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  1. 1921
    “…However, the potential role of this pathway in the regulation of bone homeostasis remains elusive. Therefore, we studied the role of IL-23R signaling in physiological bone remodeling using IL-23R deficient mice. …”
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    IL-23 receptor deficiency results in lower bone mass via indirect regulation of bone formation
  2. 1922
    “…Bone marrow mesenchymal stem cells (BMSCs) refer to a heterogeneous population of cells with the capacity for self-renewal. …”
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    The effects of locomotion on bone marrow mesenchymal stem cell fate: insight into mechanical regulation and bone formation
  3. 1923
    “…OBJECTIVE: To examine the efficacy and safety of Ilizarov bone transport in the treatment of infected nonunion of long bones. …”
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    Ilizarov bone transport to treat infected nonunion of long bones: a multicenter retrospective cohort study
  4. 1924
    “…It was demonstrated to stop bone destruction related to periodontitis by regulating the RANKL and IL-1β markers level in the blood.…”
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    Effects of curcumin gel on osteoclastogenic bone markers in experimental periodontitis and alveolar bone loss in wistar rats
  5. 1925
    “…The osteogenic potential of bone marrow mesenchymal stem cells (BMSCs) declines dramatically with aging. …”
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    Senolytics improve bone forming potential of bone marrow mesenchymal stem cells from aged mice
  6. 1926
    “…AIM: The aim of this study is to compare allogeneic bone grafts associated with platelet-rich plasma (ALBGs-PRP) to autogenous bone grafts (ATBGs) for alveolar reconstructions in patients with cleft lip and palate (CLP). …”
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    Allogeneic Bone Application in Association with Platelet-Rich Plasma for Alveolar Bone Grafting of Cleft Palate Defects
  7. 1927
    “…CONTEXT: Acromegalic patients have an increased vertebral fracture (VFx) risk due to bone quality reduction, independently of bone mineral density (BMD). …”
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    Spine Bone Texture Assessed by Trabecular Bone Score in Active and Controlled Acromegaly: A Prospective Study
  8. 1928
    “…Current treatments for osteoporosis and other bone degenerative diseases predominately rely on preventing further bone erosion rather than restoring bone mass, as the latter treatments can unintentionally trigger cancer development by undiscriminatingly promoting cell proliferation. …”
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    In vivo characterization of carbon dots–bone interactions: toward the development of bone-specific nanocarriers for drug delivery
  9. 1929
    “…Bone formation and resorption are typically coupled, such that the efficacy of anabolic osteoporosis treatments may be limited by bone destruction. …”
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    Dual targeting of salt inducible kinases and CSF1R uncouples bone formation and bone resorption
  10. 1930
    “…Metastatic bone cancer occurs in every type of cancer but is prevalent in lung, breast, and prostate cancers. …”
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    Bisphosphonate-Based Conjugates and Derivatives as Potential Therapeutic Agents in Osteoporosis, Bone Cancer and Metastatic Bone Cancer
  11. 1931
    “…Receptor activator of NF-κB ligand (RANKL)-binding peptides inhibit bone resorption and were recently shown to activate bone formation. …”
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    The Effects of Receptor Activator of NF-κB Ligand-Binding Peptides on Bone Resorption and Bone Formation
  12. 1932
    “…Background: This study aims to evaluate whether there is a correlation between implant stability, bone density, vital bone formation and implant diameter and length. …”
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    Correlation between Primary, Secondary Stability, Bone Density, Percentage of Vital Bone Formation and Implant Size
  13. 1933
    “…Disturbances in fracture healing (non-union) or even critical size bone defects, where void volume is larger than the self-healing capacity of bone tissue, are great challenges for orthopedic surgeons. …”
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    Strategies to Improve Bone Healing: Innovative Surgical Implants Meet Nano-/Micro-Topography of Bone Scaffolds
  14. 1934
    “…Microbial infections occurring during bone surgical treatment, the cause of osteomyelitis and implant failures, are still an open challenge in orthopedics. …”
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    Medicated Hydroxyapatite/Collagen Hybrid Scaffolds for Bone Regeneration and Local Antimicrobial Therapy to Prevent Bone Infections
  15. 1935
    “…We aimed to assess the prevalence of vitamin D deficiency and bone turnover marker concerning bone mineral density. …”
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    Prevalence of vitamin D inadequacy and bone turnover markers concerning bone mineral density in older persons
  16. 1936
    “…Additionally, the release of the incorporated vancomycin, ceftazidime, lidocaine, and bone morphogenetic protein-2 (BMP-2) was evaluated. …”
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    Biodegradable Antimicrobial Agent/Analgesic/Bone Morphogenetic Protein-Loaded Nanofibrous Fixators for Bone Fracture Repair
  17. 1937
    “…OBJECTIVE: To evaluate the efficacy of a novel decellularized porcine bone xenograft, produced by supercritical carbon dioxide extraction technology, on alveolar socket healing after tooth extraction compared to a commercially available deproteinized bovine bone (Bio‐Oss®). …”
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    Evaluating the bone‐regenerative role of the decellularized porcine bone xenograft in a canine extraction socket model
  18. 1938
    por Liu, Xiangwei, Wang, Binfeng
    Publicado 2021
    “…OBJECTIVE: This study evaluated the beneficial effect of fuscoside in the repair of bone defects (BDs) and the possible molecular mechanism thereof. …”
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    Fuscoside Attenuates Bone Loss in Bone Defects by Regulating The Rankl/Nlrp3/Opg Pathway in Rats
  19. 1939
    “…PURPOSE: To analyze and compare the reactions at the interface between the composite, composed of fragmented heterologous mineralized bone matrix (MOMHF) and polymethylmethacrylate (PMMA), and the rabbit’s tibias, through macroscopic evaluation and scanning electron microscopy (SEM) in different periods. …”
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    Integration of heterologous bone matrix associated with polymethylmethacrylate in induced tibial bone defects. An experimental study in rabbits
  20. 1940
    “…High erythropoietin (Epo) levels are detrimental to bone health in adult organisms. Adult mice receiving high doses of Epo lose bone mass due to suppressed bone formation and increased bone resorption. …”
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    Epo/EpoR signaling in osteoprogenitor cells is essential for bone homeostasis and Epo-induced bone loss
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