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Mostrando 121 - 140 Resultados de 556 Para Buscar '"CONAE"', tiempo de consulta: 0.15s Limitar resultados
  1. 121
    “…Background: Concanavalin A (ConA)-induced liver damage of mice is a well-established murine model mimicking the human autoimmune hepatitis (AIH). …”
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    Identification and characterization of dynamically regulated hepatitis-related genes in a concanavalin A-induced liver injury model
  2. 122
    “…C57BL/6 mice were administered orally with DPPS 6 h before intravenous injection of Concanavalin A (ConA). Liver inflammation, necrosis and hepatocytes apoptosis were determined by histological analyses. …”
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    Hepatoprotective Effect of Mixture of Dipropyl Polysulfides in Concanavalin A-Induced Hepatitis
  3. 123
    “…Moreover, the depletion of Gr-1(+) MDSCs abrogated the protective effect and immune suppression function of GSK872 in ConA-induced IMH. Altogether, our data demonstrate that RIP3 blockade prevents ConA-induced IMH through promoting MDSCs infiltration. …”
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    RIP3 blockade prevents immune-mediated hepatitis through a myeloid-derived suppressor cell dependent mechanism
  4. 124
    “…The results showed that ConA and WGA have the highest binding affinity for E. coli and S. aureus respectively and hence can effectively detect them. …”
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    A lectin-coupled porous silicon-based biosensor: label-free optical detection of bacteria in a real-time mode
  5. 125
    “…Importantly, syndecan-4–deficient (Syn4KO) mice are more susceptible to hepatic injury, and the thrombin-cleaved form of OPN is significantly elevated in Syn4KO mice as compared with wild-type mice after ConA injection. Finally, we demonstrate that administration of purified syndecan-4 protects mice from ConA-induced hepatic injury. …”
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    Syndecan-4 protects against osteopontin-mediated acute hepatic injury by masking functional domains of osteopontin
  6. 126
    “…Here we showed that CD24 deficiency protects mice from concanavalin A (ConA)-induced fulminant liver injury by reducing serum interferon-γ (IFN-γ) levels. …”
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    CD24 aggravates acute liver injury in autoimmune hepatitis by promoting IFN-γ production by CD4(+) T cells
  7. 127
    “…CONCLUSION: This study showed that fucosterol could alleviate acute liver injury induced by ConA by inhibiting P38 MAPK/PPARγ/NF-κB signaling. …”
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    Fucosterol Protects against Concanavalin A-Induced Acute Liver Injury: Focus on P38 MAPK/NF-κB Pathway Activity
  8. 128
    “…The present study investigated the effects of oral administration of AP-CF on ConA injection in mice. The results demonstrated that increases in serum alanine transaminase (ALT) levels after ConA injection were significantly suppressed in an AP-CF administered group of mice. …”
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    The effects of oral administration of Aureobasidium pullulans-cultured fluid containing β-glucan on concanavalin A injected mice
  9. 129
    “…The major secreted protein Msp1 is glycosylated with ConA reactive sugars at the serine residues at 106 and 107. …”
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    The major secreted protein Msp1/p75 is O-glycosylated in Lactobacillus rhamnosus GG
  10. 130
    “…In this study, the role of miR‐193a/b‐3p in concanavalin A (ConA)‐induced liver fibrosis in mice was evaluated. …”
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    miR‐193a/b‐3p relieves hepatic fibrosis and restrains proliferation and activation of hepatic stellate cells
  11. 131
    “…In this study, our data show that purinergic receptor P2Y12 (P2RY12) is highly expressed in concanavalin A (ConA)-induced immune hepatitis mouse model and serves as a potential metabolic regulator in promoting metabolic reprogramming from oxidative phosphorylation to glycolysis in T cells. …”
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    Purinergic receptor P2Y12 boosts autoimmune hepatitis through hexokinase 2-dependent glycolysis in T cells
  12. 132
    “…ConA-stimulated LNC or Sn and PA cooperated in inducing caspase-3-dependent RIN cell apoptosis. …”
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    T cells cooperate with palmitic acid in induction of beta cell apoptosis
  13. 133
    “…C57BL/6 mice were pretreated with galangin followed by ConA challenge. Results indicated that galangin inhibited ConA-induced liver damage. …”
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    Protective effect of galangin in Concanavalin A-induced hepatitis in mice
  14. 134
    “…While the mild iron deficiency induced by the IrDef diet did not significantly modify the steady state immune cell repertoire and systemic cytokine levels, it significantly dampened inflammatory liver damage after ConA challenge. These findings were associated with a marked decrease in T cell and NKT cell activation following ConA injection in IrDef mice. …”
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    Iron Deficiency Impairs Intra-Hepatic Lymphocyte Mediated Immune Response
  15. 135
    “…The release of IL-2 by ConA-stimulated lymphocytes was suppressed by post-treatment with ATP, as well as by pretreatment. …”
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    Adenine Nucleotides Attenuate Murine T Cell Activation Induced by Concanavalin A or T Cell Receptor Stimulation
  16. 136
    “…ConBol, ConBr and ConM showed inhibitory activity on S. mutans growth. All lectins, except ConA, stimulated significantly the growth of S. oralis. …”
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    Effect of Lectins from Diocleinae Subtribe against Oral Streptococci
  17. 137
    “…Transient gene silencing of MT1-MMP prevented the induction of SNAIL by ConA and abrogated TGF-β-induced cell chemotaxis. …”
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    MT1-MMP Cooperates with TGF-β Receptor-Mediated Signaling to Trigger SNAIL and Induce Epithelial-to-Mesenchymal-like Transition in U87 Glioblastoma Cells
  18. 138
    por Kauscher, Ulrike, Ravoo, Bart Jan
    Publicado 2012
    “…In this article, we investigate the multivalent interaction of the lectin concanavalin A (ConA) with cyclodextrin vesicles decorated with mannose–adamantane conjugates with one, two or three adamantane units as well as one or two mannose units. …”
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    Mannose-decorated cyclodextrin vesicles: The interplay of multivalency and surface density in lectin–carbohydrate recognition
  19. 139
    “…Our results demonstrate that NAC can alleviate ConA-induced hepatitis by regulating the PI3K/Akt pathway and reducing the late stages of autophagy. …”
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    Protective Effects of N-Acetylcysteine in Concanavalin A-Induced Hepatitis in Mice
  20. 140
    “…This was followed by the binding of ConA-GOx-AuNPs to the H3N2 virus through the ConA-glycan interaction. …”
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    Functionalized magnetic microparticle-based colorimetric platform for influenza A virus detection
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