Cargando…

The synergy of β amyloid 1-42 and oxidative stress in the development of Alzheimer’s disease-like neurodegeneration of hippocampal cells

Alzheimer’s disease (AD) is a type of dementia that affects memory, thinking and behavior. Symptoms eventually become severe enough to interfere with daily tasks. Understanding the etiology and pathogenesis of AD is necessary for the development of strategies for AD prevention and/or treatment, and...

Descripción completa

Detalles Bibliográficos
Autores principales:	Karapetyan, Gohar, Fereshetyan, Katarine, Harutyunyan, Hayk, Yenkoyan, Konstantin
Formato:	Online Artículo Texto
Lenguaje:	English
Publicado:	Nature Publishing Group UK 2022
Materias:	Article
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9596457/ https://www.ncbi.nlm.nih.gov/pubmed/36284177 http://dx.doi.org/10.1038/s41598-022-22761-5

Ejemplares similares

Author Correction: The synergy of β amyloid 1-42 and oxidative stress in the development of Alzheimer’s disease-like neurodegeneration of hippocampal cells
por: Karapetyan, Gohar, et al.
Publicado: (2022)

The Involvement of Insulin-Like Growth Factor 1 and Nerve Growth Factor in Alzheimer’s Disease-Like Pathology and Survival Role of the Mix of Embryonic Proteoglycans: Electrophysiological Fingerprint, Structural Changes and Regulatory Effects on Neurotrophins
por: Aghajanov, Michail, et al.
Publicado: (2021)

Assessment of behavioral, morphological and electrophysiological changes in prenatal and postnatal valproate induced rat models of autism spectrum disorder
por: Fereshetyan, Katarine, et al.
Publicado: (2021)

Effects of β-amyloid (1-42) Administration on the Main Neurogenic Niches of the Adult Brain: Amyloid-Induced Neurodegeneration Influences Neurogenesis
por: Yenkoyan, Konstantin, et al.
Publicado: (2022)

Disruption of Cholinergic Circuits as an Area for Targeted Drug Treatment of Alzheimer’s Disease: In Vivo Assessment of Short-Term Plasticity in Rat Brain
por: Chavushyan, Vergine, et al.
Publicado: (2020)

Novel Probable Glance at Inflammatory Scenario Development in Autistic Pathology
por: Harutyunyan, Aida A., et al.
Publicado: (2021)

Rates of β-amyloid accumulation are independent of hippocampal neurodegeneration
por: Jack, Clifford R., et al.
Publicado: (2014)

Tyramine and Amyloid Beta 42: A Toxic Synergy
por: Dhakal, Sudip, et al.
Publicado: (2020)

A critical role for the self-assembly of Amyloid-β1-42 in neurodegeneration
por: Marshall, Karen E., et al.
Publicado: (2016)

Alpha2-Adrenoblockers Regulate Development of Oxidative Stress and Cognitive Behaviour of Rats under Chronic Acoustic Stress Conditions
por: Melkonyan, Magdalina, et al.
Publicado: (2021)

Amyloid-β(42) stimulated hippocampal lactate release is coupled to glutamate uptake
por: Hascup, Erin R., et al.
Publicado: (2022)

Blood amyloid-β oligomerization associated with neurodegeneration of Alzheimer’s disease
por: Youn, Young Chul, et al.
Publicado: (2019)

Novel Neuroprotective Function of Apical-Basal Polarity Gene Crumbs in Amyloid Beta 42 (Aβ42) Mediated Neurodegeneration
por: Steffensmeier, Andrew M., et al.
Publicado: (2013)

Amyloid-β and hyperphosphorylated tau synergy drives metabolic decline in preclinical Alzheimer's disease
por: Pascoal, T A, et al.
Publicado: (2017)

Cerebrospinal fluid amyloid-β 2-42 is decreased in Alzheimer’s, but not in frontotemporal dementia
por: Bibl, Mirko, et al.
Publicado: (2012)

Derivation of cutoffs for the Elecsys(®) amyloid β (1–42) assay in Alzheimer's disease
por: Shaw, Leslie M., et al.
Publicado: (2018)

Cerebrospinal Fluid Aβ(42/40) Corresponds Better than Aβ(42) to Amyloid PET in Alzheimer’s Disease
por: Lewczuk, Piotr, et al.
Publicado: (2016)

Synergistic Effect on Neurodegeneration by N-Truncated Aβ(4−42) and Pyroglutamate Aβ(3−42) in a Mouse Model of Alzheimer's Disease
por: Lopez-Noguerola, Jose S., et al.
Publicado: (2018)

Hippocampal Deficits in Amyloid-β-Related Rodent Models of Alzheimer’s Disease
por: Vyas, Yukti, et al.
Publicado: (2020)

Oligomer Formation by Amyloid-β42 in a Membrane-Mimicking Environment in Alzheimer’s Disease
por: Rosenberry, Terrone L., et al.
Publicado: (2022)

Influence of Hyperglycemic Conditions on Self-Association of the Alzheimer’s Amyloid β (Aβ(1–42)) Peptide
por: Menon, Sneha, et al.
Publicado: (2017)

In the blood: biomarkers for amyloid pathology and neurodegeneration in Alzheimer’s disease
por: Toombs, Jamie, et al.
Publicado: (2020)

High Soluble Amyloid-β(42) Predicts Normal Cognition in Amyloid-Positive Individuals with Alzheimer’s Disease-Causing Mutations
por: Sturchio, Andrea, et al.
Publicado: (2022)

Guanosine Prevents Spatial Memory Impairment and Hippocampal Damage Following Amyloid-β(1–42) Administration in Mice
por: Coelho, Victor, et al.
Publicado: (2022)

Protection of TGF-β1 against Neuroinflammation and Neurodegeneration in Aβ(1–42)-Induced Alzheimer’s Disease Model Rats
por: Chen, Jia-Hui, et al.
Publicado: (2015)

Formation of the 42-mer Amyloid β Radical and the Therapeutic Role of Superoxide Dismutase in Alzheimer's Disease
por: Murakami, Kazuma, et al.
Publicado: (2011)

Aβ(1–42) Fibril Structure Illuminates Self-recognition and Replication of Amyloid in Alzheimer’s
por: Xiao, Yiling, et al.
Publicado: (2015)

Head-to-Head Comparison of 8 Plasma Amyloid-β 42/40 Assays in Alzheimer Disease
por: Janelidze, Shorena, et al.
Publicado: (2021)

Is plasma amyloid-β 1–42/1–40 a better biomarker for Alzheimer’s disease than AβX–42/X–40?
por: Klafki, Hans-Wolfgang, et al.
Publicado: (2022)

The roles of lipid and glucose metabolism in modulation of β-amyloid, tau, and neurodegeneration in the pathogenesis of Alzheimer disease
por: Sato, Naoyuki, et al.
Publicado: (2015)

Brain alterations in the early Alzheimer’s continuum with amyloid-β, tau, glial and neurodegeneration CSF markers
por: Salvadó, Gemma, et al.
Publicado: (2022)

Advantages and disadvantages of the use of the CSF Amyloid β (Aβ) 42/40 ratio in the diagnosis of Alzheimer’s Disease
por: Hansson, Oskar, et al.
Publicado: (2019)

Compulsive-like Behaviors in Amyloid-β 1-42–Induced Alzheimer’s Disease in Mice Are Associated With Hippocampo-cortical Neural Circuit Dysfunction
por: Shivakumar, Apoorva Bettagere, et al.
Publicado: (2023)

Plasma amyloid-β42/40 and apolipoprotein E for amyloid PET pre-screening in secondary prevention trials of Alzheimer’s disease
por: Cullen, Nicholas C, et al.
Publicado: (2023)

Microarray Analysis on Human Neuroblastoma Cells Exposed to Aluminum, β(1–42)-Amyloid or the β(1–42)-Amyloid Aluminum Complex
por: Gatta, Valentina, et al.
Publicado: (2011)

Linking Aβ42-Induced Hyperexcitability to Neurodegeneration, Learning and Motor Deficits, and a Shorter Lifespan in an Alzheimer’s Model
por: Ping, Yong, et al.
Publicado: (2015)

Different associations between amyloid-βeta 42, amyloid-βeta 40, and amyloid-βeta 42/40 with soluble phosphorylated-tau and disease burden in Alzheimer’s disease: a cerebrospinal fluid and fluorodeoxyglucose-positron emission tomography study
por: Motta, Caterina, et al.
Publicado: (2023)

Pyroglutamation of amyloid-βx-42 (Aβx-42) followed by Aβ1–40 deposition underlies plaque polymorphism in progressing Alzheimer's disease pathology
por: Michno, Wojciech, et al.
Publicado: (2019)

Cellular Prion Protein as a Receptor of Toxic Amyloid-β42 Oligomers Is Important for Alzheimer’s Disease
por: Zhang, Yuan, et al.
Publicado: (2019)

Ultrastructural evidence for self-replication of Alzheimer-associated Aβ42 amyloid along the sides of fibrils
por: Törnquist, Mattias, et al.
Publicado: (2020)

Cannot write session to /tmp/vufind_sessions/sess_037cv92qv7365sjcifdb1ms8ji