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Not only mutations but also tumorigenesis can be substantially attributed to DNA damage from reactive oxygen species in RUNX1::RUNX1T1-fusion-positive acute myeloid leukemia
Autores principales: | , , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group UK
2022
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9712081/ https://www.ncbi.nlm.nih.gov/pubmed/36369483 http://dx.doi.org/10.1038/s41375-022-01752-5 |
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author | Mandell, Jeffrey D. Fisk, J. Nick Cyrenne, Ethan Xu, Mina L. Cannataro, Vincent L. Townsend, Jeffrey P. |
author_facet | Mandell, Jeffrey D. Fisk, J. Nick Cyrenne, Ethan Xu, Mina L. Cannataro, Vincent L. Townsend, Jeffrey P. |
author_sort | Mandell, Jeffrey D. |
collection | PubMed |
description | |
format | Online Article Text |
id | pubmed-9712081 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2022 |
publisher | Nature Publishing Group UK |
record_format | MEDLINE/PubMed |
spelling | pubmed-97120812022-12-02 Not only mutations but also tumorigenesis can be substantially attributed to DNA damage from reactive oxygen species in RUNX1::RUNX1T1-fusion-positive acute myeloid leukemia Mandell, Jeffrey D. Fisk, J. Nick Cyrenne, Ethan Xu, Mina L. Cannataro, Vincent L. Townsend, Jeffrey P. Leukemia Correspondence Nature Publishing Group UK 2022-11-11 2022 /pmc/articles/PMC9712081/ /pubmed/36369483 http://dx.doi.org/10.1038/s41375-022-01752-5 Text en © The Author(s) 2022 https://creativecommons.org/licenses/by/4.0/Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/ (https://creativecommons.org/licenses/by/4.0/) . |
spellingShingle | Correspondence Mandell, Jeffrey D. Fisk, J. Nick Cyrenne, Ethan Xu, Mina L. Cannataro, Vincent L. Townsend, Jeffrey P. Not only mutations but also tumorigenesis can be substantially attributed to DNA damage from reactive oxygen species in RUNX1::RUNX1T1-fusion-positive acute myeloid leukemia |
title | Not only mutations but also tumorigenesis can be substantially attributed to DNA damage from reactive oxygen species in RUNX1::RUNX1T1-fusion-positive acute myeloid leukemia |
title_full | Not only mutations but also tumorigenesis can be substantially attributed to DNA damage from reactive oxygen species in RUNX1::RUNX1T1-fusion-positive acute myeloid leukemia |
title_fullStr | Not only mutations but also tumorigenesis can be substantially attributed to DNA damage from reactive oxygen species in RUNX1::RUNX1T1-fusion-positive acute myeloid leukemia |
title_full_unstemmed | Not only mutations but also tumorigenesis can be substantially attributed to DNA damage from reactive oxygen species in RUNX1::RUNX1T1-fusion-positive acute myeloid leukemia |
title_short | Not only mutations but also tumorigenesis can be substantially attributed to DNA damage from reactive oxygen species in RUNX1::RUNX1T1-fusion-positive acute myeloid leukemia |
title_sort | not only mutations but also tumorigenesis can be substantially attributed to dna damage from reactive oxygen species in runx1::runx1t1-fusion-positive acute myeloid leukemia |
topic | Correspondence |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9712081/ https://www.ncbi.nlm.nih.gov/pubmed/36369483 http://dx.doi.org/10.1038/s41375-022-01752-5 |
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