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  1. 6541
    “…Despite its importance, the molecular mechanism controlling this tumour metabolic shift remains not fully understood. Here we show that 14-3-3σ regulates cancer metabolic reprogramming and protects cells from tumourigenic transformation. 14-3-3σ opposes tumour-promoting metabolic programs by enhancing c-Myc poly-ubiquitination and subsequent degradation. 14-3-3σ demonstrates the suppressive impact on cancer glycolysis, glutaminolysis, mitochondrial biogenesis and other major metabolic processes of tumours. …”
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    The cell cycle regulator 14-3-3σ opposes and reverses cancer metabolic reprogramming
  2. 6542
    “…We demonstrate that recruitment of DAXX, H3.3, and KAP1 to ERVs are co-dependent and upstream of ESET, linking H3.3 to ERV-associated H3K9me3. …”
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    Histone H3.3 is required for endogenous retroviral element silencing in embryonic stem cells
  3. 6543
    “…POLR3-related (or 4H) leukodystrophy is an autosomal recessive disorder caused by mutations in POLR3A or POLR3B and is characterized by neurological and non-neurological features. …”
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    Large exonic deletions in POLR3B gene cause POLR3-related leukodystrophy
  4. 6544
    “…In this study, Escherichia coli was metabolically engineered to produce 1,3-diaminopropane (1,3-DAP), a monomer for engineering plastics. …”
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    Metabolic engineering of Escherichia coli for the production of 1,3-diaminopropane, a three carbon diamine
  5. 6545
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    Genome-wide analysis of thapsigargin-induced microRNAs and their targets in NIH3T3 cells
  6. 6546
    “…14-3-3 proteins play a pivotal role in controlling cell proliferation and survival, two commonly dysregulated hallmarks of cancers. 14-3-3 protein expression is enhanced in many human cancers and correlates with more aggressive tumors and poor prognosis, suggesting a role for 14-3-3 proteins in tumorigenesis and/or progression. …”
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    Destabilisation of dimeric 14-3-3 proteins as a novel approach to anti-cancer therapeutics
  7. 6547
    “…METHODS: Serum calcium, IGF1, and IGF-binding protein 3 (IGFBP3) were measured for 5368 participants in NHANES III. …”
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    The association between circulating IGF1, IGFBP3, and calcium: results from NHANES III
  8. 6548
    “…RESULTS: The expression level of EPO was highly elevated in EPO-3T3-EGFP cells, and an abundant amount of EPO secreted from EPO-3T3-EGFP cells was detected in the extracellular milieu. …”
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    Erythropoietin produced by genetic-modified NIH/3T3 fibroblasts enhances the survival of degenerating neurons
  9. 6549
    por Goldberg, Amy, Rosenberg, Noah A.
    Publicado 2015
    “…A simple framework noting that in a population with equally many females and males, two-thirds of X chromosomes appear in females, suggests that the mean X-chromosomal admixture fraction is a linear combination of female and male admixture parameters, with coefficients 2/3 and 1/3, respectively. Extending a mechanistic admixture model to accommodate the X chromosome, we demonstrate that this prediction is not generally true in admixture models, although it holds in the limit for an admixture process occurring as a single event. …”
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    Beyond 2/3 and 1/3: The Complex Signatures of Sex-Biased Admixture on the X Chromosome
  10. 6550
    “…Although histone H3 lysine 27 trimethylation (H3K27Me3) is associated with gene silencing, whether H3K27Me3 demethylation affects transcription and cell differentiation in vivo has remained elusive. …”
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    Histone H3 Lysine 27 demethylases Jmjd3 and Utx are required for T-cell differentiation
  11. 6551
    “…Here we show that a subset of cargoes depends on the ArfGAP2/3 Glo3 and ergosterol to maintain their proper localization at the plasma membrane. …”
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    The ArfGAP2/3 Glo3 and ergosterol collaborate in transport of a subset of cargoes
  12. 6552
    “…In vitro, most work has studied brown adipocytes, however, few have investigated more established adipocyte lines such as the murine 3T3-L1 line. To assess the effect of beta-adrenergic activation, mature 3T3-L1s were treated for 6 or 48 hours with or without isoproterenol (10 and 100 μM) following standard differentiation supplemented with thyroid hormone (T3; 1 nM). …”
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    Isoproterenol Increases Uncoupling, Glycolysis, and Markers of Beiging in Mature 3T3-L1 Adipocytes
  13. 6553
    “…Our team previously identified 14-3-3ε protein as a subchondral bone soluble mediator altering cartilage homeostasis. …”
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    The pro-inflammatory cytokine 14-3-3ε is a ligand of CD13 in cartilage
  14. 6554
    por Ji, Xiao-Jun, Ren, Lu-Jing, Huang, He
    Publicado 2015
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    Omega-3 Biotechnology: A Green and Sustainable Process for Omega-3 Fatty Acids Production
  15. 6555
    “…We report our first experience with 3-dimensional video-assisted surgery (3D-VATS). …”
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    Feasibility of 3-dimensional video-assisted thoracic surgery (3D-VATS) for pulmonary resection
  16. 6556
    “…This study aimed at investigating the in vivo antimalarial activities of some 2,3-disubstituted-4(3H)-quinazolinone derivatives. …”
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    In vivo antimalarial evaluation of some 2,3-disubstituted-4(3H)-quinazolinone derivatives
  17. 6557
    “…Bombesin-like receptor 3 (BRS-3) is an X-linked G protein-coupled receptor involved in the regulation of energy homeostasis. …”
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    Search for an Endogenous Bombesin-Like Receptor 3 (BRS-3) Ligand Using Parabiotic Mice
  18. 6558
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    Mini-laparascopic dismembered pyeloplasty using only 3 mm instruments (3 mmML)
  19. 6559
    “…The 14-3-3σ proteins are a family of ubiquitous conserved eukaryotic regulatory molecules involved in the regulation of mitogenic signal transduction, apoptotic cell death, and cell cycle control. …”
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    Revealing the binding modes and the unbinding of 14-3-3σ proteins and inhibitors by computational methods
  20. 6560
    “…One of the most widely studied chromatin modifications is trimethylated lysine 4 of histone 3 (H3K4me3). Here, we conducted the first comprehensive study of H3K4me3 ChIP-sequencing in neuronal chromatin from the prefrontal cortex of six HD cases and six non-neurologic controls, and its association with gene expression measured by RNA-sequencing. …”
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    The Role of H3K4me3 in Transcriptional Regulation Is Altered in Huntington’s Disease
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