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19901por Kollmer, Jennifer, Hund, Ernst, Hornung, Benjamin, Hegenbart, Ute, Schönland, Stefan O., Kimmich, Christoph, Kristen, Arnt V., Purrucker, Jan, Röcken, Christoph, Heiland, Sabine, Bendszus, Martin, Pham, Mirko“…Transthyretin familial amyloid polyneuropathy is a rare, autosomal-dominant inherited multisystem disorder usually manifesting with a rapidly progressive, axonal, distally-symmetric polyneuropathy. The detection of nerve injury by nerve conduction studies is limited, due to preferential involvement of small-fibres in early stages. …”
Publicado 2015
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19902por Lee, Sangmi, Mattingly, Aaron, Lin, Amity, Sacramento, Jeffrey, Mannent, Leda, Castel, Marie-Noelle, Canolle, Benoit, Delbary-Gossart, Sandrine, Ferzaz, Badia, Morganti, Josh M., Rosi, Susanna, Ferguson, Adam R., Manley, Geoffrey T., Bresnahan, Jacqueline C., Beattie, Michael S.“…The p75 neurotrophin receptor (p75NTR) has multiple biological functions, affecting cell survival, apoptotic cell death, axonal growth, and degeneration in pathological conditions. …”
Publicado 2016
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19903por Paterson, Ross W., Slattery, Catherine F., Poole, Teresa, Nicholas, Jennifer M., Magdalinou, Nadia K., Toombs, Jamie, Chapman, Miles D., Lunn, Michael P., Heslegrave, Amanda J., Foiani, Martha S, Weston, Philip S. J., Keshavan, Ashvini, Rohrer, Jonathan D., Rossor, Martin N., Warren, Jason D., Mummery, Catherine J., Blennow, Kaj, Fox, Nick C., Zetterberg, Henrik, Schott, Jonathan M.“…METHODS: We used immunoassays to measure conventional CSF markers of amyloid and tau pathology (amyloid beta (Aβ)1–42, total tau (T-tau), and phosphorylated tau (P-tau)) as well as amyloid processing (AβX-38, AβX-40, AβX-42, soluble amyloid precursor protein (sAPP)α, and sAPPβ), large fibre axonal degeneration (neurofilament light chain (NFL)), and neuroinflammation (YKL-40) in 245 patients with a variety of dementias and 30 controls. …”
Publicado 2018
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19904por Classen, W., Altmann, B., Gretener, P., Souppart, C., Skelton-Stroud, P., Krinke, G.“…The animals had neuronal and secondary axonal damage, most prominent in the cerebellar roof, pontine and vestibular nuclei, and in the raphe/paralemniscal region. …”
Publicado 1999
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19905“…Diabetic neuropathy (DN) is associated with damage to nerve glial cells, their axons, and endothelial cells leading to impaired function and mobility. …”
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19906por Hwang, Charles D., Pagani, Chase A., Lee, Seungyong, Qin, Qizhi, Marini, Simone, Huber, Amanda, Meyers, Carolyn A., Hespe, Geoffrey E., Strong, Amy L., Stepien, David M., Sorkin, Michael, Nunez, Johanna, James, Aaron W., Levi, Benjamin“…Nerves are difficult to capture via axons found in the periphery alone. Thus, we have interrogated from publicly available single-nuclei transcriptomic data of peripheral nerve soma (dorsal root ganglia), injured by physical transection or chemically induced pain. …”
Publicado 2021
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19907“…EMG findings were highly suggestive of acute, severe, postganglionic, axonal lesion of the right lower lumbosacral plexus/right sciatic nerve affecting predominantly tibial division/S1 myotome. …”
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19908“…Neurophysiology in vasculitic neuropathy typically shows evolving multifocal axonal neuropathy with reduced compound muscle action potential amplitudes. …”
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19909“…Nerve conduction studies showed a length-dependent, moderately severe sensory motor axonal peripheral neuropathy, which Neurology agreed was due to a vasculitic process. …”
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