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Movement Disorder in Demyelinating Disease: Tracing the Charcot's Foot Print

Movement disorders may be one of the neurological manifestations of demyelinating disorders. They can manifest in Parkinsonism or a wide spectrum of hyperkinetic movement disorders including tremor, paroxysmal dyskinesia, dystonia, chorea, and ballism. Some of these disorders occur during an acute e...

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Autores principales: Singh, Rashmi, Pandey, Sanjay
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Wolters Kluwer - Medknow 2022
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9764914/
https://www.ncbi.nlm.nih.gov/pubmed/36561038
http://dx.doi.org/10.4103/aian.aian_64_22
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author Singh, Rashmi
Pandey, Sanjay
author_facet Singh, Rashmi
Pandey, Sanjay
author_sort Singh, Rashmi
collection PubMed
description Movement disorders may be one of the neurological manifestations of demyelinating disorders. They can manifest in Parkinsonism or a wide spectrum of hyperkinetic movement disorders including tremor, paroxysmal dyskinesia, dystonia, chorea, and ballism. Some of these disorders occur during an acute episode of demyelination, whereas others can develop later or even may precede the onset of the demyelinating disorders. The pathophysiology of movement disorders in demyelination is complex and the current evidence indicates a wide involvement of different brain networks and spinal cord. Treatment is mainly symptomatic and oral pharmacological agents are the mainstay of the management. Botulinum toxin and neurosurgical interventions may be required in selected patients.
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spelling pubmed-97649142022-12-21 Movement Disorder in Demyelinating Disease: Tracing the Charcot's Foot Print Singh, Rashmi Pandey, Sanjay Ann Indian Acad Neurol AIAN Review Movement disorders may be one of the neurological manifestations of demyelinating disorders. They can manifest in Parkinsonism or a wide spectrum of hyperkinetic movement disorders including tremor, paroxysmal dyskinesia, dystonia, chorea, and ballism. Some of these disorders occur during an acute episode of demyelination, whereas others can develop later or even may precede the onset of the demyelinating disorders. The pathophysiology of movement disorders in demyelination is complex and the current evidence indicates a wide involvement of different brain networks and spinal cord. Treatment is mainly symptomatic and oral pharmacological agents are the mainstay of the management. Botulinum toxin and neurosurgical interventions may be required in selected patients. Wolters Kluwer - Medknow 2022 2022-06-08 /pmc/articles/PMC9764914/ /pubmed/36561038 http://dx.doi.org/10.4103/aian.aian_64_22 Text en Copyright: © 2022 Annals of Indian Academy of Neurology https://creativecommons.org/licenses/by-nc-sa/4.0/This is an open access journal, and articles are distributed under the terms of the Creative Commons Attribution-NonCommercial-ShareAlike 4.0 License, which allows others to remix, tweak, and build upon the work non-commercially, as long as appropriate credit is given and the new creations are licensed under the identical terms.
spellingShingle AIAN Review
Singh, Rashmi
Pandey, Sanjay
Movement Disorder in Demyelinating Disease: Tracing the Charcot's Foot Print
title Movement Disorder in Demyelinating Disease: Tracing the Charcot's Foot Print
title_full Movement Disorder in Demyelinating Disease: Tracing the Charcot's Foot Print
title_fullStr Movement Disorder in Demyelinating Disease: Tracing the Charcot's Foot Print
title_full_unstemmed Movement Disorder in Demyelinating Disease: Tracing the Charcot's Foot Print
title_short Movement Disorder in Demyelinating Disease: Tracing the Charcot's Foot Print
title_sort movement disorder in demyelinating disease: tracing the charcot's foot print
topic AIAN Review
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9764914/
https://www.ncbi.nlm.nih.gov/pubmed/36561038
http://dx.doi.org/10.4103/aian.aian_64_22
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