Neutrophil-specific STAT4 deficiency attenuates atherosclerotic burden and improves plaque stability via reduction in neutrophil activation and recruitment into aortas of Ldlr(−/−) mice.

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BACKGROUND AND AIMS: Neutrophils drive atheroprogression and directly contribute to plaque instability. We recently identified signal transducer and activator of transcription 4 (STAT4) as a critical component for bacterial host defense in neutrophils. The STAT4-dependent functions of neutrophils in...

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Detalles Bibliográficos
Autores principales: Keeter, W. Coles, Moriarty, Alina K, Akers, Rachel, Ma, Shelby, Mussbacher, Marion, Nadler, Jerry L., Galkina, Elena V.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Cold Spring Harbor Laboratory 2023
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9980123/
https://www.ncbi.nlm.nih.gov/pubmed/36865098
http://dx.doi.org/10.1101/2023.02.22.529608
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