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  1. 8281
    por King, William P., Amos, Jennifer, Azer, Magdi, Baker, Daniel, Bashir, Rashid, Best, Catherine, Bethke, Eliot, Boppart, Stephen A., Bralts, Elisabeth, Corey, Ryan M., Dietkus, Rachael, Durack, Gary, Elbel, Stefan, Elliott, Greg, Fava, Jake, Goldenfeld, Nigel, Goldstein, Molly H., Hayes, Courtney, Herndon, Nicole, Jamison, Shandra, Johnson, Blake, Johnson, Harley, Johnson, Mark, Kolaczynski, John, Lee, Tonghun, Maslov, Sergei, McGregor, Davis J., Milner, Derek, Moller, Ralf, Mosley, Jonathan, Musser, Andy, Newberger, Max, Null, David, O’Bryan, Lucas, Oelze, Michael, O’Leary, Jerry, Pagano, Alex, Philpott, Michael, Pianfetti, Brian, Pille, Alex, Pizzuto, Luca, Ricconi, Brian, Rubessa, Marcello, Rylowicz, Sam, Shipley, Clifford, Singer, Andrew C., Stewart, Brian, Switzky, Rachel, Tawfick, Sameh, Wheeler, Matthew, White, Karen, Widloski, Evan M., Wood, Eric, Wood, Charles, Wooldridge, Abigail R.
    Publicado 2020
    “…Many COVID-19 patients experience Acute Respiratory Distress Syndrome (ARDS), a condition that can be treated with mechanical ventilation. …”
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    Emergency ventilator for COVID-19
  2. 8282
    “…Furthermore, acute respiratory distress syndrome (ARDS) and septicemia induce hypoxemia and hypoperfusion, which are locally exacerbated due to the hypercoagulable state and micro-thrombosis in brain vessels, leading to oxidative stress and neurodegeneration. …”
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    Pathophysiological Clues to How the Emergent SARS-CoV-2 Can Potentially Increase the Susceptibility to Neurodegeneration
  3. 8283
    “…The compelling evidences link the progression of viral infection of SARS-CoV-2 with excessive inflammation as a result of the exaggerated immune response and elevated production of “immunocytokines” resulting in cytokine storm (CS); followed by a series of events, like acute organ damage, acute respiratory distress syndrome (ARDS) as well as death. Hence attempts to reduce cytokine storm are now being considered as a new paradigm shift in the clinical management of SARS-CoV-2. …”
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    Immune system response during viral Infections: Immunomodulators, cytokine storm (CS) and Immunotherapeutics in COVID-19
  4. 8284
    “…Based on these encouraging results, the United States Food and Drug Administration (FDA) authorized the compassionate use of MSC, but only in patients with Acute Respiratory Distress Syndrome (ARDS) and a poor prognosis. In fact, patients with severe SARS-CoV-2 can present infection and tissue damage in different organs, such as lung, heart, liver, kidney, gut and brain, affecting their function. …”
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    Mesenchymal stromal cells to fight SARS-CoV-2: Taking advantage of a pleiotropic therapy
  5. 8285
    “…Acute lung injury (ALI), a milder form of acute respiratory distress syndrome (ARDS), is a leading cause of mortality in older adults with an increasing prevalence. …”
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    Alda-1 Attenuates Hyperoxia-Induced Acute Lung Injury in Mice
  6. 8286
    “…The most common SARS-CoV-2 infection related complication was acute respiratory distress syndrome (ARDS) in 95 patients (86.4%). Of the 79 patients (71.8%) who received invasive mechanical ventilation, 46 (58.2%) received neuromuscular blockade injection, and 19 (24.1%) received ECMO treatment. …”
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    Clinical features and prognostic factors of critically ill patients with COVID-19 in Daegu, South Korea: A multi-center retrospective study
  7. 8287
    “…Corticosteroid use was also associated with the development of ARDS, exacerbation of pulmonary fibrosis, longer hospital stay and virus clearance time. …”
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    Adverse Outcomes Associated With Corticosteroid Use in Critical COVID-19: A Retrospective Multicenter Cohort Study
  8. 8288
    “…The angiotensin-converting-enzyme 2 (ACE2), a component of renin–angiotensin-system (RAS); acts on ACE/Ang-II/AT1recptor axis, and regulates pathological processes like hypertension, cardiac dysfunction, Acute Respiratory Distress Syndrome (ARDS) etc. The progression of T2DM and hypertension show decreased expression and activity of ACE2. …”
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    Angiotensin-Converting-Enzyme 2 and Renin-Angiotensin System Inhibitors in COVID-19: An Update
  9. 8289
    “…Coronavirus disease-2019 (COVID-19), a condition caused by SARS-CoV-2, results in acute lung injury (ALI)/acute respiratory distress syndrome (ARDS) associated with high mortality in the elderly and in people with underlying comorbidities. …”
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    SARS-CoV-2 and other coronaviruses negatively influence mitochondrial quality control: beneficial effects of melatonin
  10. 8290
    “…The SARS-CoV-2 infection, known as COVID-19, has various clinical presentations, from asymptomatic or mild catarrhal processes to severe pneumonia that rapidly progresses to acute respiratory distress syndrome (ARDS) and multiple organ failure. In the last few months, much scientific literature has been devoted to descriptions of different aspects of the coagulation disorders and arterial and venous thrombotic complications associated with COVID-19, particularly venous thromboembolism (VTE). …”
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    Coagulation disorders and thromboembolic disease in COVID-19: review of current evidence in search of a better approach
  11. 8291
    “…Following a common viral pneumonia, a multisystem inflammatory syndrome sometimes occurs, including an Acute Respiratory Distress Syndrome (ARDS) carrying a high mortality. Unlike most common respiratory viruses, children seem less susceptible to SARS-CoV-2 infection and generally develop a mild disease with low mortality. …”
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    COVID-19 chez l’enfant : syndrome inflammatoire multi-systémique lié à SARS-CoV-2 mimant un syndrome de Kawasaki()
  12. 8292
    “…However, in some infected individuals, this can progress to severe pneumonia and acute respiratory distress syndrome (ARDS), leading to multi-organ failure and death. …”
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    Proteomic blood profiling in mild, severe and critical COVID-19 patients
  13. 8293
    “…On multivariate analysis, proteinuria greater than 0.3 g/g was related to a higher prevalence of ICU admission [OR = 4.72, IC95 (1.16–23.21), p = 0.03], acute respiratory distress syndrome (ARDS) [OR = 6.89, IC95 (1.41–53.01, p = 0.02)], nosocomial infections [OR = 3.75, IC95 (1.11–13.55), p = 0.03], longer inpatient hospital stay (p = 0.003). …”
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    Proteinuria as a Biomarker for COVID-19 Severity
  14. 8294
    “…Most COVID-19 patients in Singapore who required mechanical ventilation because of ARDS were extubated with no mortality.…”
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    Clinical features and predictors of severity in COVID-19 patients with critical illness in Singapore
  15. 8295
    “…In addition, histopathologic pulmonary lesions such as diffuse alveolar damage, hyaline membrane formation, fibrin deposition, and proteinaceous exudates were observed due to SARS-CoV-2 infection, imitating lesions identified in people hospitalized with ARDS from COVID-19. A significant correlation exists between the degree of clinical disease identified in infected cats and pulmonary lesions. …”
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    Clinicopathologic features of a feline SARS-CoV-2 infection model parallel acute COVID-19 in humans
  16. 8296
    “…Bio-ADM levels increased with the severity of acute respiratory distress syndrome (ARDS; p < 0.001) and were significantly elevated in invasively ventilated patients (p = 0.006) and patients in need of extracorporeal membrane oxygenation (p = 0.040) or renal replacement therapy (RRT; p < 0.001) compared to patients without these conditions. …”
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    Prognostic Value of Bioactive Adrenomedullin in Critically Ill Patients with COVID-19 in Germany: An Observational Cohort Study
  17. 8297
    “…Recent evidence has shown that the upregulation of the WNT/β-catenin pathway is associated with inflammation, resulting in a cytokine storm in ARDS (acute respire distress syndrome) and especially in COVID-19 patients. …”
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    Interplay of Opposing Effects of the WNT/β-Catenin Pathway and PPARγ and Implications for SARS-CoV2 Treatment
  18. 8298
    “…The main complications associated with fatal outcomes in this group of patients were Acute Respiratory Distress Syndrome (ARDS) and sepsis (P < 0.05). Furthermore, it was evidenced that the colchicine combination showed a significant difference in reducing mortality in hospitalized patients compared to the other therapeutic regimens (P < 0.05). …”
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    Clinical outcomes of patients hospitalized for COVID-19 and evidence-based on the pharmacological management reduce mortality in a region of the Colombian Caribbean
  19. 8299
    “…While the majority of those infected will not have long-term pulmonary sequelae, 5%–10% will develop severe COVID-19 pneumonia and acute respiratory distress syndrome (ARDS). The natural history of these severely affected patients is unclear at present, but using our knowledge of closely related coronavirus outbreaks like severe acute respiratory distress syndrome (SARS) and middle east respiratory syndrome (MERS), we would hypothesize that the majority will stabilize or improve over time although some patients will progress to advanced lung fibrosis or post-COVID interstitial lung disease (PC-ILD). …”
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    Post-COVID lung fibrosis: The tsunami that will follow the earthquake
  20. 8300
    “…Use of JAK-inhibitors decreased use of invasive mechanical ventilation (RR = 0.63; [95% Confidence Interval (CI), 0.47, 0.84]; P = 0.002) and had borderline impact on rates of intensive care unit (ICU) admission (RR = 0.24 [0.06, 1.02]; P = 0.05) and acute respiratory distress syndrome (ARDS; RR = 0.50 [0.19, 1.33]; P = 0.16). JAK-inhibitors did not decrease length of hospitalization (mean difference (MD) –0.18 [–4.54, 4.18]; P = 0.94). …”
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    JAK-inhibitors for coronavirus disease-2019 (COVID-19): a meta-analysis
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