Mostrando 1 - 20 Resultados de 334 Para Buscar '"E-Type"', tiempo de consulta: 2.01s Limitar resultados
  1. 1
    por Archier, Georges, Sérieyx, Hervé
    Publicado 1986
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    Pilotes du 3e type
  2. 2
    por Antonucci, Flavia, Bozzi, Yuri
    Publicado 2023
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    Action of Botulinum Neurotoxin E Type in Experimental Epilepsies
  3. 3
    “…The large variety of biological functions governed by prostaglandin (PG) E(2) is mediated by signaling through four distinct E-type prostanoid (EP) receptors. The availability of mouse strains with genetic ablation of each EP receptor subtype and the development of selective EP agonists and antagonists have tremendously advanced our understanding of PGE(2) as a physiologically and clinically relevant mediator. …”
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    E-type prostanoid receptor 4 (EP4) in disease and therapy
  4. 4
    “…PGE(2) has pleiotropic actions depending on the activation of specific E-type prostanoid EP1-4 receptors. We investigated the involvement of PGE(2) and EP receptors in glial activation in response to an inflammatory challenge induced by LPS. …”
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    Differential expression of E-type prostanoid receptors 2 and 4 in microglia stimulated with lipopolysaccharide
  5. 5
    “…Here, we comprehensively profiled IgE type kappa MM on a genomic and immune repertoire level by integrating DNA- and single-cell RNA sequencing and comparative profiling against non-IgE type MM samples. …”
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    IgE type multiple myeloma exhibits hypermutated phenotype and tumor reactive T cells
  6. 6
    Publicado 1985
    “…Light-evoked K+o changes in the rabbit (E-type) retina resemble those previously described in amphibian (I-type) retinas. …”
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    Extracellular K+ activity changes related to electroretinogram components. II. Rabbit (E-type) retinas
  7. 7
    por Salas, Rosa Ana, Pleite, Jorge
    Publicado 2014
    “…We demonstrate that using a 2D section of the real core the results obtained are similar to the real ones, which solves the problem of convergence that appeared when E type cores were simulated in 3D, while also saving computational cost. …”
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    Nonlinear Modeling of E-Type Ferrite Inductors Using Finite Element Analysis in 2D
  8. 8
    “…However, how VP neuron populations with distinct electrophysiological properties (e-types) represent these variables is not fully understood. …”
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    Differential recruitment of ventral pallidal e-types by behaviorally salient stimuli during Pavlovian conditioning
  9. 9
    por Šprajc, Ivan
    Publicado 2021
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    Astronomical aspects of Group E-type complexes and implications for understanding ancient Maya architecture and urban planning
  10. 10
    “…These results highlight a new role for E-type cyclins as important regulators of male meiosis.…”
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    Mammalian E-type Cyclins Control Chromosome Pairing, Telomere Stability and CDK2 Localization in Male Meiosis
  11. 11
    Publicado 2014
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    Correction: Mammalian E-type Cyclins Control Chromosome Pairing, Telomere Stability and CDK2 Localization in Male Meiosis
  12. 12
    “…Interestingly, further antagonizing the E-type prostaglandin receptor EP3 removed the remaining contractile activity, resulting in relaxation evoked by PGI(2) in such vessels of TP(−/−) mice. …”
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    Role of E-type prostaglandin receptor EP3 in the vasoconstrictor activity evoked by prostacyclin in thromboxane-prostanoid receptor deficient mice
  13. 13
    “…Loss of Dek504 function leads to delayed embryogenesis and endosperm development, which produce small and collapsed kernels. Dek504 encodes an E+-type PPR protein targeted to the mitochondria, which is required for RNA editing of mitochondrial NADH dehydrogenase 3 at the nad3-317 and nad3-44 sites. …”
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    Dek504 Encodes a Mitochondrion-Targeted E+-Type Pentatricopeptide Repeat Protein Essential for RNA Editing and Seed Development in Maize
  14. 14
    “…Both GRP23 and NUWA interact with the atypical PPR-DYWs, suggesting that the PPR-E proteins recruit MEF8/MEF8S, whereas the PPR-E+ proteins specifically recruit DYW2 as the trans deaminase, and then GRP23, NUWA, and MORFs facilitate and/or stabilize the E or E+-type editosome formation.…”
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    GRP23 plays a core role in E-type editosomes via interacting with MORFs and atypical PPR-DYWs in Arabidopsis mitochondria
  15. 15
    por Rong, Ye-Jing, Lu, Xin-Jiang, Chen, Jiong
    Publicado 2016
    “…Prostaglandin E2 (PGE2) plays an important role in a broad spectrum of physiological and pathological processes by interacting with E-type prostanoid receptors (EPs). EP4 is one of four EP subtypes known to mediate the immune response in mammalian monocytes/macrophages. …”
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    Molecular Characterization of E-Type Prostanoid Receptor 4 (EP4) from Ayu (Plecoglossus altivelis) and Its Functional Analysis in the Monocytes/Macrophages
  16. 16
    “…Prostaglandin E2 plays an important role in carcinogenesis and malignant potential of prostate cancer (PC) cells by binding to its specific receptors, E-type prostanoid (EP) receptors. However, anti-carcinogenic effects of the EP receptor antagonist are unclear. …”
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    Oral administration of E-type prostanoid (EP) 1 receptor antagonist suppresses carcinogenesis and development of prostate cancer via upregulation of apoptosis in an animal model
  17. 17
    “…BACKGROUND: E7046 is a highly selective, small-molecule antagonist of the E-type prostanoid receptor 4 (EP4) for prostaglandin E2, an immunosuppressive mediator of the tumor immune microenvironment. …”
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    First-in-human phase I study of immunomodulatory E7046, an antagonist of PGE(2)-receptor E-type 4 (EP4), in patients with advanced cancers
  18. 18
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    A Gene Circuit Combining the Endogenous I-E Type CRISPR-Cas System and a Light Sensor to Produce Poly-β-Hydroxybutyric Acid Efficiently
  19. 19
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    Differential roles of prostaglandin E-type receptors in activation of hypoxia-inducible factor 1 by prostaglandin E(1) in vascular-derived cells under non-hypoxic conditions
  20. 20
    “…A deep intronic mutation in the MTRR gene, c.903+469T>C, is the most frequent mutation causing the cblE type of homocystinuria. It is well known to be associated with pre-mRNA missplicing, resulting in pseudoexon inclusion; however, the pathological mechanism remains unknown. …”
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    The Deep Intronic c.903+469T>C Mutation in the MTRR Gene Creates an SF2/ASF Binding Exonic Splicing Enhancer, Which Leads to Pseudoexon Activation and Causes the cblE Type of Homocystinuria
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