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  1. 2581
    “…METHODS: We evaluated four patients from the GLAD study (mean age 39years; 2 female, 3 AA IDH-mutant, 1 GBM IDH-wildtype) who underwent MRS at baseline and following eight weeks of KDT. …”
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    BIMG-23. SINGLE-VOXEL VERSUS MULTI-SLICE MRSI IN PATIENTS WITH GLIOMA ON A KETOGENIC DIET INTERVENTION
  2. 2582
    “…Furthermore, ALK, CDKN2A, MAP2K1, IDH2 and PTEN might involve in the formation of immune phenotypes.…”
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    Tumor Mutation Burden and Differentially Mutated Genes Among Immune Phenotypes in Patients with Lung Adenocarcinoma
  3. 2583
    “…A multivariable prediction model was developed as follows: Y(ME) = 0.672 + 0.513X(Grade) + 0.380X(SVZ) + 0.439X(MGMT) + 0.320X(IDH) + 0.333X(1p/19q). The R-square value of goodness of fit was 0.780. …”
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    Determining optimal clinical target volume margins in high-grade glioma based on microscopic tumor extension and magnetic resonance imaging
  4. 2584
    “…Assessment parameters included Pfirrmann grade, intervertebral disc height (IDH), and disc protrusion size (DPS), as measured by MRI to evaluate the morphological changes. …”
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    Selective Retention of Bone Marrow Stromal Cells with Gelatin Sponge for Repair of Intervertebral Disc Defects after Microendoscopic Discectomy: A Prospective Controlled Study and 2-Year Follow-Up
  5. 2585
    “…High risk scores were associated with an age of > 40, wild-type IDH1, a WHO grade of III, an unmethylated MGMT promoter, and 1p/19q non-codeletion. …”
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    Identification of Iron Metabolism-Related Genes as Prognostic Indicators for Lower-Grade Glioma
  6. 2586
    “…METHODS: We applied immunohistochemistry in tumor tissue sections of 18 high-grade glioma (HGG) patients (4 anaplastic astrocytoma, IDH-wildtype WHO-III; 14 glioblastomas (GBM), IDH-wildtype WHO-IV) in order to assess and quantify leucocytes (CD45) and macrophages (CD68, CD163) within the tumor core, infiltration zone and perivascular spaces. …”
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    Cerebrospinal fluid cytokine levels are associated with macrophage infiltration into tumor tissues of glioma patients
  7. 2587
    “…Correlation analysis showed that HOXD10 expression was significantly related to IDH1 status. Univariate analysis revealed that low HOXD10 expression, complete surgical resection, postoperative radiotherapy, postoperative temozolomide chemotherapy and IDH1 mutation were all beneficial prognostic factors. …”
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    Identification of HOXD10 as a Marker of Poor Prognosis in Glioblastoma Multiforme
  8. 2588
    “…At the molecular analysis, mutations on IDH and H3F3A genes were absent, whereas MGMT promoter was unmethylated. …”
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    Diagnostic challenges of an incidental finding: case report of definitely-congenital glioblastoma multiforme in a very preterm infant
  9. 2589
    “…A detailed determination was successful for IDH 1/2 mutation in 99.4% of cases, for 1p/19q codeletion in 97.4% of cases, for TERT mutation in 98.9% of cases, and for MGMT promoter methylation in 99.1% of cases. …”
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    Diagnostic Yield and Complication Rate of Stereotactic Biopsies in Precision Medicine of Gliomas
  10. 2590
    “…Expression of tRFdb-3012a/b was correlated with IDH mutant status and MGMT promoter mutation in gliomas, and tRFdb-3012a/b and ts-23 tended to be highly expressed in patients with the IDH mutant. …”
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    Identification of Novel tRNA-Leu-CAA-Derived tsRNAs for the Diagnosis and Prognosis of Diffuse Gliomas
  11. 2591
    “…The following molecular markers were analyzed: EGFR amplification, TERT promoter mutation, CDKN2A/B homozygous loss, chromosome 7 + /10- aneuploidy, MGMT methylation, IDH mutation, ATRX loss, p53 mutation and 1p19q codeletion. …”
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    The impact of heme biosynthesis regulation on glioma aggressiveness: Correlations with diagnostic molecular markers
  12. 2592
    “…To this end, we retrospectively reviewed 38 patients with pathologically confirmed glioblastoma (IDH wild-type) and suspected tumor recurrence after radiotherapy. …”
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    Sequential and Hybrid PET/MRI Acquisition in Follow-Up Examination of Glioblastoma Show Similar Diagnostic Performance
  13. 2593
    “…BACKGROUND: Gliomas with FGFR3::TACC3 fusion mainly occur in adults, display pathological features of glioblastomas (GB) and are usually classified as glioblastoma, IDH-wildtype. However, cases demonstrating pathological features of low-grade glioma (LGG) lead to difficulties in classification and clinical management. …”
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    Clinico-pathological and epigenetic heterogeneity of diffuse gliomas with FGFR3::TACC3 fusion
  14. 2594
    “…The molecular characterization of 2856 samples (1631 derived from the NGS–AML project; NCT03311815) with standardized NGS of consensus genes (ABL1, ASXL1, BRAF, CALR, CBL, CEBPA, CSF3R, DNMT3A, ETV6, EZH2, FLT3, GATA2, HRAS, IDH1, IDH2, JAK2, KIT, KRAS, MPL, NPM1, NRAS, PTPN11, RUNX1, SETBP1, SF3B1, SRSF2, TET2, TP53, U2AF1 and WT1) showed 97% of patients having at least one mutation. …”
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    Molecular Landscape and Validation of New Genomic Classification in 2668 Adult AML Patients: Real Life Data from the PETHEMA Registry
  15. 2595
    Publicado 2013
    Tabla de Contenidos: “…Condiciones sociales de la población indígena e inversión federal en los 125 municipios con menor IDH / Jesús Mena Vázquez -- Cap. 5. Incidencia delictiva en los 125 municipios más marginados del país / Juan Pablo Aguirre Quezada.…”
    Libro
    Los 125 municipios con menor índice de desarrollo humano en México
  16. 2596
    por Guo, Xu, Wang, Gang
    Publicado 2023
    “…BACKGROUND: Glioma is the most common primary intracranial malignancy in clinical practice, and in particular, IDH‐wildtype glioblastoma has the worst prognosis. …”
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    Advances in research on immune escape mechanism of glioma
  17. 2597
    por Aggarwal, Surinder K.
    Publicado 1998
    “…In an attempt to elucidate its mechanism(s) of action, liver and kidney tissues from normal and CDDP treated (1.8 mg/kg) dogs were evaluated for changes in various dehydrogenases [MDH, SDH, β-HBDH, IDH and G-6-PDH] and nonspecific lipase enzymes. …”
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    Calcium Modulation of Toxicities Due to Cisplatin
  18. 2598
    por Cardaci, Simone, Ciriolo, Maria Rosa
    Publicado 2012
    “…In the last ten years, a causal role in carcinogenesis has been documented for inherited and acquired alterations in three TCA cycle enzymes, succinate dehydrogenase (SDH), fumarate hydratase (FH), and isocitrate dehydrogenase (IDH), pointing towards metabolic alterations as the underlying hallmark of cancer. …”
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    TCA Cycle Defects and Cancer: When Metabolism Tunes Redox State
  19. 2599
    “…Protein structure modeling showed that somatic mutations in IDH2, RASGEF1B, and MSH4 can affect protein structures.…”
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    Bioinformatics Interpretation of Exome Sequencing: Blood Cancer
  20. 2600
    “…Third, genetic disruptions directly within the core epigenetic machinery, exemplified by the recently identified mutations within isocitrate dehydrogenase genes IDH1/2 and variant histone genes H3.3/H3F3A. These constitute the ‘good, the bad and the ugly’ of epigenetic mechanisms in cancer.…”
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    The good, the bad and the ugly: Epigenetic mechanisms in glioblastoma
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