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  1. 41
    “…Importantly, we identified a "hypermethylated cell" state as evidenced by an accumulation of S-adenosyl-homocysteine (SAH) and a further reduction of S-adenosylmethionine (SAM) in the hPheo1 SDHB knock out cells compared to the wild-type cells. …”
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    SUN-LB053 Metabolic Interaction of SAH and SAM in hPheo1 Cells
  2. 42
    “…Subarachnoid hemorrhage (SAH) patients’ surgery is performed to prevent extravasation of blood into the subarachnoid space. …”
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    lncRNA Signature for Predicting Cerebral Vasospasm in Patients with SAH: Implications for Precision Neurosurgery
  3. 43
    “…RESULTS: Of 119 patients with SAH, good and poor outcomes were found in 83 (69.7%) and 36 (30.3%) patients, respectively. …”
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    Early Microcirculatory Hemodynamic Changes Are Correlated With Functional Outcomes at Discharge in Patients With Aneurysmal SAH
  4. 44
    “…Aneurysmal subarachnoid hemorrhage (aSAH), resulting majorly from the rupture of intracranial aneurysms, is a potentially devastating disease with high morbidity and mortality. …”
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    Janus Faced HMGB1 and Post-Aneurysmal Subarachnoid Hemorrhage (aSAH) Inflammation
  5. 45
    “…Post-aSAH sequela is characterized by the onset of life-threatening complications along with the upregulated underlying inflammation. …”
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    Clustering of serum biomarkers involved in post-aneurysmal subarachnoid hemorrhage (aSAH) complications
  6. 46
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    Comparison of surgical or endovascular treated aneurismal SAH patients with a special emphasis on cerebral vasospasm
  7. 47
    “…Delayed cerebral ischemia (DCI) is a feared and significant medical complication following aneurysmal subarachnoid hemorrhage (aSAH). It occurs in about 30% of patients surviving the initial hemorrhage, mostly between days 4 and 10 after aSAH. …”
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    Monitoring in Neurointensive Care – The Challenge to Detect Delayed Cerebral Ischemia in High-Grade Aneurysmal SAH
  8. 48
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    Correlation of hyponatremia, leukocytosis, hypomagnesemia and fever after sah with delayed cerebral ischemia and poor outcome
  9. 49
    “…BACKGROUND: Aneurysmal subarachnoid haemorrhage (aSAH) is an acute cerebrovascular event with high socioeconomic impact as it tends to affect younger patients. …”
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    Effect of weekend admission on in-hospital mortality and functional outcomes for patients with acute subarachnoid haemorrhage (SAH)
  10. 50
    “…BACKGROUND: Up to 15% of all spontaneous subarachnoid hemorrhages (SAH) have a non-aneurysmal SAH (NASAH). The evaluation of SAH patients with negative digital subtraction angiography (DSA) is sometimes a diagnostic challenge. …”
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    MRI-detection rate and incidence of lumbar bleeding sources in 190 patients with non-aneurysmal SAH
  11. 51
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    Correction: Distribution and evolution of stable single α-helices (SAH domains) in myosin motor proteins
  12. 52
    “…OBJECTIVE: Subarachnoid haemorrhage (SAH), caused by the rupture of intracranial aneurysms, is a devastating event with high rates of morbidity and mortality. …”
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    Aneurysmal subarachnoid haemorrhage (aSAH): Five consecutive years' experience of Fars province, Iran
  13. 53
    “…Aneurysmal subarachnoid hemorrhage (aSAH) represents only a small portion of all strokes, but accounts for almost half of the deaths caused by stroke worldwide. …”
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    Role of Damage Associated Molecular Pattern Molecules (DAMPs) in Aneurysmal Subarachnoid Hemorrhage (aSAH)
  14. 54
    “…The purpose of the study was to determine expression levels of serum miR-1297 in patients with aneurysmal subarachnoid hemorrhage (aSAH), and to assess whether miR-1297 was the prognostic indicator of aSAH. …”
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    Early serum miR-1297 is an indicator of poor neurological outcome in patients with aSAH
  15. 55
    “…OBJECTIVE: Subarachnoid haemorrhage (SAH) accounts for 3% of all strokes, and is associated with significant morbidity and mortality. …”
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    Apolipoprotein E mimetic peptide CN-105 improves outcome in a murine model of SAH
  16. 56
    “…Subarachnoid hemorrhage (SAH) is a devastating form of stroke that leads to incurable outcomes. …”
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    Inhibition of Necroptosis Rescues SAH-Induced Synaptic Impairments in Hippocampus via CREB-BDNF Pathway
  17. 57
    “…Apolipoprotein E (APOE) and Factor XIIIA (F13A) genes are involved in the pathogenetic mechanism of aneurysmal subarachnoid haemorrhage (aSAH). We evaluated the association of promoter methylation status of APOE and F13A gene and risk of aSAH. …”
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    Prognostic significance of factor XIIIA promoter methylation status in aneurysmal subarachnoid haemorrhage (aSAH)
  18. 58
    “…We present a case of symptomatic SAH in the third trimester of pregnancy that was successfully managed conservatively.…”
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    A Successful Conservative Management of Spontaneous Adrenal Hemorrhage (SAH) in Pregnancy: A Case Report
  19. 59
    por Kumar, Ajoy
    Publicado 2004
    Libro
    Thermal engineering /
  20. 60
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    Presente, pasado y futuro de la conexión entre el SAHS y el cáncer
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