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A potential explanation for the global increase in tropical cyclone rapid intensification
Tropical cyclone rapid intensification events often cause destructive hurricane landfalls because they are associated with the strongest storms and forecasts with the highest errors. Multi-decade observational datasets of tropical cyclone behavior have recently enabled documentation of upward trends...
Autores principales: | , , , , , , , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group UK
2022
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9636401/ https://www.ncbi.nlm.nih.gov/pubmed/36333371 http://dx.doi.org/10.1038/s41467-022-34321-6 |
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author | Bhatia, Kieran Baker, Alexander Yang, Wenchang Vecchi, Gabriel Knutson, Thomas Murakami, Hiroyuki Kossin, James Hodges, Kevin Dixon, Keith Bronselaer, Benjamin Whitlock, Carolyn |
author_facet | Bhatia, Kieran Baker, Alexander Yang, Wenchang Vecchi, Gabriel Knutson, Thomas Murakami, Hiroyuki Kossin, James Hodges, Kevin Dixon, Keith Bronselaer, Benjamin Whitlock, Carolyn |
author_sort | Bhatia, Kieran |
collection | PubMed |
description | Tropical cyclone rapid intensification events often cause destructive hurricane landfalls because they are associated with the strongest storms and forecasts with the highest errors. Multi-decade observational datasets of tropical cyclone behavior have recently enabled documentation of upward trends in tropical cyclone rapid intensification in several basins. However, a robust anthropogenic signal in global intensification trends and the physical drivers of intensification trends have yet to be identified. To address these knowledge gaps, here we compare the observed trends in intensification and tropical cyclone environmental parameters to simulated natural variability in a high-resolution global climate model. In multiple basins and the global dataset, we detect a significant increase in intensification rates with a positive contribution from anthropogenic forcing. Furthermore, thermodynamic environments around tropical cyclones have become more favorable for intensification, and climate models show anthropogenic warming has significantly increased the probability of these changes. |
format | Online Article Text |
id | pubmed-9636401 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2022 |
publisher | Nature Publishing Group UK |
record_format | MEDLINE/PubMed |
spelling | pubmed-96364012022-11-06 A potential explanation for the global increase in tropical cyclone rapid intensification Bhatia, Kieran Baker, Alexander Yang, Wenchang Vecchi, Gabriel Knutson, Thomas Murakami, Hiroyuki Kossin, James Hodges, Kevin Dixon, Keith Bronselaer, Benjamin Whitlock, Carolyn Nat Commun Article Tropical cyclone rapid intensification events often cause destructive hurricane landfalls because they are associated with the strongest storms and forecasts with the highest errors. Multi-decade observational datasets of tropical cyclone behavior have recently enabled documentation of upward trends in tropical cyclone rapid intensification in several basins. However, a robust anthropogenic signal in global intensification trends and the physical drivers of intensification trends have yet to be identified. To address these knowledge gaps, here we compare the observed trends in intensification and tropical cyclone environmental parameters to simulated natural variability in a high-resolution global climate model. In multiple basins and the global dataset, we detect a significant increase in intensification rates with a positive contribution from anthropogenic forcing. Furthermore, thermodynamic environments around tropical cyclones have become more favorable for intensification, and climate models show anthropogenic warming has significantly increased the probability of these changes. Nature Publishing Group UK 2022-11-04 /pmc/articles/PMC9636401/ /pubmed/36333371 http://dx.doi.org/10.1038/s41467-022-34321-6 Text en © The Author(s) 2022, corrected publication 2023 https://creativecommons.org/licenses/by/4.0/Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/ (https://creativecommons.org/licenses/by/4.0/) . |
spellingShingle | Article Bhatia, Kieran Baker, Alexander Yang, Wenchang Vecchi, Gabriel Knutson, Thomas Murakami, Hiroyuki Kossin, James Hodges, Kevin Dixon, Keith Bronselaer, Benjamin Whitlock, Carolyn A potential explanation for the global increase in tropical cyclone rapid intensification |
title | A potential explanation for the global increase in tropical cyclone rapid intensification |
title_full | A potential explanation for the global increase in tropical cyclone rapid intensification |
title_fullStr | A potential explanation for the global increase in tropical cyclone rapid intensification |
title_full_unstemmed | A potential explanation for the global increase in tropical cyclone rapid intensification |
title_short | A potential explanation for the global increase in tropical cyclone rapid intensification |
title_sort | potential explanation for the global increase in tropical cyclone rapid intensification |
topic | Article |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9636401/ https://www.ncbi.nlm.nih.gov/pubmed/36333371 http://dx.doi.org/10.1038/s41467-022-34321-6 |
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