Mostrando 21 - 40 Resultados de 703 Para Buscar '"Socing"', tiempo de consulta: 0.21s Limitar resultados
  1. 21
    “…Calcium signalling in platelets through store operated Ca(2+) entry (SOCE) or receptor-operated Ca(2+) entry (ROCE) mechanisms is crucial for platelet activation and function. …”
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    Reduction in SOCE and Associated Aggregation in Platelets from Mice with Platelet-Specific Deletion of Orai1
  2. 22
    “…Store-operated Ca(2+) entry (SOCE), the fundamental Ca(2+) signaling mechanism in myogenesis, is mediated by stromal interaction molecule (STIM), which senses the depletion of endoplasmic reticulum Ca(2+) stores and induces Ca(2+) influx by activating Orai channels in the plasma membrane. …”
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    Orai1 inhibitor STIM2β regulates myogenesis by controlling SOCE dependent transcriptional factors
  3. 23
    “…Ca transient kinetics (time to peak, decay time) were not affected by SOCE stimulation. Direct SOCE measurements revealed a negligible magnitude in untreated myocytes but a dramatic increase in SOCE upon Dex-pretreatment. …”
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    Glucocorticoid stimulation increases cardiac contractility by SGK1-dependent SOCE-activation in rat cardiac myocytes
  4. 24
    “…N‐MYLK‐induced SOCE augmented endothelial permeability without MLC‐P via an actin‐binding motif, DVRGLL. …”
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    Noncanonical function of long myosin light chain kinase in increasing ER‐PM junctions and augmentation of SOCE
  5. 25
    “…Therefore, the serosal TRPV4-constituted SOCE mechanism is likely universal for the most common and important secretagogues-induced and Ca(2+)-dependent intestinal anion secretion. …”
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    Role of Serosal TRPV4-Constituted SOCE Mechanism in Secretagogues-Stimulated Intestinal Epithelial Anion Secretion
  6. 26
    “…Store-operated calcium entry (SOCE) is an important process in calcium signaling. …”
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    Purinergic Activation of Store-Operated Calcium Entry (SOCE) Regulates Cell Migration in Metastatic Ovarian Cancer Cells
  7. 27
    “…Store-operated calcium entry (SOCE) channels play an important role in Ca(2+) signaling. …”
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    Accelerated Activation of SOCE Current in Myotubes from Two Mouse Models of Anesthetic- and Heat-Induced Sudden Death
  8. 28
    “…Overall, our study suggests that RASSF4 controls SOCE and ER–PM junctions through ARF6-dependent regulation of PM PI(4,5)P(2) levels, pivotal for a variety of physiological processes.…”
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    RASSF4 controls SOCE and ER–PM junctions through regulation of PI(4,5)P(2)
  9. 29
    “…A short hairpin RNA of the β1 integrin gene was designed and transfected into mouse models of asthma in vivo, in order to evaluate whether silencing of the β1 integrin gene affects airway smooth muscle cell proliferation and inflammation by regulating the mRNA expression of store-operated Ca(2+) entry (SOCE)-associated genes. Silencing the β1 integrin gene may downregulate β1 integrin mRNA while not statistically decreasing α-smooth muscle actin gene expression and airway smooth muscle thickness. β1 integrin silencing was able to downregulate the transcription of SOCE-associated genes to normal levels, including calcium release-activated calcium modulator 1 and short transient receptor potential channel member 1, but not stromal interaction molecule 1, in asthma. …”
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    Silencing of β1 integrin regulates airway remodeling by regulating the transcription of SOCE-associated genes in asthmatic mice
  10. 30
    “…Store-operated Ca(2+) entry (SOCE), a major Ca(2+) signaling mechanism in non-myocyte cells, has recently emerged as a component of Ca(2+) signaling in cardiac myocytes. …”
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    Enhancement of Cardiac Store Operated Calcium Entry (SOCE) within Novel Intercalated Disk Microdomains in Arrhythmic Disease
  11. 31
    “…A reduction in store-operated Ca(2+) entry (SOCE) is shared by numerous FAD-linked mutations, and SOCE is involved in Aβ accumulation in different model cells. …”
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    ORAI2 Down-Regulation Potentiates SOCE and Decreases Aβ42 Accumulation in Human Neuroglioma Cells
  12. 32
    “…SARAF, a negative regulator of store-operated Ca(2+) entry (SOCE), showed the strongest binding activity. Biochemical analysis of Strep-tagged and GFP-fused SARAF proteins revealed ubiquitination that proceeded during pulldown assays under certain buffer conditions. …”
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    The Penta-EF-Hand ALG-2 Protein Interacts with the Cytosolic Domain of the SOCE Regulator SARAF and Interferes with Ubiquitination
  13. 33
    “…Mechanistically, FGF19/FGFR4 signaling stimulated store-operated Ca(2+) entry (SOCE) through both the PLCγ and ERK1/2 pathways. …”
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    FGF19/SOCE/NFATc2 signaling circuit facilitates the self-renewal of liver cancer stem cells
  14. 34
    “…This review provides a brief overview of the molecular mechanisms underlying STIM1/Orai1-dependent SOCE in skeletal muscle, focusing on how SOCE alteration could contribute to skeletal muscle wasting disorders and on how SOCE components could represent pharmacological targets with high therapeutic potential.…”
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    Alteration of STIM1/Orai1-Mediated SOCE in Skeletal Muscle: Impact in Genetic Muscle Diseases and Beyond
  15. 35
    “…Our data indicate that SOCE contributes to the normal physiological contractile response of young healthy skeletal muscle and that defective extracellular Ca(2+) entry through SOCE contributes to the reduced contractile force characteristic of aged skeletal muscle.…”
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    Store-Operated Ca(2+) Entry (SOCE) Contributes to Normal Skeletal Muscle Contractility in young but not in aged skeletal muscle
  16. 36
    “…Store-operated calcium entry (SOCE) is the predominant Ca(2+) entry mechanism in nonexcitable cells and controls a variety of physiological and pathological processes. …”
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    The KSR2-calcineurin complex regulates STIM1-ORAI1 dynamics and store-operated calcium entry (SOCE)
  17. 37
    “…[Image: see text] Store-operated calcium entry (SOCE) is important in the maintenance of calcium homeostasis and alterations in this mechanism are responsible for several pathological conditions, including acute pancreatitis. …”
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    Store-Operated Calcium Entry as a Therapeutic Target in Acute Pancreatitis: Discovery and Development of Drug-Like SOCE Inhibitors
  18. 38
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    Functional communication between IP(3)R and STIM2 at subthreshold stimuli is a critical checkpoint for initiation of SOCE
  19. 39
    “…Downregulation or absence of native Stim1A results in increased SOCE. Despite reducing SOCE, Stim1A leads to increased NFAT translocation. …”
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    A longer isoform of Stim1 is a negative SOCE regulator but increases cAMP‐modulated NFAT signaling
  20. 40
    “…This study investigated malaria hotspots in Keur Soce sites by using geographically-weighted regression. …”
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    Application of geographically-weighted regression analysis to assess risk factors for malaria hotspots in Keur Soce health and demographic surveillance site
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