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281por Holla, Sahana, Prakhar, Praveen, Singh, Vikas, Karnam, Anupama, Mukherjee, Tanushree, Mahadik, Kasturi, Parikh, Pankti, Singh, Amit, Rajmani, R. S., Ramachandra, Subbaraya G., Balaji, Kithiganahalli Narayanaswamy“…Further, NOTCH1-responsive RNA-binding protein MUSASHI (MSI), targets a transcriptional repressor of JMJD3, Msx2-interacting nuclear target protein, to positively regulate infection-induced JMJD3 expression, FM generation and M2 phenotype. …”
Publicado 2016
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282“…We use a new in vitro model based on the overexpression of Muscle Segment Homeobox 1 (MSX1) to induce Endothelial-to-Mesenchymal Transition (EndMT), a process involved in cardiovascular development and disease. …”
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287por Krabberød, Anders K., Orr, Russell J.S., Bråte, Jon, Kristensen, Tom, Bjørklund, Kjell R., Shalchian-Tabrizi, KamranEnlace del recurso
Publicado 2017
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288“…The loss of Lifr results in both the failure for STAT3 to translocate to the LE nuclei and a reduction in the expression of the LIF regulated gene Msx1 that regulates uterine receptivity. These results reveal that uterine expression of the LIFR is essential for embryo implantation and further define the components of the LIF signaling pathway necessary for effective implantation.…”
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289“…Retinoic acid represses many anteriorly expressed genes, including Bmp4, Lhx9, Msx2, and Alx4. We provide evidence that retinoic acid influences transcription via induction of dHAND and inhibition of Gli3 to establish a new anteroposterior pre‐pattern. …”
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291por Xu, Duo, Pavlidis, Pavlos, Taskent, Recep Ozgur, Alachiotis, Nikolaos, Flanagan, Colin, DeGiorgio, Michael, Blekhman, Ran, Ruhl, Stefan, Gokcumen, OmerEnlace del recurso
Publicado 2017
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292por Duan, Yuange, Dou, Shengqian, Zhang, Hong, Wu, Changcheng, Wu, Mingming, Lu, JianEnlace del recurso
Publicado 2018
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295por Cherlin, Svetlana, Heaps, Sarah E, Nye, Tom M W, Boys, Richard J, Williams, Tom A, Embley, T MartinEnlace del recurso
Publicado 2018
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296“…Predator presence/absence altered selection on dorsal spines and allele frequencies at the Msx2a gene across a generation. Evolutionary trajectories of alleles at a second gene, Pitx1, and the pelvic spine trait it controls, were more variable. …”
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297“…While a number of lncRNAs such as MEG3, AL162231.4 and NONHSAT044111 have been down-regulated in the course of psoriasis, others including PRINS, MIR31HG, RP6‐65G23.1, MSX2P1, SLC6A14-1:1, NR_003062 have been up-regulated. …”
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298“…Furthermore, the hub genes of AD- and epilepsy-associated GCMs were captured by weighted key driver analysis (wKDA), including TRPC1, C2ORF40, NR3C1, KIAA0368, MMT00043109, STEAP1, MSX1, KL, and CLIC6. The shared GCMs and hub genes might provide novel therapeutic targets for AD and epilepsy.…”
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299“…Hub genes from key genes as BMP4, MSX1, HAND2, TBX2, SIX1, IRX1, EDN1, DLX5, MEF2C, and DLX2 were identified. …”
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300por Wu, Xianxian, Zhang, Hongdian, Sui, Zhilin, Gao, Yongyin, Gong, Lei, Chen, Chuangui, Ma, Zhao, Tang, Peng, Yu, Zhentao“…HIF‐1α‐induced ESCC cell migration and invasion were reversed by CXCR4 knockdown or treatment with MSX‐122, a CXCR4 antagonist. Collectively, these data revealed that the HIF‐1α/CXCR4 axis plays key roles in ESCC growth and metastasis and indicated CXCR4 as a potential target for ESCC treatment.…”
Publicado 2022
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