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Hobit and Blimp‐1 regulate T(RM) abundance after LCMV infection by suppressing tissue exit pathways of T(RM)precursors
Tissue‐resident memory T cells (Trm) are retained in peripheral tissues after infection for enhanced protection against secondary encounter with the same pathogen. We have previously shown that the transcription factor Hobit and its homolog Blimp‐1 drive Trm development after viral infection, but ho...
Autores principales: | Parga‐Vidal, Loreto, Taggenbrock, Renske L.R.E., Beumer‐Chuwonpad, Ammarina, Aglmous, Hajar, Kragten, Natasja A.M., Behr, Felix M., Bovens, Astrid A., van Lier, Rene A.W., Stark, Regina, van Gisbergen, Klaas P.J.M. |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
John Wiley and Sons Inc.
2022
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9545210/ https://www.ncbi.nlm.nih.gov/pubmed/35389518 http://dx.doi.org/10.1002/eji.202149665 |
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