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Influence of Nutrition, Food Supplements and Lifestyle in Hair Disorders

Genetic, androgenic, epigenetic, environmental, lifestyle, diet, and nutritional factors have influence over promoting or regulating inflammation, immunity, and genetic expression predisposing to hair loss. Oxidative stress is a major mediator for various mechanisms of hair loss, including the relea...

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Autor principal: Rajput, Rajendrasingh J.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Wolters Kluwer - Medknow 2022
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9650738/
https://www.ncbi.nlm.nih.gov/pubmed/36386748
http://dx.doi.org/10.4103/idoj.idoj_175_22
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author Rajput, Rajendrasingh J.
author_facet Rajput, Rajendrasingh J.
author_sort Rajput, Rajendrasingh J.
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description Genetic, androgenic, epigenetic, environmental, lifestyle, diet, and nutritional factors have influence over promoting or regulating inflammation, immunity, and genetic expression predisposing to hair loss. Oxidative stress is a major mediator for various mechanisms of hair loss, including the release of transforming growth factor-β ( TGF-β). Nutrients counter oxidative stress, repair cellular damage, support cellular functions, and restore hair growth. Nutrients can be synergistic or antagonistic. Covert subclinical nutritional deficiencies are common. Higher dose of nutrients does not mean higher efficiency but may reverse the benefits converting an antioxidant to become prooxidant. Nutrients do not work alone but are supported by accessory micronutrients which ensure biological utilization. Providing proper nutritional environment can neutralize free radicals and perpetuate active hair growth cycles.
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spelling pubmed-96507382022-11-15 Influence of Nutrition, Food Supplements and Lifestyle in Hair Disorders Rajput, Rajendrasingh J. Indian Dermatol Online J Trichology Symposium - Review Article Genetic, androgenic, epigenetic, environmental, lifestyle, diet, and nutritional factors have influence over promoting or regulating inflammation, immunity, and genetic expression predisposing to hair loss. Oxidative stress is a major mediator for various mechanisms of hair loss, including the release of transforming growth factor-β ( TGF-β). Nutrients counter oxidative stress, repair cellular damage, support cellular functions, and restore hair growth. Nutrients can be synergistic or antagonistic. Covert subclinical nutritional deficiencies are common. Higher dose of nutrients does not mean higher efficiency but may reverse the benefits converting an antioxidant to become prooxidant. Nutrients do not work alone but are supported by accessory micronutrients which ensure biological utilization. Providing proper nutritional environment can neutralize free radicals and perpetuate active hair growth cycles. Wolters Kluwer - Medknow 2022-10-21 /pmc/articles/PMC9650738/ /pubmed/36386748 http://dx.doi.org/10.4103/idoj.idoj_175_22 Text en Copyright: © 2022 Indian Dermatology Online Journal https://creativecommons.org/licenses/by-nc-sa/4.0/This is an open access journal, and articles are distributed under the terms of the Creative Commons Attribution-NonCommercial-ShareAlike 4.0 License, which allows others to remix, tweak, and build upon the work non-commercially, as long as appropriate credit is given and the new creations are licensed under the identical terms.
spellingShingle Trichology Symposium - Review Article
Rajput, Rajendrasingh J.
Influence of Nutrition, Food Supplements and Lifestyle in Hair Disorders
title Influence of Nutrition, Food Supplements and Lifestyle in Hair Disorders
title_full Influence of Nutrition, Food Supplements and Lifestyle in Hair Disorders
title_fullStr Influence of Nutrition, Food Supplements and Lifestyle in Hair Disorders
title_full_unstemmed Influence of Nutrition, Food Supplements and Lifestyle in Hair Disorders
title_short Influence of Nutrition, Food Supplements and Lifestyle in Hair Disorders
title_sort influence of nutrition, food supplements and lifestyle in hair disorders
topic Trichology Symposium - Review Article
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9650738/
https://www.ncbi.nlm.nih.gov/pubmed/36386748
http://dx.doi.org/10.4103/idoj.idoj_175_22
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