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  1. 81
    “…Functionally, PSII-enriched membranes from WT and cia3 show the same photosynthetic activity at optimal pH 6.5. …”
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    The Main Structural and Functional Characteristics of Photosystem-II-Enriched Membranes Isolated from Wild Type and cia3 Mutant Chlamydomonas reinhardtii
  2. 82
    “…Objectives: We would like to clarify the imaging findings of the main tumor that may omit the requirement for lymph node dissection in clinical IA (cIA) lung adenocarcinoma. Methods: A total of 336 patients with cIA lung adenocarcinomas with normal preoperative carcinoembryonic antigen (CEA) who underwent surgical resection were analyzed. …”
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    Selection of Pathological N0 (pN0) in Clinical IA (cIA) Lung Adenocarcinoma by Imaging Findings of the Main Tumor
  3. 83
    “…We investigated arthritis development in male DBA/1 mice which were injected with bovine collagen II (bCII) and human fibrinogen (hFib) on days 0 and 21, leading to stable and reproducible disease induction in 100% of immunized mice (FIA-CIA). In a second study, two bCII—derived peptides were given three times in the course of 6 weeks after FIA-CIA induction to test for impact on arthritis. …”
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    A fructosylated peptide derived from a collagen II T cell epitope for long-term treatment of arthritis (FIA-CIA) in mice
  4. 84
    “…Fiber cross‐sectional area (CSA) was 57% lower in CIA than controls (p < 0.0001), thus smaller but more fibers visible per field of view. …”
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    Unresolved intramuscular inflammation, not diminished skeletal muscle regenerative capacity, is at the root of rheumatoid cachexia: insights from a rat CIA model
  5. 85
    “…We identified unique potentiators of response to ATRi/Chk1i and describe a new role for components of the cytosolic iron–sulfur assembly (CIA) pathway, MMS19 and CIA2B-FAM96B, in replication stress tolerance of TNBC.…”
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    The cytosolic iron–sulfur cluster assembly (CIA) pathway is required for replication stress tolerance of cancer cells to Chk1 and ATR inhibitors
  6. 86
    por Schlesinger, Stephen C.
    Publicado 1982
    Libro
    Fruta amarga : la CIA en Guatemala /
  7. 87
    por Marín Ronzón, José Guadalupe
    Publicado 1988
    Tesis Libro
    Análisis de la efectividad del departamento de relaciones públicas e industriales de la Cía. Nestle de Coatepec, Veracruz
  8. 88
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    Different pattern of synthesis and secretion of IL-1beta in patients with CIAS-1 and TNFRSF1A mutations responding to IL-1 blockade
  9. 89
    por Brenner, Max, Gulko, Pércio S
    Publicado 2012
    “…BACKGROUND: Cia5a is a locus on rat chromosome 10 that regulates disease severity and joint damage in two models of rheumatoid arthritis, collagen- and pristane-induced arthritis (PIA). …”
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    The arthritis severity locus Cia5a regulates the expression of inflammatory mediators including Syk pathway genes and proteases in pristane-induced arthritis
  10. 90
    por Cía, Alfredo Horacio
    Publicado 2002
    Libro
    La ansiedad y sus trastornos : manual diagnóstico y terapéutico /
  11. 91
    “…The biofilm phenotype was reversed by inactivation of CiaRH or by the addition of competence stimulating peptide, the production of which is blocked by CiaRH activity. …”
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    Mutation of the Streptococcus gordonii Thiol-Disulfide Oxidoreductase SdbA Leads to Enhanced Biofilm Formation Mediated by the CiaRH Two-Component Signaling System
  12. 92
    “…Collagen-induced arthritis (CIA) rat is an autoimmune disease model and in many ways shares features with RA. …”
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    Metabolic fingerprinting of joint tissue of collagen-induced arthritis (CIA) rat: In vitro, high resolution NMR (nuclear magnetic resonance) spectroscopy based analysis
  13. 93
    por Cía, Alfredo Horacio
    Publicado 2004
    Libro
    Trastorno de ansiedad social : primera parte, diagnóstico; segunda parte, terapeútica /
  14. 94
    por Dai, Qiaomei, Li, Ji, Yun, Yu, Wang, Jianwei
    Publicado 2018
    “…OBJECTIVE: To investigate the effect of shikonin on (CIA) collagen-induced arthritis and its influence and mechanism on the balance between Th17 cells and Treg cells. …”
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    Toll-Like Receptor 4–Myeloid Differentiation Primary Response Gene 88 Pathway Is Involved in the Shikonin Treatment of CIA by Regulating Treg/Th17 Expression
  15. 95
    “…Using Tepp-46 (100 μM) and SAICAR (4 μM), we demonstrated that PNS (10 μg/mL) inhibited STAT3 phosphorylation and Th17 cell differentiation by suppressing nuclear PKM2 accumulation. In CIA mice, PNS attenuated CIA symptoms, reduced the number of splenic Th17 cells and nuclear PKM2/STAT3 signaling. …”
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    Panax notoginseng saponins (PNS) attenuate Th17 cell differentiation in CIA mice via inhibition of nuclear PKM2-mediated STAT3 phosphorylation
  16. 96
    por Weiner, Tim
    Publicado 2008
    Libro
    Legado de cenizas : la historia de la CIA
  17. 97
    “…Moreover, the nature of the biofilm was assessed by scanning electron microscopy (SEM) and PCR assay for detecting PIA genes, ciaA-D and FnBPA genes. RESULTS: this study showed that applied phages with 0.08 % benezenthonium chloride, for PIA biofilms, and 0.06 % ethanol, for proteinacious FnBPA biofilms, exerted 100 % eradication for MSSA biofilms and about 78 % of MRSA biofilms. …”
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    The potential of bacteriophage cocktail in eliminating Methicillin-resistant Staphylococcus aureus biofilms in terms of different extracellular matrices expressed by PIA, ciaA-D and FnBPA genes
  18. 98
    por Dai, Qiaomei, Li, Ji, Yun, Yu, Wang, Jianwei
    Publicado 2019
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    Corrigendum to “Toll-Like Receptor 4–Myeloid Differentiation Primary Response Gene 88 Pathway Is Involved in the Shikonin Treatment of CIA by Regulating Treg/Th17 Expression”
  19. 99
    por Meitín Casas, Enrique
    Publicado 1984
    Libro
    El sindicalismo libre en América Latina : un engendro de la CIA
  20. 100
    por Marín, Germán
    Publicado 1976
    Libro
    Una historia fantástica y calculada : la CIA en el país de los chilenos
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