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The heart and SARS-CoV-2
SARS-Cov2 is currently causing a persistent Covid-19 pandemic, which poses a risk of causing long-term cardiovascular sequels in the population. The viral mechanism of cell infection through the angiotensin 2 converter enzyme receptor and the limited antiviral innate immune response are the suspecte...
Autores principales: | , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Elsevier España, S.L.U.
2022
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9562083/ https://www.ncbi.nlm.nih.gov/pubmed/36268184 http://dx.doi.org/10.1016/j.medcle.2022.10.001 |
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author | González-Calle, David Eiros, Rocío Sánchez, Pedro L. |
author_facet | González-Calle, David Eiros, Rocío Sánchez, Pedro L. |
author_sort | González-Calle, David |
collection | PubMed |
description | SARS-Cov2 is currently causing a persistent Covid-19 pandemic, which poses a risk of causing long-term cardiovascular sequels in the population. The viral mechanism of cell infection through the angiotensin 2 converter enzyme receptor and the limited antiviral innate immune response are the suspected causes for a more frequent cardiovascular damage in SARS-Cov2 infection. Knowledge of: the appearance during acute infection of other cardiac conditions beyond the classical myocarditis and pericarditis), the long-term cardiac manifestations (persistent Covid-19), and the increased incidence of myocarditis and pericarditis after vaccination; it is of special interest in order to offer our patients best practices based on current scientific evidence. |
format | Online Article Text |
id | pubmed-9562083 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2022 |
publisher | Elsevier España, S.L.U. |
record_format | MEDLINE/PubMed |
spelling | pubmed-95620832022-10-16 The heart and SARS-CoV-2 González-Calle, David Eiros, Rocío Sánchez, Pedro L. Med Clin (Engl Ed) Review SARS-Cov2 is currently causing a persistent Covid-19 pandemic, which poses a risk of causing long-term cardiovascular sequels in the population. The viral mechanism of cell infection through the angiotensin 2 converter enzyme receptor and the limited antiviral innate immune response are the suspected causes for a more frequent cardiovascular damage in SARS-Cov2 infection. Knowledge of: the appearance during acute infection of other cardiac conditions beyond the classical myocarditis and pericarditis), the long-term cardiac manifestations (persistent Covid-19), and the increased incidence of myocarditis and pericarditis after vaccination; it is of special interest in order to offer our patients best practices based on current scientific evidence. Elsevier España, S.L.U. 2022-11-11 2022-10-14 /pmc/articles/PMC9562083/ /pubmed/36268184 http://dx.doi.org/10.1016/j.medcle.2022.10.001 Text en © 2022 Elsevier España, S.L.U. All rights reserved. Since January 2020 Elsevier has created a COVID-19 resource centre with free information in English and Mandarin on the novel coronavirus COVID-19. The COVID-19 resource centre is hosted on Elsevier Connect, the company's public news and information website. Elsevier hereby grants permission to make all its COVID-19-related research that is available on the COVID-19 resource centre - including this research content - immediately available in PubMed Central and other publicly funded repositories, such as the WHO COVID database with rights for unrestricted research re-use and analyses in any form or by any means with acknowledgement of the original source. These permissions are granted for free by Elsevier for as long as the COVID-19 resource centre remains active. |
spellingShingle | Review González-Calle, David Eiros, Rocío Sánchez, Pedro L. The heart and SARS-CoV-2 |
title | The heart and SARS-CoV-2 |
title_full | The heart and SARS-CoV-2 |
title_fullStr | The heart and SARS-CoV-2 |
title_full_unstemmed | The heart and SARS-CoV-2 |
title_short | The heart and SARS-CoV-2 |
title_sort | heart and sars-cov-2 |
topic | Review |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9562083/ https://www.ncbi.nlm.nih.gov/pubmed/36268184 http://dx.doi.org/10.1016/j.medcle.2022.10.001 |
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