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  1. 1
    “…Pain, the primary symptom of osteoarthritis (OA), reduces both the quality and quantity of life for patients. …”
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    Mechanisms of Peripheral and Central Sensitization in Osteoarthritis Pain
  2. 2
    “…PURPOSE: Pain from osteoarthritis (OA) is generally classified as nociceptive (inflammatory). …”
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    Existence of a Neuropathic Pain Component in Patients with Osteoarthritis of the Knee
  3. 3
    “…Osteoarthritis (OA) is a chronic degenerative musculoskeletal disease that causes articular cartilage degeneration and chronic pain. …”
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    NGF Expression and Elevation in Hip Osteoarthritis Patients with Pain and Central Sensitization
  4. 4
    “…Further, while there were no differences in the CD14+ fraction between RA and OA, MINCLE expression in the CD14– fraction was elevated in RA compared to OA. …”
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    Elevated macrophage-inducible C-type lectin expression in the synovial tissue of patients with rheumatoid arthritis
  5. 5
    “…BACKGROUND: Patients with persistent pain due to osteoarthritis (OA) complain of multiple symptoms that cannot be explained solely by structural changes. …”
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    Central sensitization inventory scores correlate with pain at rest in patients with hip osteoarthritis: a retrospective study
  6. 6
    “…Calcitonin gene-related peptide (CGRP) is a vasodilatory neuropeptide that is localized to joint tissues and has recently been implicated in the development of knee OA and OA pain. To date, however, the expression levels of CGRP in the IPFP of human knee OA patients have not been examined. …”
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    Expression of calcitonin gene-related peptide in the infrapatellar fat pad in knee osteoarthritis patients
  7. 7
    “…BACKGROUND: While epidemiological studies have reported a potential role for hypercholesterolemia (HCE) in osteoarthritis (OA), the association between HCE and OA has yet to be clarified. …”
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    Down-regulation of microsomal prostaglandin E2 synthase-1 in the infrapatellar fat pad of osteoarthritis patients with hypercholesterolemia
  8. 8
    “…Pregabalin was developed as a pain killer for NP; however, there are no reports on pregabalin use in OA patients. The purpose of this study was to investigate the efficacy of pregabalin for pain in OA patients. …”
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    Efficacy of Combination of Meloxicam and Pregabalin for Pain in Knee Osteoarthritis
  9. 9
    “…To investigate the role of macrophages as a regulator and producer of nerve growth factor (NGF) in the synovial tissue (ST) of osteoarthritis (OA) joints, the gene expression profiles of several inflammatory cytokines in the ST, including synovial macrophages and fibroblasts, of OA mice (STR/Ort) were characterized. …”
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    Nerve Growth Factor Regulation by TNF-α and IL-1β in Synovial Macrophages and Fibroblasts in Osteoarthritic Mice
  10. 10
    “…CONCLUSION: Obese patients with knee OA showed elevated TPSB2 expression in the SM. Tryptase may play a role in synovial inflammation in obese patients with OA.…”
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    Increase in Tryptase and Its Role in the Synovial Membrane of Overweight and Obese Patients with Osteoarthritis of the Knee
  11. 11
    “…BACKGROUND: Recent studies suggest that the vasodilatory neuropeptide calcitonin gene-related peptide (CGRP) is localized in the synovial tissue and may be involved in the pathology of hip and knee osteoarthritis (OA). However, the regulation and relationship between pain and CGRP expression levels in the synovial tissue of human OA patients are not fully understood. …”
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    Increase and regulation of synovial calcitonin gene-related peptide expression in patients with painful knee osteoarthritis
  12. 12
    “…Although recent studies suggest that hyperlipidemia is a risk factor for osteoarthritis (OA), the link between OA and hyperlipidemia is not fully understood. …”
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    Increase of Circulating CD11b(+)Gr1(+) cells and Recruitment into the Synovium in Osteoarthritic Mice with Hyperlipidemia
  13. 13
    “…BACKGROUND: In patients with hip osteoarthritis (OA), pain at rest, unlike pain on activity, is due to pain mechanisms that cannot be explained by nociceptive pain. …”
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    Adverse Effects of Higher Preoperative Pain at Rest, a Central Sensitization-Related Symptom, on Outcomes After Total Hip Arthroplasty in Patients with Osteoarthritis
  14. 14
    “…BACKGROUND: Previous studies suggest the presence of an association of vascular endothelial growth factor (VEGF) with osteoarthritis (OA) severity and pain in patients with knee OA. …”
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    Vascular Endothelial Growth Factor Is Regulated by the Canonical and Noncanonical Transforming Growth Factor-β Pathway in Synovial Fibroblasts Derived from Osteoarthritis Patients
  15. 15
    “…Moreover, it has been reported that pain in knee OA patients is linked to specific fibroblast subsets. …”
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    CD39+CD55− Fb Subset Exhibits Myofibroblast-Like Phenotype and Is Associated with Pain in Osteoarthritis of the Knee
  16. 16
    “…A recent study demonstrated that measuring muscle volume using computed tomography (CT)-based analysis and comparing bilateral muscles in the same patient allowed for accurate evaluation of muscle volume in unilateral hip osteoarthritis (OA) patients. Here, we evaluated muscle volume using CT-based analysis and compared bilateral muscles in knee OA (KOA) patients. …”
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    Unilateral-dominant reduction in muscle volume in female knee osteoarthritis patients: computed tomography-based analysis of bilateral sides
  17. 17
    “…The patients in which OA progression was noted were identified as having radiographical OA preoperatively and acetabular cartilage damage in the arthroscopic findings. …”
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    Clinical results of arthroscopic surgery in patients over 50 years of age—what viability does it have as a joint preservative surgery?
  18. 18
    “…The role of TAK1 in synovial tissue in OA is not fully understood. Using synovial cells harvested from OA patients during surgery, we investigated whether TAK1 inhibition suppresses production of TNF-α-induced extracellular matrix degrading enzymes and expression of pain-related molecules. …”
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    Transforming growth factor activating kinase 1 regulates extracellular matrix degrading enzymes and pain-related molecule expression following tumor necrosis factor-α stimulation of synovial cells: an in vitro study
  19. 19
    “…Synovial inflammation plays a crucial role in the destruction of joints and the experience of pain in osteoarthritis (OA). Emerging evidence suggests that certain antibiotic agents and their derivatives possess anti-inflammatory properties. …”
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    Medermycin Inhibits TNFα-Promoted Inflammatory Reaction in Human Synovial Fibroblasts
  20. 20
    “…BACKGROUND: Femoroacetabular impingement (FAI) is one factor known to cause pain and osteoarthritis (OA) of the hips. Although secondary OA due to hip dysplasia is common among Japanese populations, primary OA is seldom observed. …”
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    Prevalence of radiographic findings of femoroacetabular impingement in the Japanese population
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