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Expression of Concern: miR-517a is upregulated in glioma and promotes glioma tumorigenesis in vitro and in vivo

Detalles Bibliográficos
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Portland Press Ltd. 2022
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9620486/
https://www.ncbi.nlm.nih.gov/pubmed/36287110
http://dx.doi.org/10.1042/BSR-2018-1196_EOC
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spelling pubmed-96204862022-11-04 Expression of Concern: miR-517a is upregulated in glioma and promotes glioma tumorigenesis in vitro and in vivo Biosci Rep Expression of Concern Portland Press Ltd. 2022-10-26 /pmc/articles/PMC9620486/ /pubmed/36287110 http://dx.doi.org/10.1042/BSR-2018-1196_EOC Text en © 2022 The Author(s). https://creativecommons.org/licenses/by/4.0/This is an open access article published by Portland Press Limited on behalf of the Biochemical Society and distributed under the Creative Commons Attribution License 4.0 (CC BY) (https://creativecommons.org/licenses/by/4.0/) .
spellingShingle Expression of Concern
Expression of Concern: miR-517a is upregulated in glioma and promotes glioma tumorigenesis in vitro and in vivo
title Expression of Concern: miR-517a is upregulated in glioma and promotes glioma tumorigenesis in vitro and in vivo
title_full Expression of Concern: miR-517a is upregulated in glioma and promotes glioma tumorigenesis in vitro and in vivo
title_fullStr Expression of Concern: miR-517a is upregulated in glioma and promotes glioma tumorigenesis in vitro and in vivo
title_full_unstemmed Expression of Concern: miR-517a is upregulated in glioma and promotes glioma tumorigenesis in vitro and in vivo
title_short Expression of Concern: miR-517a is upregulated in glioma and promotes glioma tumorigenesis in vitro and in vivo
title_sort expression of concern: mir-517a is upregulated in glioma and promotes glioma tumorigenesis in vitro and in vivo
topic Expression of Concern
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9620486/
https://www.ncbi.nlm.nih.gov/pubmed/36287110
http://dx.doi.org/10.1042/BSR-2018-1196_EOC