Mostrando 61 - 80 Resultados de 703 Para Buscar '"Socing"', tiempo de consulta: 0.18s Limitar resultados
  1. 61
    “…Furthermore, EGCG significantly decreased SOCE in both murine and human T cells. EGCG treatment increased miRNA-15b (miR-15b) abundance in both murine and human T cells. …”
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    Epigallocatechin-3-gallate (EGCG) up-regulates miR-15b expression thus attenuating store operated calcium entry (SOCE) into murine CD4(+) T cells and human leukaemic T cell lymphoblasts
  2. 62
    por Sullins, D. Paul
    Publicado 2022
    “…RESULTS: The SOCE group was statistically indistinguishable from the non-SOCE group on any measure of harm. …”
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    Absence of Behavioral Harm Following Non-efficacious Sexual Orientation Change Efforts: A Retrospective Study of United States Sexual Minority Adults, 2016–2018
  3. 63
    “…One of these is store-operated Ca(2+) entry (SOCE), mediated by an interaction between STIM1 and Orai1. …”
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    Coupling of store-operated calcium entry to vasoconstriction is acid-sensing ion channel 1a dependent in pulmonary but not mesenteric arteries
  4. 64
    por Deb, Bipan K., Hasan, Gaiti
    Publicado 2016
    “…The mechanism of store-operated Ca(2+) entry (SOCE) brings extracellular Ca(2+) into cells after depletion of intracellular Ca(2+) stores. …”
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    Regulation of Store-Operated Ca(2+) Entry by Septins
  5. 65
    por Machaca, Khaled, Haun, Shirley
    Publicado 2002
    “…We have previously shown that SOCE inactivates during Xenopus oocyte meiosis. SOCE inactivation may be important in preventing premature egg activation. …”
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    Induction of maturation-promoting factor during Xenopus oocyte maturation uncouples Ca(2+) store depletion from store-operated Ca(2+) entry
  6. 66
    “…Current knowledge on store-operated Ca(2+) entry (SOCE) regarding its localization, kinetics, and regulation is mostly derived from studies performed in non-excitable cells. …”
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    Store-Operated Calcium Entry in Skeletal Muscle: What Makes It Different?
  7. 67
    “…Transcriptional regulation by Store-operated Calcium Entry (SOCE) is well studied in non-excitable cells. However, the role of SOCE has been poorly documented in neuronal cells with more complicated calcium dynamics. …”
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    A pupal transcriptomic screen identifies Ral as a target of store-operated calcium entry in Drosophila neurons
  8. 68
    “…In many non-excitable and excitable cells, store-operated calcium entry (SOCE) represents an additional pathway for calcium entry upon Ca(2+) store depletion. …”
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    Store-operated calcium entry and the localization of STIM1 and Orai1 proteins in isolated mouse sinoatrial node cells
  9. 69
    “…Both SOCE mechanisms are poorly understood. In particular, pSOCE has not been described in detail because the conditions required for its detection in mouse skeletal muscle have not been established to date. …”
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    Phasic Store-Operated Ca(2+) Entry During Excitation-Contraction Coupling in Skeletal Muscle Fibers From Exercised Mice
  10. 70
    “…Store-operated Ca(2+) entry (SOCE) is an essential Ca(2+) signaling mechanism present in most animal cells. …”
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    Suppression of store-operated calcium entry causes dilated cardiomyopathy of the Drosophila heart
  11. 71
    “…Pharmacological manipulation showed that the increase in SOCE mainly depends on tonic modulation by diacylglycerol kinase, which prevents the protein kinase C-dependent inhibition of SOCE activity. …”
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    Store-Operated Ca(2+) Entry Is Up-Regulated in Tumour-Infiltrating Lymphocytes from Metastatic Colorectal Cancer Patients
  12. 72
    por Rahman, Saifur, Rahman, Taufiq
    Publicado 2017
    “…Abnormal SOCE seem to underlie several diseases that notably include allergy, inflammation and cancer. …”
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    Unveiling some FDA-approved drugs as inhibitors of the store-operated Ca(2+) entry pathway
  13. 73
    “…NED induced by androgen deprivation resulted in a decrease of SOCE due to cortical F-actin over-polymerization which inhibits thapsigargin-induced SOCE. …”
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    Cytoskeleton Reorganization as an Alternative Mechanism of Store-Operated Calcium Entry Control in Neuroendocrine-Differentiated Cells
  14. 74
    “…BACKGROUND: Store-Operated Calcium Entry (SOCE) is the major Ca(2+ )ion entry pathway in lymphocytes and is responsible of a severe combined immunodeficiency (SCID) when deficient. …”
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    The boron-oxygen core of borinate esters is responsible for the store-operated calcium entry potentiation ability
  15. 75
    “…Simultaneous gene-silencing of all three Orai subtypes exhibited a 67.6% reduction of SOCE. Based on the detailed population analysis, we predict that Orai1 and Orai3 are expressed in astrocytes with a large SOCE, whereas TrpC1 is exclusively expressed in astrocytes with a small SOCE. …”
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    Orai1 and Orai3 in Combination with Stim1 Mediate the Majority of Store-operated Calcium Entry in Astrocytes
  16. 76
    “…Here we show that treatment of non-tumoral breast epithelial cells with tunicamycin attenuates SOCE. Meanwhile, tunicamycin was without effect on SOCE in luminal MCF7 and triple negative breast cancer (TNBC) MDA-MB-231 cells. …”
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    Store-Operated Calcium Entry in Breast Cancer Cells Is Insensitive to Orai1 and STIM1 N-Linked Glycosylation
  17. 77
    “…Treatment with a SOCE blocker (2-APB) inhibited the differentiation. …”
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    Stromal interaction molecule 2 regulates C2C12 myoblast differentiation
  18. 78
    “…However, it is currently unknown whether soluble αKlotho regulates Orai1-mediated SOCE via PI3K-dependent signaling. Among the Klotho family, αKlotho downregulates SOCE while βKlotho or γKlotho does not affect SOCE. …”
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    Soluble αKlotho downregulates Orai1-mediated store-operated Ca(2+) entry via PI3K-dependent signaling
  19. 79
    “…The fluctuating SOCE activity during cell cycle progression is universal in different cell types, in which SOCE is upregulated in G1/S transition and downregulated from S to G2/M transition. …”
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    The STIM1-Orai1 pathway of store-operated Ca(2+) entry controls the checkpoint in cell cycle G1/S transition
  20. 80
    “…Disruption of neuronal SOCE also underlies Alzheimer’s disease (AD) pathogenesis, since both in genetic mouse models and in human sporadic AD brain samples, reduced SOCE contributes to synaptic loss and cognitive decline. …”
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    On the Role of Store-Operated Calcium Entry in Acute and Chronic Neurodegenerative Diseases
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