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CFTR—a (novel) target in ARDS: Commentary on “Loss of endothelial CFTR drives barrier failure and edema formation in lung infection and can be targeted by CFTR potentiation”, Erfinanda et al., Sci. Transl. Med. 14, eabg8577 (2022)
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Formato: | Online Artículo Texto |
Lenguaje: | English |
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Springer Berlin Heidelberg
2023
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Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9969384/ https://www.ncbi.nlm.nih.gov/pubmed/36847846 http://dx.doi.org/10.1007/s00424-023-02800-4 |
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author | Frick, Manfred |
author_facet | Frick, Manfred |
author_sort | Frick, Manfred |
collection | PubMed |
description | |
format | Online Article Text |
id | pubmed-9969384 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2023 |
publisher | Springer Berlin Heidelberg |
record_format | MEDLINE/PubMed |
spelling | pubmed-99693842023-02-28 CFTR—a (novel) target in ARDS: Commentary on “Loss of endothelial CFTR drives barrier failure and edema formation in lung infection and can be targeted by CFTR potentiation”, Erfinanda et al., Sci. Transl. Med. 14, eabg8577 (2022) Frick, Manfred Pflugers Arch Commentary Springer Berlin Heidelberg 2023-02-27 2023 /pmc/articles/PMC9969384/ /pubmed/36847846 http://dx.doi.org/10.1007/s00424-023-02800-4 Text en © The Author(s) 2023 https://creativecommons.org/licenses/by/4.0/Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/ (https://creativecommons.org/licenses/by/4.0/) . |
spellingShingle | Commentary Frick, Manfred CFTR—a (novel) target in ARDS: Commentary on “Loss of endothelial CFTR drives barrier failure and edema formation in lung infection and can be targeted by CFTR potentiation”, Erfinanda et al., Sci. Transl. Med. 14, eabg8577 (2022) |
title | CFTR—a (novel) target in ARDS: Commentary on “Loss of endothelial CFTR drives barrier failure and edema formation in lung infection and can be targeted by CFTR potentiation”, Erfinanda et al., Sci. Transl. Med. 14, eabg8577 (2022) |
title_full | CFTR—a (novel) target in ARDS: Commentary on “Loss of endothelial CFTR drives barrier failure and edema formation in lung infection and can be targeted by CFTR potentiation”, Erfinanda et al., Sci. Transl. Med. 14, eabg8577 (2022) |
title_fullStr | CFTR—a (novel) target in ARDS: Commentary on “Loss of endothelial CFTR drives barrier failure and edema formation in lung infection and can be targeted by CFTR potentiation”, Erfinanda et al., Sci. Transl. Med. 14, eabg8577 (2022) |
title_full_unstemmed | CFTR—a (novel) target in ARDS: Commentary on “Loss of endothelial CFTR drives barrier failure and edema formation in lung infection and can be targeted by CFTR potentiation”, Erfinanda et al., Sci. Transl. Med. 14, eabg8577 (2022) |
title_short | CFTR—a (novel) target in ARDS: Commentary on “Loss of endothelial CFTR drives barrier failure and edema formation in lung infection and can be targeted by CFTR potentiation”, Erfinanda et al., Sci. Transl. Med. 14, eabg8577 (2022) |
title_sort | cftr—a (novel) target in ards: commentary on “loss of endothelial cftr drives barrier failure and edema formation in lung infection and can be targeted by cftr potentiation”, erfinanda et al., sci. transl. med. 14, eabg8577 (2022) |
topic | Commentary |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9969384/ https://www.ncbi.nlm.nih.gov/pubmed/36847846 http://dx.doi.org/10.1007/s00424-023-02800-4 |
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