Cargando…
Transcriptomic survey of key reproductive and metabolic tissues in mouse models of polycystic ovary syndrome
Excessive androgen production and obesity are key to polycystic ovary syndrome (PCOS) pathogenesis. Prenatal androgenized (PNA), peripubertal androgenized, and overexpression of nerve growth factor in theca cells (17NF) are commonly used PCOS-like mouse models and diet-induced maternal obesity model...
Autores principales: | Pei, Yu, Risal, Sanjiv, Jiang, Hong, Lu, Haojiang, Lindgren, Eva, Stener-Victorin, Elisabet, Deng, Qiaolin |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group UK
2023
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9849269/ https://www.ncbi.nlm.nih.gov/pubmed/36653487 http://dx.doi.org/10.1038/s42003-022-04362-0 |
Ejemplares similares
-
Origins and Impact of Psychological Traits in Polycystic Ovary Syndrome
por: Stener-Victorin, Elisabet, et al.
Publicado: (2019) -
MON-035 Does Androgen Exposure Result in Germline Transmission of PCOS-Like Phenotypes and Can It Be Reversed?
por: Lu, Haojiang, et al.
Publicado: (2020) -
Transgenerational transmission of reproductive and metabolic dysfunction in the male progeny of polycystic ovary syndrome
por: Risal, Sanjiv, et al.
Publicado: (2023) -
MON-032 Excessive Ovarian Sympathetic Activity Impairs Embryonic Development and Causes Reproductive and Metabolic Dysfunction
por: Manti, Maria, et al.
Publicado: (2020) -
SAT-201 Diet-Induced Obesity and Prenatal Androgen Exposure Increase Transgenerational Susceptibility to Metabolic Dysfunction in Male Adult Offspring
por: Lu, Haojiang, et al.
Publicado: (2019)