Cargando…
PSUN136 CCN5/WISP2 gene deficiency improves insulin sensitivity and glucose tolerance in diet-induced obesity, especially in male mice
: CCN5/WISP2 is a matricellular protein, the expression of which is under the regulation of Wnt signaling and IGF-1. Our initial characterization supports the notion that CCN5 promotes the proliferation and survival of pancreatic β-cells leading to metabolic benefits [1]. Recently, the effects of C...
Autores principales: | Xega, Viktoria, Zakikhani, Mahvash, Lopez, Noah, Shahraki, Marzieh Eskandari, Lyons, Karen M, Liu, Jun-Li |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Oxford University Press
2022
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9624652/ http://dx.doi.org/10.1210/jendso/bvac150.749 |
Ejemplares similares
-
SAT045 Sexually Dimorphic Effects Of CCN5/WISP2 Overexpression On HFD-STZ Induced Diabetes And Glucose Intolerance
por: Alami, Tara, et al.
Publicado: (2023) -
PSUN299 GLP-1 Contribution to Insulin Secretory Response to Protein Ingestion and Insulin Kinetics Following Gastric Bypass and Sleeve Gastrectomy
por: Rayas, Maria, et al.
Publicado: (2022) -
PSUN140 “Awakening of the Beta Cell(©)”
por: Jasper, Josephine, et al.
Publicado: (2022) -
PSUN274 Chlorthalidone-Induced Diabetic Ketoacidosis.
por: Zafar, Rafia, et al.
Publicado: (2022) -
PSUN254 Hirata's Disease - An Unexpected Surprise
por: Al-Bahadili, Huda, et al.
Publicado: (2022)