Mostrando 401 - 420 Resultados de 703 Para Buscar '"Socing"', tiempo de consulta: 0.22s Limitar resultados
  1. 401
    “…We show that organ-restricted RiPI targeting Stromal interaction molecule (Stim), an essential factor of store-operated calcium entry (SOCE), results in progressive fat accumulation in fly adipose tissue. …”
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    Chronic dysfunction of Stromal interaction molecule by pulsed RNAi induction in fat tissue impairs organismal energy homeostasis in Drosophila
  2. 402
    por Zhang, Isis, Hu, Huijuan
    Publicado 2020
    “…Store-operated calcium channels (SOCs) are widely expressed in excitatory and non-excitatory cells where they mediate significant store-operated calcium entry (SOCE), an important pathway for calcium signaling throughout the body. …”
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    Store-Operated Calcium Channels in Physiological and Pathological States of the Nervous System
  3. 403
    “…The store-operated calcium entry (SOCE) channels are assumed to be activated by the calcium sensor stromal interaction molecule (STIM)which leads to activation of its associated Orai channel. …”
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    Calcium Sensors STIM1 and STIM2 Regulate Different Calcium Functions in Cultured Hippocampal Neurons
  4. 404
    “…Stromal interaction molecule 1 (STIM1), the sarcoplasmic reticulum (SR) transmembrane protein, activates store-operated Ca(2+) entry (SOCE) in skeletal muscle and, thereby, coordinates Ca(2+) homeostasis, Ca(2+)-dependent gene expression, and contractility. …”
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    Desmin interacts with STIM1 and coordinates Ca(2+) signaling in skeletal muscle
  5. 405
    “…It indicates some impairment in the SOCE mechanism of immune cells, which is associated with TB.…”
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    Expression of ORAI1 and STIM1 genes in blood of patients with pulmonary tuberculosis
  6. 406
    “…Altogether, this study points toward the hypotheses of the involvement of HTT in EC coupling via its interaction with JPH1, and on SOCE via its interaction with JPH1 and/or STIM1.…”
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    Huntingtin regulates calcium fluxes in skeletal muscle
  7. 407
    “…Additionally, we found that these variants may disturbed stromal-interacting molecule 1 (STIM1)/Orai1-mediated store-operated Ca(2+) entry (SOCE) machinery in the endoplasmic reticulum (ER), in which GLT-1 may be a new partner of SOCE. …”
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    Functional investigation of SLC1A2 variants associated with epilepsy
  8. 408
    “…Specifically, the presence of STIM1 overexpression in COADs, which represented a disturbance of the SOCE pathway, was associated with cell migration and cell motility properties. …”
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    Correlation of clinical features and genetic profiles of stromal interaction molecule 1 (STIM1) in colorectal cancers
  9. 409
    “…The Orai1-mediated store-operated Ca(2+) entry (SOCE) is a ubiquitous signaling mechanism that may become overactivated in pathological states resulting in intracellular Ca(2+) overload. …”
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    Orai1 calcium channel inhibition prevents progression of chronic pancreatitis
  10. 410
    “…Conversely, depletion of ER luminal calcium and subsequent activation of SOCE increases the amount of Sac1 in contact with the PM, depleting PM PI(4)P. …”
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    Dynamic formation of ER–PM junctions presents a lipid phosphatase to regulate phosphoinositides
  11. 411
    “…Store-operated calcium entry (SOCE) plays an important role in the invasion and migration of cancer cells. …”
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    Suppression of STIM1 inhibits the migration and invasion of human prostate cancer cells and is associated with PI3K/Akt signaling inactivation
  12. 412
    “…Overall, we show that MSC-derived DNs are functional with regard to SOCE mechanisms, and MPP(+) exposure dysregulates calcium signaling, making them vulnerable to neurodegeneration. …”
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    MPP(+) decreases store-operated calcium entry and TRPC1 expression in Mesenchymal Stem Cell derived dopaminergic neurons
  13. 413
    “…Here, we show that plasma membrane Ca(2+) ATPase 4 (PMCA4) is differentially expressed in human CD4(+) T compartments yielding distinct store operated Ca(2+) entry (SOCE) profiles. Modulation of PMCA4 yielded a more prominent increase of SOCE in memory than in naïve CD4(+) T cell. …”
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    Plasma Membrane Calcium ATPase Regulates Stoichiometry of CD4(+) T-Cell Compartments
  14. 414
    “…The molecular basis for CRAC channel regulation by the SOCE-associated regulatory factor (SARAF) remained insufficiently understood. …”
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    Bidirectional regulation of calcium release–activated calcium (CRAC) channel by SARAF
  15. 415
    “…STIM1 is an ER/SR transmembrane protein that interacts with ORAI1 to activate store operated Ca(2+) entry (SOCE) upon ER/SR depletion of calcium. Normally highly expressed in skeletal muscle, STIM1 deficiency causes significant changes to mitochondrial ultrastructure that do not occur with loss of ORAI1 or other components of SOCE. …”
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    Proteomics and phosphoproteomics datasets of a muscle-specific STIM1 loss-of-function mouse model
  16. 416
    “…Our results indicated that Orai3 could be one of the therapy targets for PF in which remodels Orai channel, suppresses SOCE activity and activated fibroblast to alleviate fibrosis progress.…”
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    Orai3 mediates Orai channel remodelling to activate fibroblast in pulmonary fibrosis
  17. 417
    “…It is well-known that glial cells play important roles in central sensitization. SOC entry (SOCE) has been implicated in many cell types including cortical astrocytes. …”
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    STIMs and Orai1 regulate cytokine production in spinal astrocytes
  18. 418
    “…•LEFTY2 downregulates the expression of Orai1 and inhibits SOCE. •LEFTY2 and the Orai1 blockers 2-APB, MRS-1845, and YM-58483 inhibit COX2, BMP2, and WNT4 expression in endometrial cells. …”
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    LEFTY2 inhibits endometrial receptivity by downregulating Orai1 expression and store-operated Ca(2+) entry
  19. 419
    “…Uniquely expressed in the colon, MS4A12 exhibits store-operated Ca(2+) entry (SOCE) activity. However, compared to MS4A1 (CD20), a Ca(2+) channel and ideal target for successful leukaemia immunotherapy, MS4A12 has rarely been studied. …”
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    Plasma Membrane Localized GCaMP-MS4A12 by Orai1 Co-Expression Shows Thapsigargin- and Ca(2+)-Dependent Fluorescence Increases
  20. 420
    “…We discovered that HSFs abolished the lipopolysaccharide (LPS)- or thapsigargin (Tg)-induced increase in intracellular Ca(2+) accumulation by blocking the ER store release and SOCE. Moreover, electrophysiological recordings demonstrated HSF-mediated inhibition of LPS- or Tg-induced SOCE currents through both TRPC1 and ORAI1 Ca(2+) channels on plasma membrane. …”
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    Inhibition of store-operated calcium entry in microglia by helminth factors: implications for immune suppression in neurocysticercosis
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