Mostrando 441 - 460 Resultados de 816 Para Buscar '"The Normal Heart"', tiempo de consulta: 1.76s Limitar resultados
  1. 441
    “…BACKGROUND/AIMS: Brain natriuretic peptide (BNP) levels are known to be elevated in patients with chronic kidney disease (CKD) and normal heart function. Therefore, the present study was performed to examine the effectiveness of BNP level in diagnosing heart failure in patients with CKD and to determine its effects on survival rate and prognosis. …”
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    Relationship between serum brain natriuretic peptide and heart function in patients with chronic kidney disease
  2. 442
    “…BACKGROUND: The fibroblast growth factor (FGF) family is essential to normal heart development. Yet, its contribution to cardiomyocyte differentiation from stem cells has not been systemically studied. …”
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    Fibroblast Growth Factor-10 Promotes Cardiomyocyte Differentiation from Embryonic and Induced Pluripotent Stem Cells
  3. 443
    “…The expression of 417 genes was barely detectable in the normal heart but was suddenly activated in PAH. Among them, Foxm1 and Plk1 are of particular interest, since Ingenuity Pathway Analysis (IPA) using DEGs and upstream motif analysis showed that they are essential hub proteins that regulate the expression of downstream proteins associated with PAH. …”
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    Deep RNA Sequencing Reveals Novel Cardiac Transcriptomic Signatures for Physiological and Pathological Hypertrophy
  4. 444
    “…To understand fully the mechanisms underpinning the regulation of RyR2 in the normal heart and how these systems are altered in pathological conditions, we must first gain a detailed knowledge of the fundamental processes of RyR2 gating. …”
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    A mechanistic description of gating of the human cardiac ryanodine receptor in a regulated minimal environment
  5. 445
    “…RESULTS: GLUT4, GLUT1, and GLUT8 were found to be the most abundant GLUT transcripts in the normal heart, while GLUT3, GLUT10, and GLUT12 are present at relatively lower levels. …”
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    GLUT4, GLUT1, and GLUT8 are the dominant GLUT transcripts expressed in the murine left ventricle
  6. 446
    “…CONCLUSIONS: Diclofenac at therapeutic concentrations and even at high dose does not prolong repolarization markedly and does not increase the risk of arrhythmia in normal heart. However, high dose diclofenac treatment may lengthen repolarization and enhance proarrhythmic risk in hearts with reduced repolarization reserve.…”
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    Diclofenac Prolongs Repolarization in Ventricular Muscle with Impaired Repolarization Reserve
  7. 447
    “…This tissue regeneration translated into long-term normal heart functions as assessed by echocardiography. …”
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    Complete cardiac regeneration in a mouse model of myocardial infarction
  8. 448
    “…The identification of molecular signals that cause or modify cardiomyopathies is necessary to understand how the normal heart progresses to cardiac hypertrophy and heart failure. …”
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    Raf-mediated cardiac hypertrophy in adult Drosophila
  9. 449
    “…These data suggest that ilk/mys function is necessary for establishing and maintaining normal heart structure and function, and appropriate fine-tuning of this pathway can retard the age-dependent decline in cardiac performance and extend lifespan. …”
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    A dual role for integrin-linked kinase and β1-integrin in modulating cardiac aging
  10. 450
    “…The levels of miRNAs and expressed genes were analysed in myocardial biopsy samples from patients with end-stage heart failure (n = 14) and those from normal heart samples (n = 8). Four networks were built including the Gene regulatory network, Signal-Network, miRNA-GO-Network and miRNA-Gene-Network. …”
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    Identification of micro-RNA networks in end-stage heart failure because of dilated cardiomyopathy
  11. 451
    “…During cardiogenesis the epicardium, covering the surface of the myocardial tube, has been ascribed several functions essential for normal heart development of vertebrates from lampreys to mammals. …”
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    Evolution and Development of Ventricular Septation in the Amniote Heart
  12. 452
    “…Analysis of a range of markers of polarised epithelial tissues showed that in the normal heart, undifferentiated SHF cells move from the dorsal pericardial wall into the distal outflow tract where they acquire an epithelial phenotype, before moving proximally where they differentiate into cardiomyocytes. …”
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    Vangl2-Regulated Polarisation of Second Heart Field-Derived Cells Is Required for Outflow Tract Lengthening during Cardiac Development
  13. 453
    “…Relative carbohydrate and fat oxidation levels were measured in both normal and ischemic hearts using a 1-(13)C glucose clamp coupled with NMR-based isotopomer analysis, as well as in adult rat CMs by monitoring pH and O(2) consumption in the presence of glucose or palmitate. In normal heart, GLP-1 increased glucose uptake (↑64%, p<0.05) without affecting glycogen levels. …”
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    Cardioprotection Resulting from Glucagon-Like Peptide-1 Administration Involves Shifting Metabolic Substrate Utilization to Increase Energy Efficiency in the Rat Heart
  14. 454
    “…CONCLUSIONS: During normal heart development, spatio-temporal differences in contribution of WT-1 and Tcf21-(LacZ) + cells to right versus left ventricular myocardium occur parallel to myocardial thickening. …”
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    Regional differences in WT-1 and Tcf21 expression during ventricular development: implications for myocardial compaction
  15. 455
    “…METHODS: Ten patients with an MRI-conditional pacemaker system and a structurally normal heart were studied. Patient-specific 3D thorax volume models were reconstructed from the MRI images. …”
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    Non-invasive focus localization, right ventricular epicardial potential mapping in patients with an MRI-conditional pacemaker system ‐ a pilot study
  16. 456
    “…Although the three therapies reduced myocarditis and fibrosis and ameliorated electrical alterations, only Bz plus PTX restored normal heart rate-corrected QT (QTc) intervals. Bz-plus-PTX-treated mice presented complementary effects of Bz and PTX, which reduced TNF expression (37%) in heart tissue and restored normal TNF receptor 1 expression on CD8(+) T cells, respectively. …”
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    Combination Chemotherapy with Suboptimal Doses of Benznidazole and Pentoxifylline Sustains Partial Reversion of Experimental Chagas' Heart Disease
  17. 457
    “…General physical examination revealed a healthy parturient with normal vital signs, clear lungs and normal heart sounds. Obstetric examination revealed a fundal height compatible with the gestational age; there was a single living fetus in breech presentation; she was not in labor. …”
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    Fusion of lower limbs with severe urogenital malformation in a newborn, a rare congenital clinical syndrome: case report
  18. 458
    “…Research outcomes revealed PTZ-induced dose-dependent inhibition on A2780 cancer cells (IC(50) =0.62 µM), with significant less cytotoxicity toward HEK293 normal kidney cells and H9C2 normal heart cells. Generation of reactive oxygen species (ROS) and polarization of mitochondrial membrane potential (ΔΨm) suggests PTZ-induced cell death through oxidative damage. …”
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    10H-3,6-Diazaphenothiazine induces G(2)/M phase cell cycle arrest and caspase-dependent apoptosis and inhibits cell invasion of A2780 ovarian carcinoma cells through the regulation of NF-κB and (BIRC6-XIAP) complexes
  19. 459
    “…The suspected chronic heart failure outpatients were divided into heart failure with reduced ejection fraction, heart failure with mid-range ejection fraction, heart failure with preserved ejection fraction, and normal heart function groups. Area under the receiver-operating characteristic (ROC) curve, cut-off value, and logistic regression analysis were used to select the model variables, sensitivity and specificity. …”
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    NT-proBNP test with improved accuracy for the diagnosis of chronic heart failure
  20. 460
    “…Therefore, we quantitatively analyzed various hemodynamic parameters related to the RV under normal, heart failure (HF), and HF incorporated with continuous flow LVAD therapy by using a computational model. …”
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    The effect of heart failure and left ventricular assist device treatment on right ventricular mechanics: a computational study
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