Mostrando 441 - 460 Resultados de 578 Para Buscar '"PRD"', tiempo de consulta: 0.16s Limitar resultados
  1. 441
    por Pope, Gunnar C., Halter, Ryan J.
    Publicado 2019
    “…At a compression ratio (CR) of 23.3×, the root mean square error (RMSErr) is below 0.016 [Formula: see text] S and the percent root-mean-square difference (PRD) is below 1%. Tonic EDA features are preserved at a CR = 23.3× while phasic EDA features are more prone to reconstruction errors at CRs > 8.8×. …”
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  2. 442
  3. 443
    por Zhang, Qingzhan, Ma, Jinyou, Yoo, Dongwan
    Publicado 2017
    “…PEDV blocked the p65 activation in infected cells and suppressed the PRD II-mediated NF-κB activity. Of the total of 22 viral proteins, nine proteins were identified as NF-κB antagonists, and nsp1 was the most potent suppressor of proinflammatory cytokines. …”
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  4. 444
    “…We found that IL-1β plus TNF-α left-shifted the dye uptake rate vs. dye concentration relationship for Etd and 2-NBDG, but the opposite took place for DAPI or YO-PRO-1, whereas no alterations were observed for Prd. The latter modifications were accompanied of changes in K(d) (Etd, DAPI, YO-PRO-1 or 2-NBDG) and Hill coefficients (Etd and YO-PRO-1), but not in alterations of V(max). …”
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  5. 445
  6. 446
    “…The performance of each considered algorithm is evaluated by assessing the Percentage of Root-mean-squared Difference (PRD) and compared with the state of the art techniques. …”
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  7. 447
  8. 448
    “…To monitor the effect of the ventilation on the virus movement, PRD1 bacteriophages as virus simulants were disseminated in a ¾ scale air-conditioned hospital test room with twelve PM2.5 samplers at 15 L/min. …”
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  9. 449
  10. 450
    “…This report reviewed and summarized our studies on the aberrant changes in intracellular components and biological functions in the brains of prion disease (PrD). Omics analyses proposed extensive abnormal alterations of brain mRNAs transcriptions, protein expressions, and post-translational modifications. …”
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  11. 451
  12. 452
  13. 453
    “…DCP5 requires SSF for FLC regulation, and loss of SSF or its PrD disrupts DCP5 function. Our results reveal that DCP5 interacts with SSF, and the nuclear DCP5–SSF complex regulates FLC expression at the transcriptional level.…”
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  14. 454
    “…In addition, Ror2-mediated enhancement of canonical signaling requires the extracellular CRD, but not the intracellular PRD domain of Ror2. We further provide evidence that the positive effect of Ror2 on canonical Wnt signaling is inhibited by Dkk1 and Krm1 suggesting that Ror2 enhances an Lrp-dependent STF response. …”
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  15. 455
    “…SCHULTE produced significant nucleotide BLAST alignments with repeat DNA elements from all four families of plants represented in the TIGR plant repeat database (PRD); the best nucleotide sequence alignment was to ToRTL1 in Lycopersicon esculentum. …”
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  16. 456
    “…Brown adipose cells are specialized to dissipate chemical energy in the form of heat, as a physiological defense against cold and obesity1. PRDM16 (PRD1-BF1-RIZ1 homologous domain containing 16) is a 140 kDa zinc finger protein that robustly induces brown fat determination and differentiation2. …”
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  17. 457
  18. 458
    “…Our results suggest that alterations in calcium homeostasis and increased susceptibility to ER stress are common pathological features of both infectious and familial PrD models.…”
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  19. 459
    “…Here we show that the autoinhibitory, intramolecular interactions in pacsin-1 can be released upon binding to the entire proline-rich domain (PRD) of dynamin-1, resulting in potent membrane deformation activity that is distinct from the isolated F-BAR domain. …”
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  20. 460
    “…Moreover, Ada was not involved in the repair of any O(4)-alkyldT lesions, Ogt was able to repair O(4)-MedT and, to a lesser extent, O(4)-EtdT and O(4)-nPrdT, but not other O(4)-alkyldT lesions. Together, our study provided important new knowledge about the repair of the O(4)-alkyldT lesions and their recognition by the E. coli replication machinery.…”
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