Increased IL-26 associates with markers of hyperinflammation and tissue damage in patients with acute COVID-19

Interleukin-26 (IL-26) is released by several immune and structural cells following stimulation of toll-like receptors (TLRs), whereupon it can directly inhibit viral replication and enhance neutrophil chemotaxis. Given these unique properties, IL-26 has emerged as an intriguing mediator of host def...

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Detalles Bibliográficos
Autores principales: Cardenas, Eduardo I., Ekstedt, Sandra, Piersiala, Krzysztof, Petro, Marianne, Karlsson, Agneta, Kågedal, Åsa, Kumlien Georén, Susanna, Cardell, Lars-Olaf, Lindén, Anders
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Frontiers Media S.A. 2022
Materias:
Immunology
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9712219/
https://www.ncbi.nlm.nih.gov/pubmed/36466824
http://dx.doi.org/10.3389/fimmu.2022.1016991

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9712219/
https://www.ncbi.nlm.nih.gov/pubmed/36466824
http://dx.doi.org/10.3389/fimmu.2022.1016991

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