, glycolytic enzymes, and by regulating intracellular pH. Both alter lipid and amino acid metabolism by stimulating lipid and amino acid uptake and synthesis, thereby providing the tumor with additional energy for growth and metastasis. Third, YAP/TAZ signaling is not merely regulated by the tumor microenvironment and cell metabolism, but it also regulates it primarily through its target c-Myc. Taken together, this review provides a better understanding of the crosstalk between hypoxia and the ECM in breast cancer. Additionally, it points to a role for the YAP/TAZ mechanotransduction pathway as an important link between hypoxia and the ECM in the tumor microenvironment, driving breast cancer progression. MDPI 2022-09-03 /pmc/articles/PMC9498429/ /pubmed/36140753 http://dx.doi.org/10.3390/genes13091585 Text en © 2022 by the authors. https://creativecommons.org/licenses/by/4.0/Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).
spellingShingle Review
Dekker, Yasmin
Le Dévédec, Sylvia E.
Danen, Erik H. J.
Liu, Qiuyu
Crosstalk between Hypoxia and Extracellular Matrix in the Tumor Microenvironment in Breast Cancer
title Crosstalk between Hypoxia and Extracellular Matrix in the Tumor Microenvironment in Breast Cancer
title_full Crosstalk between Hypoxia and Extracellular Matrix in the Tumor Microenvironment in Breast Cancer
title_fullStr Crosstalk between Hypoxia and Extracellular Matrix in the Tumor Microenvironment in Breast Cancer
title_full_unstemmed Crosstalk between Hypoxia and Extracellular Matrix in the Tumor Microenvironment in Breast Cancer
title_short Crosstalk between Hypoxia and Extracellular Matrix in the Tumor Microenvironment in Breast Cancer
title_sort crosstalk between hypoxia and extracellular matrix in the tumor microenvironment in breast cancer
topic Review
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9498429/
https://www.ncbi.nlm.nih.gov/pubmed/36140753
http://dx.doi.org/10.3390/genes13091585
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