Cargando…
Advanced glycation end products induce senescence of atrial myocytes and increase susceptibility of atrial fibrillation in diabetic mice
Diabetes mellitus (DM) is a common chronic metabolic disease caused by significant accumulation of advanced glycation end products (AGEs). Atrial fibrillation (AF) is a common cardiovascular complication of DM. Here, we aim to clarify the role and mechanism of atrial myocyte senescence in the suscep...
Autores principales: | Zheng, Dan‐Lin, Wu, Qing‐Rui, Zeng, Peng, Li, Sui‐Min, Cai, Yong‐Jiang, Chen, Shu‐Zhen, Luo, Xue‐Shan, Kuang, Su‐Juan, Rao, Fang, Lai, Ying‐Yu, Zhou, Meng‐Yuan, Wu, Fei‐Long, Yang, Hui, Deng, Chun‐Yu |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
John Wiley and Sons Inc.
2022
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9741501/ https://www.ncbi.nlm.nih.gov/pubmed/36278684 http://dx.doi.org/10.1111/acel.13734 |
Ejemplares similares
-
Cross-talk between macrophages and atrial myocytes in atrial fibrillation
por: Sun, Zewei, et al.
Publicado: (2016) -
Atrial myocyte-derived exosomal microRNA contributes to atrial fibrosis in atrial fibrillation
por: Hao, Hongting, et al.
Publicado: (2022) -
Advanced Glycation End Products Downregulate Connexin 43 and Connexin 40 in Diabetic Atrial Myocytes via the AMPK Pathway
por: Yang, Fan, et al.
Publicado: (2023) -
Connexin 43 is involved in the sympathetic atrial fibrillation in canine and canine atrial myocytes
por: Shu, Chenglin, et al.
Publicado: (2017) -
Proarrhythmic Remodeling of Atrial Myocyte Ca(2+) Handling in Atrial Fibrillation
por: Houser, Steven R.
Publicado: (2023)