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Interaction between the Gut Microbiota and Intestinal Motility

The gut microbiota is the largest symbiotic ecosystem with the host and has been proven to play an important role in maintaining the stability of the intestinal environment. The imbalance of the gut microbiota is caused by the imbalance between the symbiotic microbiota and the pathogenic microbiota....

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Detalles Bibliográficos
Autores principales: Liu, Qihong, Luo, Yunfeng, Ke, Xiao
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Hindawi 2022
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9681544/
https://www.ncbi.nlm.nih.gov/pubmed/36425261
http://dx.doi.org/10.1155/2022/3240573
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author Liu, Qihong
Luo, Yunfeng
Ke, Xiao
author_facet Liu, Qihong
Luo, Yunfeng
Ke, Xiao
author_sort Liu, Qihong
collection PubMed
description The gut microbiota is the largest symbiotic ecosystem with the host and has been proven to play an important role in maintaining the stability of the intestinal environment. The imbalance of the gut microbiota is caused by the imbalance between the symbiotic microbiota and the pathogenic microbiota. The commensal microbiome regulates intestinal motility, while the pathogenic microbiome causes intestinal motility disorder, resulting in disease development. Intestinal motility is a relatively general term, and its meaning may include intestinal muscle contraction, intestinal wall biomechanics, intestinal compliance, and transmission. The role of intestinal microecology and intestinal motility are interrelated, intestinal flora disorder mediates intestinal motility, and abnormal intestinal motility affects colonization of the intestinal flora. In this review, we briefly outlined the interaction between gut microbiota and intestinal motility and provided a reference for future studies.
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spelling pubmed-96815442022-11-23 Interaction between the Gut Microbiota and Intestinal Motility Liu, Qihong Luo, Yunfeng Ke, Xiao Evid Based Complement Alternat Med Review Article The gut microbiota is the largest symbiotic ecosystem with the host and has been proven to play an important role in maintaining the stability of the intestinal environment. The imbalance of the gut microbiota is caused by the imbalance between the symbiotic microbiota and the pathogenic microbiota. The commensal microbiome regulates intestinal motility, while the pathogenic microbiome causes intestinal motility disorder, resulting in disease development. Intestinal motility is a relatively general term, and its meaning may include intestinal muscle contraction, intestinal wall biomechanics, intestinal compliance, and transmission. The role of intestinal microecology and intestinal motility are interrelated, intestinal flora disorder mediates intestinal motility, and abnormal intestinal motility affects colonization of the intestinal flora. In this review, we briefly outlined the interaction between gut microbiota and intestinal motility and provided a reference for future studies. Hindawi 2022-11-15 /pmc/articles/PMC9681544/ /pubmed/36425261 http://dx.doi.org/10.1155/2022/3240573 Text en Copyright © 2022 Qihong Liu et al. https://creativecommons.org/licenses/by/4.0/This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
spellingShingle Review Article
Liu, Qihong
Luo, Yunfeng
Ke, Xiao
Interaction between the Gut Microbiota and Intestinal Motility
title Interaction between the Gut Microbiota and Intestinal Motility
title_full Interaction between the Gut Microbiota and Intestinal Motility
title_fullStr Interaction between the Gut Microbiota and Intestinal Motility
title_full_unstemmed Interaction between the Gut Microbiota and Intestinal Motility
title_short Interaction between the Gut Microbiota and Intestinal Motility
title_sort interaction between the gut microbiota and intestinal motility
topic Review Article
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9681544/
https://www.ncbi.nlm.nih.gov/pubmed/36425261
http://dx.doi.org/10.1155/2022/3240573
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