Cargando…
Maternal immune activation-induced proBDNF-mediated neural information processing dysfunction at hippocampal CA3-CA1 synapses associated with memory deficits in offspring
Prenatal exposure to maternal infection increases the risk of offspring developing schizophrenia in adulthood. Current theories suggest that the consequences of MIA on mBDNF secretion may underlie the increased risk of cognitive disorder. There is little evidence for whether the expression of its pr...
Autores principales: | Sun, Wei, Mei, Yazi, Li, Xiaoliang, Yang, Yang, An, Lei |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Frontiers Media S.A.
2022
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9691851/ https://www.ncbi.nlm.nih.gov/pubmed/36438556 http://dx.doi.org/10.3389/fcell.2022.1018586 |
Ejemplares similares
-
Requirements of Postnatal proBDNF in the Hippocampus for Spatial Memory Consolidation and Neural Function
por: Sun, Wei, et al.
Publicado: (2021) -
Aluminium oxide nanoparticles compromise spatial memory performance and proBDNF-mediated neuronal function in the hippocampus of rats
por: Sun, Wei, et al.
Publicado: (2022) -
Increased Hippocampal ProBDNF Contributes to Memory Impairments in Aged Mice
por: Buhusi, Mona, et al.
Publicado: (2017) -
Proteolysis of proBDNF Is a Key Regulator in the Formation of Memory
por: Barnes, Philip, et al.
Publicado: (2008) -
Injection of Anti-proBDNF Attenuates Hippocampal-Dependent Learning and Memory Dysfunction in Mice With Sepsis-Associated Encephalopathy
por: Cui, Yan-Hui, et al.
Publicado: (2021)