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  1. 1381
    “…Moreover, these robust features were able to differentiate patients with IDH-wildtype glioma from those with an IDH mutation. …”
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    Online Artículo Texto
    Evaluation of FET PET Radiomics Feature Repeatability in Glioma Patients
  2. 1382
    “…While NPM1 variant allele frequency decreased below 2.5% in remission in all patients, IDH1/2 mutations (typically IDH2 R140Q) persisted in 24% of cases. …”
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    Online Artículo Texto
    Nucleophosmin1 and isocitrate dehydrogenase 1 and 2 as measurable residual disease markers in acute myeloid leukemia
  3. 1383
    “…FREM2 pathway activation level was also a poor prognosis biomarker for OS and PFS in LGG with IDH mutation, for PFS in LGG with wild type IDH and mutant IDH with 1p/19q codeletion, in GBM with unmethylated MGMT, and in GBM with wild type IDH. …”
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    Algorithmically Deduced FREM2 Molecular Pathway Is a Potent Grade and Survival Biomarker of Human Gliomas
  4. 1384
    “…BACKGROUND: Conventionally, identifying isocitrate dehydrogenase (IDH) mutation in gliomas is based on histopathological analysis of tissue specimens acquired via stereotactic biopsy or definitive resection. …”
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    Deep learning for prediction of isocitrate dehydrogenase mutation in gliomas: a critical approach, systematic review and meta-analysis of the diagnostic test performance using a Bayesian approach
  5. 1385
    “…Additionally, our sc-ATAC-seq analyses revealed differences in regulatory networks in NGBs, IDH-mut and IDH-wt glioma associated immune cells. …”
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    OTME-23. Single-cell transcriptomic and epigenomic immune landscape of isocitrate dehydrogenase stratified human gliomas
  6. 1386
    “…In one case, the primary tumor was diffuse astrocytoma with IDH2 mutant, and the secondary tumor was glioblastoma with IDH 1/2 wild-type. …”
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    Assessment of therapeutic outcome and role of reirradiation in patients with radiation-induced glioma
  7. 1387
    “…In this system, IDH activity has been found to be remarkably robust with respect to wide variations in the total IDH protein concentration. …”
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    Robustness in Glyoxylate Bypass Regulation
  8. 1388
    “…Approximately 20% of unselected cases and 30% cytogenetically diploid cases of acute myeloid leukemia (AML) and 80% of grade II–III gliomas and secondary glioblastomas carry mutations in the isocitrate dehydrogenase (IDH) 1 and 2 genes. IDH1/2 mutations prevent oxidative decarboxylation of isocitrate to α-ketoglutarate (α-KG) and modulate the function of IDH (neomorphic activity) thereby facilitating reduction of α-KG to D-2-hydroxyglutarate (D-2HG), a putative oncometabolite. …”
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    The Emerging Role of D-2-Hydroxyglutarate as an Oncometabolite in Hematolymphoid and Central Nervous System Neoplasms
  9. 1389
    “…NAD(+)-dependent Isocitrate Dehydrogenase (NAD(+)-IDH) could be one of the cell phone radiation targets. …”
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    Mobile phones electromagnetic radiation and NAD(+)-dependent isocitrate dehydrogenase as a mitochondrial marker in asthenozoospermia
  10. 1390
    “…However, recent DS reports in trials of isocitrate dehydrogenase (IDH)-inhibitor drugs in patients with IDH-mutated AML have raised concerns. …”
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    Incidence of Differentiation Syndrome Associated with Treatment Regimens in Acute Myeloid Leukemia: A Systematic Review of the Literature
  11. 1391
    “…Background and Objectives: Intradialytic hypotension (IDH) complicates 4 to 39.9% of hemodialysis (HD) sessions. …”
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    Circumferential Strain as a Marker of Vessel Reactivity in Patients with Intradialytic Hypotension
  12. 1392
    “…Provider-level and patient-level decisions influence the occurrence of IDH; thus, IDH may be preventable in routine hemodialysis care. …”
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    Enhancing the Cardiovascular Safety of Hemodialysis Care Using Multimodal Provider Education and Patient Activation Interventions: Protocol for a Cluster Randomized Controlled Trial
  13. 1393
    “…We evaluated the performance of an exemplar deep learning model for differentiating (1) isocitrate dehydrogenase (IDH)-mutation versus IDH-wildtype; (2) 1p/19q codeletion versus 1p/19q non-codeletion; and (3) IDH-mutation with or without 1p/19q codeletion, and IDH-wildtype. …”
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    Combining Multi-Shell Diffusion with Conventional MRI Improves Molecular Diagnosis of Diffuse Gliomas with Deep Learning
  14. 1394
    “…Thirty-eight patients with confirmed non-enhancing glioma were categorized into three subtypes: Oligo (IDH-mut&1p/19q-codeleted), Astro (IDH-mut), and GBM (IDH-wt). …”
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    Microvasculature Features Derived from Hybrid EPI MRI in Non-Enhancing Adult-Type Diffuse Glioma Subtypes
  15. 1395
    “…Secondary outcomes included symptomatic IDH rate and time to recover post hemodialysis. …”
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    A Randomized Trial Examining the Impact of Timing of Intradialytic Cycling on Intradialytic Hypotension
  16. 1396
    “…TERT promoter (TERTp) mutations are found in the majority of World Health Organization (WHO) grade IV adult IDH wild-type glioblastoma (IDH-wt GBM). Here, we characterized the subset of IDH-wt GBMs that do not have TERTp mutations. …”
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    TERT promoter wild-type glioblastomas show distinct clinical features and frequent PI3K pathway mutations
  17. 1397
    “…Moreover, IDH mutation and 2-HG production inhibit GABA oxidation in glioma cells. …”
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    GABA, glutamine, glutamate oxidation and succinic semialdehyde dehydrogenase expression in human gliomas
  18. 1398
    “…SIMPLE SUMMARY: In acute myeloid leukemia (AML) TET2 mutations have been observed to be mutually exclusive with IDH1, IDH2, and WT1 mutations, all of them showing a similar impact on the transcription profile. …”
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    The Potential Equivalents of TET2 Mutations
  19. 1399
    “…The effective cardiovascular risk linked to IDH is debated. HYPOTHESIS: IDH might contribute marginally to hypertension‐related target organ damage (TOD) development. …”
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    Isolated diastolic hypertension and target organ damage: Findings from the STANISLAS cohort
  20. 1400
    “…Similar analysis in the IDH wild type (IDH wt.) subgroup revealed 20 differentially regulated proteins. …”
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    Deep proteome investigation of high-grade gliomas reveals heterogeneity driving differential metabolism of 5-aminolevulinic acid
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