Mostrando 141 - 160 Resultados de 703 Para Buscar '"Socing"', tiempo de consulta: 0.17s Limitar resultados
  1. 141
    “…The 32d-p210 cells showed modified Orai1/STIM1 ratio and reduced TRPC1 expression that could explain SOCE reduction. Decrease in SOCE and thrombin induced calcium entry was associated to reduced Nuclear Factor of Activated T cells (NFAT) nucleus translocation in 32d-p210 cells. …”
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    Deregulation of calcium homeostasis in Bcr-Abl-dependent chronic myeloid leukemia
  2. 142
    “…By contrast, SOCE in MCF10A cells was unaffected by melatonin. …”
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    Melatonin downregulates TRPC6, impairing store-operated calcium entry in triple-negative breast cancer cells
  3. 143
    “…Store-operated Ca(2+) entry (SOCE) regulates a diversity of cellular processes in a variety of cell types. …”
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    Enhanced Orai1-mediated store-operated Ca(2+) channel/calpain signaling contributes to high glucose-induced podocyte injury
  4. 144
    “…The molecular mechanisms responsible for SOCE activation in vascular endothelial cells have engendered a long-lasting controversy. …”
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    The Molecular Heterogeneity of Store-Operated Ca(2+) Entry in Vascular Endothelial Cells: The Different roles of Orai1 and TRPC1/TRPC4 Channels in the Transition from Ca(2+)-Selective to Non-Selective Cation Currents
  5. 145
    “…Moreover, extracellular Ca(2+) influx through the SOCE was significantly reduced in PC-9 cells pre-treated with afatinib than in the control group. …”
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    Afatinib Mediates Autophagic Degradation of ORAI1, STIM1, and SERCA2, Which Inhibits Proliferation of Non–Small Cell Lung Cancer Cells
  6. 146
    “…Conversely, using the same calcium add-back protocol, knockdown of endogenous TRPC3 with shRNA-mediated interference or pharmacological inhibition with the selective TRPC3 antagonist Pyr10 induced a substantial decrease of SOCE, indicating a significant role of TRPC3 in SOCE in DRG nociceptors. …”
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    Contribution of TRPC3 to store-operated calcium entry and inflammatory transductions in primary nociceptors
  7. 147
    “…SOCE was induced following pharmacological depletion of the ER Ca(2+) store, but not by InsP(3)-dependent Ca(2+) release. …”
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    Store-Operated Ca(2+) Entry Does Not Control Proliferation in Primary Cultures of Human Metastatic Renal Cellular Carcinoma
  8. 148
    “…Their paralogs, Stim2, Orai2 and Orai3, support SOCE in heterologous expression systems, but their physiological role is still obscure. …”
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    Stim and Orai proteins in neuronal Ca(2+) signaling and excitability
  9. 149
    “…Interaction between the endoplasmic reticulum (ER)-located stromal interaction molecue1 (STIM1) and the plasma membrane-located Ca(2+) channel subunit, Orai1, underlies store-operated Ca(2+) entry (SOCE). Calsequestrin1 (CSQ1), a sarcoplasmic reticulum Ca(2+) buffering protein, inhibits SOCE, but the mechanism of action is unknown. …”
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    Retrograde regulation of STIM1-Orai1 interaction and store-operated Ca(2+) entry by calsequestrin
  10. 150
    “…Furthermore, the TGF-β2 effect on FGF23 production was blunted by SOCE inhibitor 2-APB. We conclude that TGF-β2 induces FGF23 production, an effect involving up-regulation of SOCE.…”
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    The production of fibroblast growth factor 23 is controlled by TGF-β2
  11. 151
    “…Transmembrane adenylyl cyclase inhibition had no effect on the SOCE-induced rise in cAMP, while sAC inhibition abolished SOCE-generated cAMP and significantly reduced SOCE-induced VASP and CREB phosphorylation. …”
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    Soluble adenylyl cyclase links Ca(2+) entry to Ca(2+)/cAMP-response element binding protein (CREB) activation in vascular smooth muscle
  12. 152
    “…Moreover, knockdown of Orai1, an essential pore-subunit of SOCE channels, decreased HMGB1-induced endothelial hyperpermeability. …”
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    Store-Operated Ca(2+) Entry Plays a Role in HMGB1-Induced Vascular Endothelial Cell Hyperpermeability
  13. 153
    “…Blocking nSOCE by its inhibitor SKF96365 enhanced NMDA-driven [Ca(2+)](i). …”
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    STIM Protein-NMDA2 Receptor Interaction Decreases NMDA-Dependent Calcium Levels in Cortical Neurons
  14. 154
    “…Both the store-operated Ca(2+) entry (SOCE) and excessive autophagy are the major causative factors for pathological cardiac hypertrophy. …”
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    Ang II Promotes Cardiac Autophagy and Hypertrophy via Orai1/STIM1
  15. 155
    “…The results show that SOCE in these cells is enhanced upon lentiviral expression of the Htt138Q-1exon. …”
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    Both Orai1 and TRPC1 are Involved in Excessive Store-Operated Calcium Entry in Striatal Neurons Expressing Mutant Huntingtin Exon 1
  16. 156
    “…Therefore, persistent activation of SOCE represents a novel pathological mechanism in HBV-mediated hepatocarcinogenesis.…”
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    Hepatitis B virus PreS2-mutant large surface antigen activates store-operated calcium entry and promotes chromosome instability
  17. 157
    “…Reduced SOCE in cells with CMT recessive missense mutations in the α-loop of GDAP1, but not dominant mutations, was associated with smaller SOCE-stimulated respiration. …”
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    CMT-linked loss-of-function mutations in GDAP1 impair store-operated Ca(2+) entry-stimulated respiration
  18. 158
    “…Moreover, SOCE was largely diminished in cells where STIM1 and/or ORAI1 had been silenced by small interfering (si)RNA. …”
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    Extracellular ATP activates store-operated Ca(2+) entry in white adipocytes: functional evidence for STIM1 and ORAI1
  19. 159
    “…Third, we investigated the role of SOCE and STIM1 in cell migration using wound healing assays and Boyden chamber assays. …”
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    STIM1 expression is associated with osteosarcoma cell survival
  20. 160
    “…As expected, SOCE was decreased in STIM1/2- deficient pmLF and ROCE was decreased in TRPC1/6(−/−) pmLF compared to control cells. …”
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    Store-operated Ca(2+) entry in primary murine lung fibroblasts is independent of classical transient receptor potential (TRPC) channels and contributes to cell migration
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