Cargando…
TRAF6 autophagic degradation by avibirnavirus VP3 inhibits antiviral innate immunity via blocking NFKB/NF-κB activation
Ubiquitination is an important reversible post-translational modification. Many viruses hijack the host ubiquitin system to enhance self-replication. In the present study, we found that Avibirnavirus VP3 protein was ubiquitinated during infection and supported virus replication by ubiquitination. Ma...
Autores principales: | Deng, Tingjuan, Hu, Boli, Wang, Xingbo, Ding, Shuxiang, Lin, Lulu, Yan, Yan, Peng, Xiran, Zheng, Xiaojuan, Liao, Min, Jin, Yulan, Dong, Weiren, Gu, Jinyan, Zhou, Jiyong |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Taylor & Francis
2022
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9673932/ https://www.ncbi.nlm.nih.gov/pubmed/35266845 http://dx.doi.org/10.1080/15548627.2022.2047384 |
Ejemplares similares
-
Inhibition of Antiviral Innate Immunity by Avibirnavirus VP3 via Blocking TBK1-TRAF3 Complex Formation and IRF3 Activation
por: Deng, Tingjuan, et al.
Publicado: (2021) -
DeSUMOylation of Apoptosis Inhibitor 5 by Avibirnavirus VP3 Supports Virus Replication
por: Deng, Tingjuan, et al.
Publicado: (2021) -
Ubiquitination Is Essential for Avibirnavirus Replication by Supporting VP1 Polymerase Activity
por: Wu, Huansheng, et al.
Publicado: (2019) -
Avibirnavirus VP4 Protein Is a Phosphoprotein and Partially Contributes to the Cleavage of Intermediate Precursor VP4-VP3 Polyprotein
por: Wang, Sanying, et al.
Publicado: (2015) -
Cytoplasmic Cargo Receptor p62 Inhibits Avibirnavirus Replication by Mediating Autophagic Degradation of Viral Protein VP2
por: Li, Yahui, et al.
Publicado: (2020)