Cargando…
Tamoxifen-independent Cre-activity in SMMHC-CreER(T2) mice
BACKGROUND AND AIMS: Recent technological advances have established vascular smooth muscle cells (SMCs) as central players in atherosclerosis. Increasingly complex genetic mouse models have unveiled that 30–70% of cells in experimentally induced atherosclerotic lesions derive from a handful of media...
Autores principales: | Steffensen, L.B., Stubbe, J., Overgaard, M., Larsen, J.H. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Elsevier
2022
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9833216/ https://www.ncbi.nlm.nih.gov/pubmed/36644559 http://dx.doi.org/10.1016/j.athplu.2022.01.002 |
Ejemplares similares
-
Tamoxifen-Independent Recombination in the RIP-CreER Mouse
por: Liu, Yanmei, et al.
Publicado: (2010) -
Comparison of the Opn-CreER and Ck19-CreER Drivers in Bile Ducts of Normal and Injured Mouse Livers
por: Lesaffer, Bram, et al.
Publicado: (2019) -
Effects of CreER(T2), 4-OH Tamoxifen, and Gender on CFU-F Assays
por: McHaffie, Sophie L., et al.
Publicado: (2016) -
Genetic targeting of the endoderm with claudin-6(CreER)
por: Anderson, William J, et al.
Publicado: (2008) -
Tamoxifen-independent recombination of reporter genes limits lineage tracing and mosaic analysis using CreER(T2) lines
por: Álvarez-Aznar, A., et al.
Publicado: (2019)