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Hepatitis B virus promotes hepatocellular carcinoma development by activating GP73 to repress the innate immune response

BACKGROUND: Hepatitis B virus (HBV) causes acute and chronic infection in the clinic. Hepatocellular carcinoma (HCC) is closely linked to HBV infection. Serum Golgi protein 73 (GP73) increases during HBV infection. However, the role of GP73 during HBV infection and the occurrence of HBV-related HCC...

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Detalles Bibliográficos
Autores principales: Liu, Long, Huang, Yanping, Fu, Yanan, Rao, Jingjing, Zeng, Feng, Ji, Manshan, Xu, Xiang, Zhu, Jianyong, Du, Weixing, Liu, Zhixin
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2022
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9533540/
https://www.ncbi.nlm.nih.gov/pubmed/36195933
http://dx.doi.org/10.1186/s13027-022-00462-y

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