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Increased IL-26 associates with markers of hyperinflammation and tissue damage in patients with acute COVID-19
Interleukin-26 (IL-26) is released by several immune and structural cells following stimulation of toll-like receptors (TLRs), whereupon it can directly inhibit viral replication and enhance neutrophil chemotaxis. Given these unique properties, IL-26 has emerged as an intriguing mediator of host def...
| Autores principales: | , , , , , , , , |
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
| Publicado: |
Frontiers Media S.A.
2022
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| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9712219/ https://www.ncbi.nlm.nih.gov/pubmed/36466824 http://dx.doi.org/10.3389/fimmu.2022.1016991 |