-
261por Terekhanova, Nadezhda V., Seplyarskiy, Vladimir B., Soldatov, Ruslan A., Bazykin, Georgii A.Enlace del recurso
Publicado 2017
Enlace del recurso
Enlace del recurso
Online Artículo Texto -
262por Smith, Oliver, Palmer, Sarah A., Clapham, Alan J., Rose, Pamela, Liu, Yuan, Wang, Jun, Allaby, Robin G.Enlace del recurso
Publicado 2017
Enlace del recurso
Enlace del recurso
Online Artículo Texto -
263por Wymant, Chris, Hall, Matthew, Ratmann, Oliver, Bonsall, David, Golubchik, Tanya, de Cesare, Mariateresa, Gall, Astrid, Cornelissen, Marion, Fraser, ChristopheEnlace del recurso
Publicado 2018
Enlace del recurso
Enlace del recurso
Online Artículo Texto -
264
-
265
-
266por Morgado, Lionel, Preite, Veronica, Oplaat, Carla, Anava, Sarit, Ferreira de Carvalho, Julie, Rechavi, Oded, Johannes, Frank, Verhoeven, Koen J.F.Enlace del recurso
Publicado 2017
Enlace del recurso
Enlace del recurso
Online Artículo Texto -
267
-
268por Porath, Hagit T., Schaffer, Amos A., Kaniewska, Paulina, Alon, Shahar, Eisenberg, Eli, Rosenthal, Joshua, Levanon, Erez Y., Levy, OrenEnlace del recurso
Publicado 2017
Enlace del recurso
Enlace del recurso
Online Artículo Texto -
269
-
270por Parmar, Bhaval, Verma, Urja, Khaire, Kashmira, Danes, Dhanush, Balakrishnan, Suresh“…The compromised level of cell adhesion molecules and their upstream regulators, namely CDH1 (E-cadherin), CDH2 (N-cadherin), MSX1 (Msh homeobox 1), and TGF-β (Transforming growth factor beta), observed in the etoricoxib-treated embryos indicate that COX-2, through its downstream effector PGE(2), regulates the expression of these factors perhaps to aid the migration of CNCCs. …”
Publicado 2021
Enlace del recurso
Enlace del recurso
Enlace del recurso
Online Artículo Texto -
271por Varma, Ashish Ramesh, Meshram, Revat Jagdish, Varma, Anuj Ramesh, Dixit, Anubhuti Sunil, Zabak, Siddhart Sunil, Kulkarni, Chaitanya Ajay“…The MSX homeobox genes cause Goldenhar syndrome (GHS) or facio-auriculo-vertebral dysplasia, a rare developmental defect. …”
Publicado 2021
Enlace del recurso
Enlace del recurso
Enlace del recurso
Online Artículo Texto -
272por Nihashi, Yuma, Miyoshi, Mana, Umezawa, Koji, Shimosato, Takeshi, Takaya, Tomohide“…An 18-base ODN, iSN40, was identified to enhance alkaline phosphatase activity of osteoblasts within 48 h. iSN40 also induced the expression of osteogenic genes such as Msx2, osterix, collagen type 1α, osteopontin, and osteocalcin. …”
Publicado 2022
Enlace del recurso
Enlace del recurso
Enlace del recurso
Online Artículo Texto -
273por Brunello, Giulia, Zanotti, Federica, Scortecci, Gerard, Sapoznikov, Lari, Sivolella, Stefano, Zavan, Barbara“…DPSC differentiation into odontoblast-like cells was identified from the upregulation of odontogenic markers (DSPP and MSX) and osteogenic markers (RUNX2, alkaline phosphatase, osteonectin, osteocalcin, collagen type I, bmp2, smad5/8). …”
Publicado 2022
Enlace del recurso
Enlace del recurso
Enlace del recurso
Online Artículo Texto -
274por Biedziak, Barbara, Firlej, Ewa, Dąbrowska, Justyna, Bogdanowicz, Agnieszka, Zadurska, Małgorzata, Mostowska, Adrianna“…Additionally, our results support the importance of already known ns-TA candidate genes (AXIN2, EDA, EDAR, IRF6, LAMA3, LRP6, MSX1, PAX9 and WNT10A) and provide additional evidence that ns-TA might be an oligogenic condition involving the cumulative effect of rare variants in two or more distinct genes.…”
Publicado 2022
Enlace del recurso
Enlace del recurso
Enlace del recurso
Online Artículo Texto -
275por Safari, Shiva, Ebadifar, Asghar, Najmabadi, Hossien, Kamali, Koorosh, Abedini, Seyedeh Sedigheh, Mousavi, Mohammad“…Nevertheless, the main causes are still unknown. Mutations in PAX9, MSX1, WNT10A, and AXIN2 genes are most commonly associated with non-syndromic tooth agenesis in the literature. …”
Publicado 2022
Enlace del recurso
Enlace del recurso
Enlace del recurso
Online Artículo Texto -
276por Yang, Xuehui, Kilgallen, Sean, Andreeva, Viktoria, Spicer, Douglas B, Pinz, Ilka, Friesel, Robert“…In addition, the domains of expression of several key transcription factors important to normal craniofacial and cardiac development including AP2, Msx2, Dlx5, and Dlx6 were reduced in Spry1;Wnt1-Cre transgenic embryos. …”
Publicado 2010
Enlace del recurso
Enlace del recurso
Enlace del recurso
Texto -
277“…While humans have lost only the Msx3 gene, mice have lost Ventx, Argfx, Dprx, Shox, Rax2, LOC647589, Tprx1 and Nanognb. …”
Enlace del recurso
Enlace del recurso
Enlace del recurso
Online Artículo Texto -
278por Deprez, Pierre M. L., Nichane, Miloud G., Lengelé, Benoît G., Rezsöhazy, René, Nyssen-Behets, Catherine“…Using protein immunodetection and histological techniques comparing transgenic mice to controls, we show here that the persistent expression of Hoxa2 in chondrogenic territories provokes a general down-regulation of the main factors controlling the differentiation cascade, such as Bapx1, Bmp7, Bmpr1a, Ihh, Msx1, Pax9, Sox6, Sox9 and Wnt5a. These data confirm the impairment of chondrogenic differentiation by Hoxa2 overexpression. …”
Publicado 2013
Enlace del recurso
Enlace del recurso
Enlace del recurso
Online Artículo Texto -
279por De Cecco, Loris, Negri, Tiziana, Brich, Silvia, Mauro, Valentina, Bozzi, Fabio, Dagrada, GianPaolo, Disciglio, Vittoria, Sanfilippo, Roberta, Gronchi, Alessandro, D'Incalci, Maurizio, Casali, Paolo G., Canevari, Silvana, Pierotti, Marco A., Pilotti, Silvana“…In this model, a switch in the vascular landscape from a normal to a pro-angiogenic signature and the silencing of DLK1-DIO3 region mark the progression from ML to RC in concert with the acquisition by the latter of the over-expression of YY1/C-MYC/HDAC2, together with over-expression of genes involved in cell proliferation and stemness: MKNK2, MSX1 and TRIM71. Taken together, these findings strongly suggest that to progress from ML to RC liposarcoma the cells have to overcome the epigenetic silencing restriction point in order to reset their new stem-like differentiation signature. …”
Publicado 2014
Enlace del recurso
Enlace del recurso
Online Artículo Texto -
280por Trujillo-Paredes, Niurka, Valencia, Concepción, Guerrero-Flores, Gilda, Arzate, Dulce-María, Baizabal, José-Manuel, Guerra-Crespo, Magdalena, Fuentes-Hernández, Ayari, Zea-Armenta, Iván, Covarrubias, Luis“…Early in differentiation, decreasing Notch signalling caused a reduction in NPCs and an increase in dopaminergic neurons in association with dynamic changes in the proportion of sequentially-linked dopaminergic NPCs (Msx1/2+, Ngn2+, Nurr1+). These effects in differentiation caused a significant reduction in the number of dopaminergic neurons produced. …”
Publicado 2016
Enlace del recurso
Enlace del recurso
Enlace del recurso
Online Artículo Texto