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Hypertrophic cardiomyopathy with heart failure and ST‐segment elevation of the lateral wall

Hypertrophic cardiomyopathy (HCM) is the most common cardiovascular disease that is inherited from a single gene. Its clinical manifestations range from asymptomatic mutant gene carriers to patients with severe left ventricular effluent tract obstruction and end‐stage HCM with motor restriction. In...

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Detalles Bibliográficos
Autores principales: Bai, Lin, Wang, Yali, Chen, Fei, Peng, Yong
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2022
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9871721/
https://www.ncbi.nlm.nih.gov/pubmed/36178133
http://dx.doi.org/10.1002/ehf2.14185
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author Bai, Lin
Wang, Yali
Chen, Fei
Peng, Yong
author_facet Bai, Lin
Wang, Yali
Chen, Fei
Peng, Yong
author_sort Bai, Lin
collection PubMed
description Hypertrophic cardiomyopathy (HCM) is the most common cardiovascular disease that is inherited from a single gene. Its clinical manifestations range from asymptomatic mutant gene carriers to patients with severe left ventricular effluent tract obstruction and end‐stage HCM with motor restriction. In this case, we present a patient with the main presentation of heart failure and ST‐segment elevation of the lateral wall, as determined by electrocardiogram. The patient was finally diagnosed with HCM because of genetic testing and the presentation of extensive myocardial fibrosis with reduced systolic function on cardiac magnetic resonance imaging. The patient's clinical findings, electrocardiogram, and cardiac magnetic resonance imaging were different from those of typical patients with HCM.
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spelling pubmed-98717212023-01-27 Hypertrophic cardiomyopathy with heart failure and ST‐segment elevation of the lateral wall Bai, Lin Wang, Yali Chen, Fei Peng, Yong ESC Heart Fail Case Reports Hypertrophic cardiomyopathy (HCM) is the most common cardiovascular disease that is inherited from a single gene. Its clinical manifestations range from asymptomatic mutant gene carriers to patients with severe left ventricular effluent tract obstruction and end‐stage HCM with motor restriction. In this case, we present a patient with the main presentation of heart failure and ST‐segment elevation of the lateral wall, as determined by electrocardiogram. The patient was finally diagnosed with HCM because of genetic testing and the presentation of extensive myocardial fibrosis with reduced systolic function on cardiac magnetic resonance imaging. The patient's clinical findings, electrocardiogram, and cardiac magnetic resonance imaging were different from those of typical patients with HCM. John Wiley and Sons Inc. 2022-09-30 /pmc/articles/PMC9871721/ /pubmed/36178133 http://dx.doi.org/10.1002/ehf2.14185 Text en © 2022 The Authors. ESC Heart Failure published by John Wiley & Sons Ltd on behalf of European Society of Cardiology. https://creativecommons.org/licenses/by-nc/4.0/This is an open access article under the terms of the http://creativecommons.org/licenses/by-nc/4.0/ (https://creativecommons.org/licenses/by-nc/4.0/) License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited and is not used for commercial purposes.
spellingShingle Case Reports
Bai, Lin
Wang, Yali
Chen, Fei
Peng, Yong
Hypertrophic cardiomyopathy with heart failure and ST‐segment elevation of the lateral wall
title Hypertrophic cardiomyopathy with heart failure and ST‐segment elevation of the lateral wall
title_full Hypertrophic cardiomyopathy with heart failure and ST‐segment elevation of the lateral wall
title_fullStr Hypertrophic cardiomyopathy with heart failure and ST‐segment elevation of the lateral wall
title_full_unstemmed Hypertrophic cardiomyopathy with heart failure and ST‐segment elevation of the lateral wall
title_short Hypertrophic cardiomyopathy with heart failure and ST‐segment elevation of the lateral wall
title_sort hypertrophic cardiomyopathy with heart failure and st‐segment elevation of the lateral wall
topic Case Reports
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9871721/
https://www.ncbi.nlm.nih.gov/pubmed/36178133
http://dx.doi.org/10.1002/ehf2.14185
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