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  1. 81
    “…DNA polymerases of the Y-family, such as Escherichia coli UmuC and DinB, are specialized enzymes induced by the SOS response, which bypass lesions allowing the continuation of DNA replication. umuDC orthologs are absent in Caulobacter crescentus and other bacteria, raising the question about the existence of SOS mutagenesis in these organisms. …”
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    An SOS-regulated operon involved in damage-inducible mutagenesis in Caulobacter crescentus
  2. 82
    “…A mechanistic understanding of how Sos-1 coordinates Ras and Rac activity is, however, still missing. …”
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    Mechanisms through which Sos-1 coordinates the activation of Ras and Rac
  3. 83
    por Janion, Celina
    Publicado 2008
    “…Under normal growth conditions the SOS genes are expressed at a basal level, which increases distinctly upon induction of the SOS response. …”
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    Inducible SOS Response System of DNA Repair and Mutagenesis in Escherichia coli
  4. 84
    “…We show that some of the Rec proteins implicated previously promote TLD via facilitating activation of the SOS response and that, of the roughly 40 proteins upregulated by SOS, SulA, an SOS–inducible inhibitor of cell division, accounts for most or all of how SOS causes TLD. …”
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    Role of RecA and the SOS Response in Thymineless Death in Escherichia coli
  5. 85
    “…The eubacterial SOS system is a paradigm of cellular DNA damage and repair, and its activation can contribute to antibiotic resistance.1,2,3 Under normal conditions, LexA represses the transcription of many DNA repair proteins by binding to SOS “boxes” in their operators. …”
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    Structure of the LexA-DNA complex and implications for SOS box measurement
  6. 86
    “…In this study, we cloned and identified three salt overly sensitive 2 (SOS2) genes in the woody plant Populus trichocarpa, designated as PtSOS2.1, PtSOS2.2, and PtSOS2.3, which were transformed into hybrid poplar clone T89 (Populus tremula× Populus tremuloides Michx clone T89) mediated by Agrobacterium tumefaciens. …”
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    Overexpression of PtSOS2 Enhances Salt Tolerance in Transgenic Poplars
  7. 87
    “…The Escherichia coli SOS system is a well-established model for the cellular response to DNA damage. …”
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    Suppression of the E. coli SOS response by dNTP pool changes
  8. 88
    “…Cytological analyses of mucilage indicates that heteromannans are associated with cellulose, and not in the pathway involving SOS5 or FEI2. A SOS5 fluorescent protein fusion (SOS5-mCITRINE) was localized throughout the mucilage pocket, consistent with a structural role in pectin adhesion. …”
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    Dissecting Seed Mucilage Adherence Mediated by FEI2 and SOS5
  9. 89
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    The small 6C RNA of Corynebacterium glutamicum is involved in the SOS response
  10. 90
    “…SOS is a key activator of the small GTPase Ras. …”
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    One-way membrane trafficking of SOS in receptor-triggered Ras activation
  11. 91
    “…Neither Ethylene-Insensitive 2 (EIN2) nor EIN3 changed the expression patterns of SOS genes including SOS1, SOS2, SOS3 and SOS3-like Calcium Binding Protein 8 (SCaBP8), but SOS2 activated the expression of one target gene of EIN3, Ethylene and Salt-inducible ERF 1 (ESE1). …”
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    EIN3 and SOS2 synergistically modulate plant salt tolerance
  12. 92
    “…In Escherichia coli , this inducible system is termed the SOS response. The SOS global regulatory network consists of multiple factors promoting the integrity of DNA as well as error‐prone factors allowing for survival and continuous replication upon extensive DNA damage at the cost of elevated mutagenesis. …”
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    The SOS system: A complex and tightly regulated response to DNA damage
  13. 93
    “…However, SOS1 mutations are rare in human malignancies and patients with germline SOS1 mutations may not be at increased risk of developing cancer. …”
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    TBIO-27. RASOPATHIES AND BRAIN TUMOROGENESIS: ARE SOS1 MUTATIONS ARE CONCERNED?
  14. 94
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    SOS to the Soccer World. Each Time the Preseason Games Are Less Friendly
  15. 95
    “…The guanine nucleotide exchange factor Son of Sevenless (Sos) is the main Ras activator, but the role of the ubiquitously expressed Sos1 in the development of fibrosis has not been studied. …”
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    Sos1 Modulates Extracellular Matrix Synthesis, Proliferation, and Migration in Fibroblasts
  16. 96
    “…Lesions to DNA compromise chromosome integrity, posing a direct threat to cell survival. The bacterial SOS response is a widespread transcriptional regulatory mechanism to address DNA damage. …”
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    Non-canonical LexA proteins regulate the SOS response in the Bacteroidetes
  17. 97
    “…In Escherichia coli, sub-minimum inhibitory concentrations (sub-MICs) of the widely used fluoroquinolones are known to induce the SOS response. Interestingly, the expression of several PMQR qnr genes is controlled by the SOS master regulator, LexA. …”
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    A qnr-plasmid allows aminoglycosides to induce SOS in Escherichia coli
  18. 98
    por White, Paul
    Publicado 2015
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    The SOS guide to live sound: optimizing your band's live-performance audio
  19. 99
    por SOS Meetings
    Publicado 1987
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    Transparencies of SOS Meetings on 29th April and 7th July 1987
  20. 100
    “…After implementation of the SOS-SAH, the number of symptoms discussed during consultation did not increase. …”
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    The SOS-SAH questionnaire in clinical practice: a multi-method evaluation study
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