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Red blood cells from endothelial nitric oxide synthase-deficient mice induce vascular dysfunction involving oxidative stress and endothelial arginase I
BACKGROUND & AIMS: Nitric oxide bioactivity (NO) from endothelial NO synthase (eNOS) importantly contributes to the maintenance of vascular homeostasis, and reduced eNOS activity has been associated with cardiovascular disease. Emerging evidence suggests interaction(s) between red blood cells (R...
Autores principales: | Zhuge, Zhengbing, McCann Haworth, Sarah, Nihlén, Carina, Carvalho, Lucas Rannier R.A., Heuser, Sophia K., Kleschyov, Andrei L., Nasiell, Josefine, Cortese-Krott, Miriam M., Weitzberg, Eddie, Lundberg, Jon O., Carlström, Mattias |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Elsevier
2023
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9868875/ https://www.ncbi.nlm.nih.gov/pubmed/36681048 http://dx.doi.org/10.1016/j.redox.2023.102612 |
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