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Ortner syndrome caused by aberrant right subclavian artery: A case report
Ortner syndrome (cardiovocal hoarseness) is characterized by recurrent laryngeal nerve paralysis secondary to a cardiovascular abnormality. Ortner syndrome caused by an aberrant right subclavian artery following a retroesophageal course without aneurysm formation is rare, with only 1 case reported i...
Autores principales: | , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Lippincott Williams & Wilkins
2022
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9750515/ https://www.ncbi.nlm.nih.gov/pubmed/36626475 http://dx.doi.org/10.1097/MD.0000000000032272 |
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author | Chen, Yen-Wen Chang, Shih-Lun Wu, Nan-Chun Shih, Yun-Ju |
author_facet | Chen, Yen-Wen Chang, Shih-Lun Wu, Nan-Chun Shih, Yun-Ju |
author_sort | Chen, Yen-Wen |
collection | PubMed |
description | Ortner syndrome (cardiovocal hoarseness) is characterized by recurrent laryngeal nerve paralysis secondary to a cardiovascular abnormality. Ortner syndrome caused by an aberrant right subclavian artery following a retroesophageal course without aneurysm formation is rare, with only 1 case reported in the literature. Cardiovascular abnormalities could be life-threatening and require early diagnosis and treatment. However, such abnormalities are not often considered by clinical practitioners when patients initially present with hoarseness. PATIENT CONCERNS: A 34-year-old woman without any medical history presented to our outpatient department with hoarseness and mild dysphagia for 1 month. DIAGNOSIS: Upon stroboscopic examination, left vocal cord incomplete paralysis was noted. Contrast-enhanced computed tomography revealed an aberrant right subclavian artery arising from the left aortic arch, causing focal compression of the esophagus and, potentially, compression of the left recurrent laryngeal nerve compression. The patient was diagnosed as left recurrent laryngeal nerve paralysis caused by an aberrant right subclavian artery following a retroesophageal course without aneurysm formation. INTERVENTIONS: The patient was referred to a cardiovascular surgeon for resection and bypass surgery. Both the dysphagia and the hoarseness improved after the surgery. OUTCOMES: Significant improvement of the left vocal cord paralysis and no vocal cord adduction were seen upon stroboscopic examination after 3 months. During the 5-year follow-up period, the patient remained well, and no signs of recurrence were noted. CONCLUSION: This case can increase otolaryngologists’ awareness of this etiology of hoarseness and consider it in their differential diagnosis. |
format | Online Article Text |
id | pubmed-9750515 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2022 |
publisher | Lippincott Williams & Wilkins |
record_format | MEDLINE/PubMed |
spelling | pubmed-97505152022-12-28 Ortner syndrome caused by aberrant right subclavian artery: A case report Chen, Yen-Wen Chang, Shih-Lun Wu, Nan-Chun Shih, Yun-Ju Medicine (Baltimore) 6000 Ortner syndrome (cardiovocal hoarseness) is characterized by recurrent laryngeal nerve paralysis secondary to a cardiovascular abnormality. Ortner syndrome caused by an aberrant right subclavian artery following a retroesophageal course without aneurysm formation is rare, with only 1 case reported in the literature. Cardiovascular abnormalities could be life-threatening and require early diagnosis and treatment. However, such abnormalities are not often considered by clinical practitioners when patients initially present with hoarseness. PATIENT CONCERNS: A 34-year-old woman without any medical history presented to our outpatient department with hoarseness and mild dysphagia for 1 month. DIAGNOSIS: Upon stroboscopic examination, left vocal cord incomplete paralysis was noted. Contrast-enhanced computed tomography revealed an aberrant right subclavian artery arising from the left aortic arch, causing focal compression of the esophagus and, potentially, compression of the left recurrent laryngeal nerve compression. The patient was diagnosed as left recurrent laryngeal nerve paralysis caused by an aberrant right subclavian artery following a retroesophageal course without aneurysm formation. INTERVENTIONS: The patient was referred to a cardiovascular surgeon for resection and bypass surgery. Both the dysphagia and the hoarseness improved after the surgery. OUTCOMES: Significant improvement of the left vocal cord paralysis and no vocal cord adduction were seen upon stroboscopic examination after 3 months. During the 5-year follow-up period, the patient remained well, and no signs of recurrence were noted. CONCLUSION: This case can increase otolaryngologists’ awareness of this etiology of hoarseness and consider it in their differential diagnosis. Lippincott Williams & Wilkins 2022-12-09 /pmc/articles/PMC9750515/ /pubmed/36626475 http://dx.doi.org/10.1097/MD.0000000000032272 Text en Copyright © 2022 the Author(s). Published by Wolters Kluwer Health, Inc. https://creativecommons.org/licenses/by/4.0/This is an open access article distributed under the Creative Commons Attribution License 4.0 (CCBY) (https://creativecommons.org/licenses/by/4.0/) , which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. |
spellingShingle | 6000 Chen, Yen-Wen Chang, Shih-Lun Wu, Nan-Chun Shih, Yun-Ju Ortner syndrome caused by aberrant right subclavian artery: A case report |
title | Ortner syndrome caused by aberrant right subclavian artery: A case report |
title_full | Ortner syndrome caused by aberrant right subclavian artery: A case report |
title_fullStr | Ortner syndrome caused by aberrant right subclavian artery: A case report |
title_full_unstemmed | Ortner syndrome caused by aberrant right subclavian artery: A case report |
title_short | Ortner syndrome caused by aberrant right subclavian artery: A case report |
title_sort | ortner syndrome caused by aberrant right subclavian artery: a case report |
topic | 6000 |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9750515/ https://www.ncbi.nlm.nih.gov/pubmed/36626475 http://dx.doi.org/10.1097/MD.0000000000032272 |
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