Concurrent Waldenstrom Macroglobulinemia and Mutant Transthyretin Cardiac Amyloidosis

Cardiac amyloidosis is caused by abnormal deposit of amyloid in the myocardium and can be divided into light chain (AL) amyloidosis and transthyretin (ATTR) amyloidosis. ATTR amyloidosis can be further divided into wild-type and mutant type based on genetic mutation. Differentiation between AL, wild...

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Autores principales: Qian, Xiaoxiao, Bakhshi, Hooman, Biswas, Rakesh, Gattani, Raghav, Kennedy, Jamie L.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Greater Baltimore Medical Center 2022
Materias:
Case Report
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9949727/
https://www.ncbi.nlm.nih.gov/pubmed/36845574
http://dx.doi.org/10.55729/2000-9666.1117
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author Qian, Xiaoxiao
Bakhshi, Hooman
Biswas, Rakesh
Gattani, Raghav
Kennedy, Jamie L.
author_facet Qian, Xiaoxiao
Bakhshi, Hooman
Biswas, Rakesh
Gattani, Raghav
Kennedy, Jamie L.
author_sort Qian, Xiaoxiao
collection PubMed
description Cardiac amyloidosis is caused by abnormal deposit of amyloid in the myocardium and can be divided into light chain (AL) amyloidosis and transthyretin (ATTR) amyloidosis. ATTR amyloidosis can be further divided into wild-type and mutant type based on genetic mutation. Differentiation between AL, wild-type, and mutant type ATTR amyloidosis has significant prognostic and therapeutic implications.
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spelling pubmed-99497272023-02-24 Concurrent Waldenstrom Macroglobulinemia and Mutant Transthyretin Cardiac Amyloidosis Qian, Xiaoxiao Bakhshi, Hooman Biswas, Rakesh Gattani, Raghav Kennedy, Jamie L. J Community Hosp Intern Med Perspect Case Report Cardiac amyloidosis is caused by abnormal deposit of amyloid in the myocardium and can be divided into light chain (AL) amyloidosis and transthyretin (ATTR) amyloidosis. ATTR amyloidosis can be further divided into wild-type and mutant type based on genetic mutation. Differentiation between AL, wild-type, and mutant type ATTR amyloidosis has significant prognostic and therapeutic implications. Greater Baltimore Medical Center 2022-11-07 /pmc/articles/PMC9949727/ /pubmed/36845574 http://dx.doi.org/10.55729/2000-9666.1117 Text en © 2022 Greater Baltimore Medical Center https://creativecommons.org/licenses/by-nc/4.0/This is an open access article under the CC BY-NC license (http://creativecommons.org/licenses/by-nc/4.0/ (https://creativecommons.org/licenses/by-nc/4.0/) )
spellingShingle Case Report
Qian, Xiaoxiao
Bakhshi, Hooman
Biswas, Rakesh
Gattani, Raghav
Kennedy, Jamie L.
Concurrent Waldenstrom Macroglobulinemia and Mutant Transthyretin Cardiac Amyloidosis
title Concurrent Waldenstrom Macroglobulinemia and Mutant Transthyretin Cardiac Amyloidosis
title_full Concurrent Waldenstrom Macroglobulinemia and Mutant Transthyretin Cardiac Amyloidosis
title_fullStr Concurrent Waldenstrom Macroglobulinemia and Mutant Transthyretin Cardiac Amyloidosis
title_full_unstemmed Concurrent Waldenstrom Macroglobulinemia and Mutant Transthyretin Cardiac Amyloidosis
title_short Concurrent Waldenstrom Macroglobulinemia and Mutant Transthyretin Cardiac Amyloidosis
title_sort concurrent waldenstrom macroglobulinemia and mutant transthyretin cardiac amyloidosis
topic Case Report
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9949727/
https://www.ncbi.nlm.nih.gov/pubmed/36845574
http://dx.doi.org/10.55729/2000-9666.1117
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AT gattaniraghav concurrentwaldenstrommacroglobulinemiaandmutanttransthyretincardiacamyloidosis
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