UBR7 inhibits HCC tumorigenesis by targeting Keap1/Nrf2/Bach1/HK2 and glycolysis

BACKGROUND: Glycolysis metabolism is an attractive target for cancer therapy. Reprogramming metabolic pathways could improve the ability of metabolic inhibitors to suppress cancers with limited treatment options. The ubiquitin–proteasome system facilitates the turnover of most intracellular proteins...

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Detalles Bibliográficos
Autores principales: Zhao, Liang, Kang, Min, Liu, Xiaomeng, Wang, Zhenran, Wang, Yan, Chen, Haiqiang, Liu, Wenhui, Liu, Shiqian, Li, Baibei, Li, Chong, Chang, Antao, Tang, Bo
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2022
Materias:
Research
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9686014/
https://www.ncbi.nlm.nih.gov/pubmed/36419136
http://dx.doi.org/10.1186/s13046-022-02528-6

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9686014/
https://www.ncbi.nlm.nih.gov/pubmed/36419136
http://dx.doi.org/10.1186/s13046-022-02528-6

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