Cargando…
Author Correction: Ectopic JAK–STAT activation enables the transition to a stem-like and multilineage state conferring AR-targeted therapy resistance
Autores principales: | Deng, Su, Wang, Choushi, Wang, Yunguan, Xu, Yaru, Li, Xiaoling, Johnson, Nickolas A., Mukherji, Atreyi, Lo, U-Ging, Xu, Lingfan, Gonzalez, Julisa, Metang, Lauren A., Ye, Jianfeng, Tirado, Carla Rodriguez, Rodarte, Kathia, Zhou, Yinglu, Xie, Zhiqun, Arana, Carlos, Annamalai, Valli, Liu, Xihui, Vander Griend, Donald J., Strand, Douglas, Hsieh, Jer-Tsong, Li, Bo, Raj, Ganesh, Wang, Tao, Mu, Ping |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group US
2022
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9586863/ https://www.ncbi.nlm.nih.gov/pubmed/36241729 http://dx.doi.org/10.1038/s43018-022-00458-y |
Ejemplares similares
-
Ectopic JAK–STAT activation enables the transition to a stem-like and multilineage state conferring AR-targeted therapy resistance
por: Deng, Su, et al.
Publicado: (2022) -
Loss of SYNCRIP unleashes APOBEC-driven mutagenesis, tumor heterogeneity, and AR-targeted therapy resistance in prostate cancer
por: Li, Xiaoling, et al.
Publicado: (2023) -
The role of microRNAs in prostate cancer progression
por: Lo, U-Ging, et al.
Publicado: (2013) -
The evolving landscape of prostate cancer stem cell: Therapeutic implications and future challenges
por: Yun, Eun-Jin, et al.
Publicado: (2016) -
The regulatory pathways leading to stem-like cells underlie prostate cancer progression
por: Lin, Chun-Jung, et al.
Publicado: (2019)