Mostrando 481 - 500 Resultados de 703 Para Buscar '"Socing"', tiempo de consulta: 0.12s Limitar resultados
  1. 481
    “…GDAP1 silencing in vitro reduces Ca(2+) inflow through store-operated Ca(2+) entry (SOCE) upon mobilization of endoplasmic reticulum (ER) Ca(2+), likely in association with an abnormal distribution of the mitochondrial network. …”
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  2. 482
    “…Thus, MG53 could participate in regulating extracellular Ca(2+) entry via Orai1 during SOCE and also intracellular Ca(2+) release via RyR1 during skeletal muscle contraction.…”
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  3. 483
    “…The involvement of inositol trisphosphate receptor (IP3R) in modulating store-operated calcium entry (SOCE) was established many years ago. Nevertheless, the molecular mechanism responsible for this observation has not been elucidated to this date. …”
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  4. 484
    “…Since ER stress, mitochondrial dysfunction and oxidative stress were involved in the effects of resveratrol with imbalance of calcium bio-transmission, store operated calcium entry (SOCE), a novel intracellular calcium regulatory pathway, may also participate in this process. …”
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  5. 485
    “…RESULTS: We observed the inhibition of Stim1 expression, association of Stim1 to Orai1 or Trpc1, and SOCE in Dox-treated mouse myocardium and cardiomyocytes. …”
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  6. 486
    “…The inhibitor of store-operated Ca(2+) entry (SOCE) BTP2 was reported to inhibit ryanodine receptor Ca(2+) leak and electrically evoked Ca(2+) release from the sarcoplasmic reticulum when introduced into mechanically skinned muscle fibers. …”
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  7. 487
    “…Here, we show that both CFTR blockers, at concentrations routinely employed by many researchers, caused a significant inhibition of store-operated calcium entry (SOCE) that was time-dependent, poorly reversible and independent of CFTR. …”
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  8. 488
    “…Here, we present the complete genome sequence of C. activa strain SOCE 004, assembled from the phycosphere of a long-term laboratory-maintained culture of the diatom Skeletonema tropicum. …”
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  9. 489
    “…Store-operated Ca(2+) entry (SOCE) stimulated with thapsigargin was reduced in RyR2(KO) cells compared to controls, but was unchanged in IRBIT(KO) cells. …”
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  10. 490
    “…Human ASM cells express STIM1, STIM2, and Orai1 mRNAs. SOCE activated by thapsigargin, an inhibitor of SR Ca(2+)-ATPase, was significantly blocked by STIM1 siRNA and Orai1 siRNA but not by STIM2 siRNA. …”
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  11. 491
    “…Here, we investigate the downstream signaling pathways of mAR and identify rapid DHT induced activation of store-operated Ca(2+) entry (SOCE) in primary cultures of human prostate epithelial cells (hPEC) from non-tumorous tissue. …”
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  12. 492
    “…Sequestration of Ca(2+) using BAPTA-AM or blockage of store-operated Ca(2+) entry (SOCE) using BTP-2 both attenuated the compensatory activation of MEK/ERK signaling in pevonedistat-treated ALL cells. …”
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  13. 493
    “…It also demonstrated that inhibition of macrophage‐secreted matricellular protein, osteopontin (OPN), led to a significant decrease in cardiomyocyte store‐operated calcium entry (SOCE). In the direct model, stem cell‐derived cardiomyocytes were co‐cultured with polarized macrophage subsets for up to 3 days. …”
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  14. 494
  15. 495
    “…Stromal-interaction molecule 1 (STIM1) triggered store-operated Ca(2+) entry (SOCE) is the major route of Ca(2+) influx for non-excitable cells including hepatocellular carcinoma (HCC) cells. …”
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  16. 496
    “…TEER transient response and extracellular Ca(2+) entry induced by QD deposition were completely inhibited in cells treated with stretched-activated (SA) inhibitor GdCl(3) and store-operated calcium entry (SOCE) inhibitor BTP2 and in cells immersed in Ca(2+)-free medium. …”
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  17. 497
    “…Store-operated Ca(2+) entry (SOCE) via store-operated Ca(2+) channels (SOCC), encoded by transient receptor potential canonical (TRPC) channel proteins, is an important underlying mechanism regulating intracellular Ca(2+) concentration ([Ca(2+)](i)) and various intracellular functions in endothelial cells (ECs). …”
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  18. 498
  19. 499
  20. 500
    “…IP(3)-mediated store-operated calcium entry (SOCE), obtained after store depletion elicited by G(q)-linked purinergic P(2)Y receptors activation, are either sensitized or desensitized, following GSH depletion or increase, respectively. …”
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