Cargando…
METTL3 Regulates Osteoclast Biological Behaviors via iNOS/NO-Mediated Mitochondrial Dysfunction in Inflammatory Conditions
Excessive differentiation of osteoclasts contributes to the disruption of bone homeostasis in inflammatory bone diseases. Methyltransferase-like 3 (METTL3), the core methyltransferase that installs an N6-methyladenosine (m(6)A) modification on RNA, has been reported to participate in bone pathophysi...
Autores principales: | Li, Di, He, Jinlin, Fang, Caihong, Zhang, Yiwen, He, Mingli, Zhang, Zhanqi, Hou, Jinsong, Xu, Qiong |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2023
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9862541/ https://www.ncbi.nlm.nih.gov/pubmed/36674918 http://dx.doi.org/10.3390/ijms24021403 |
Ejemplares similares
-
iNOS contributes to heart failure with preserved ejection fraction through mitochondrial dysfunction and Akt S-nitrosylation
por: Guo, Yongzheng, et al.
Publicado: (2022) -
Obesity-Induced Coronary Microvascular Disease Is Prevented by iNOS Deletion and Reversed by iNOS Inhibition
por: Shah, Soham A., et al.
Publicado: (2023) -
The dual role of iNOS in cancer()
por: Vannini, Federica, et al.
Publicado: (2015) -
eNOS/iNOS and endoplasmic reticulum stress-induced apoptosis in the placentas of patients with preeclampsia
por: Du, L, et al.
Publicado: (2017) -
Brain function in iNOS knock out or iNOS inhibited (l-NIL) mice under endotoxic shock
por: Schweighöfer, Hanna, et al.
Publicado: (2014)