Mostrando 761 - 780 Resultados de 816 Para Buscar '"The Normal Heart"', tiempo de consulta: 0.29s Limitar resultados
  1. 761
    “…RESULTS: Major complication occurred in 4.4% of all procedures and significantly more often in patients with structural heart disease than in structurally normal hearts (6.0 vs. 1.8%). The frequency of these periprocedural complications was significantly different between procedures with sole right ventricular and a combination of RV and LV access (0.5 vs. 3.1%). …”
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    Complications and mortality after catheter ablation of ventricular arrhythmias: risk in VT ablation (RIVA) score
  2. 762
    “…Computer simulations and animal studies in normal hearts showed that restoring of AV-coupling with unchanged ventricular activation sequence significantly increased LV filling, mean arterial pressure, and cardiac output by 10–15%. …”
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    Pacing therapy for atrioventricular dromotropathy: a combined computational–experimental–clinical study
  3. 763
    “…HIV-positive patients with an echocardiographically confirmed diagnosis of dilated cardiomyopathy and HIV-positive controls with echocardiographically normal hearts were studied. Patients with secondary causes of cardiomyopathy (such as hypertension, diabetes, pregnancy, alcoholism, valvular heart disease, and opportunistic infection) were excluded from the study. …”
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    The mitochondrial DNA T16189C polymorphism and HIV-associated cardiomyopathy: a genotype-phenotype association study
  4. 764
    “…Fontan patients had aberrant flow patterns as compared to normal hearts in terms of position, shape and sphericity of the main vortices. …”
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    Visualization of the intracavitary blood flow in systemic ventricles of Fontan patients by contrast echocardiography using particle image velocimetry
  5. 765
    “…. • Differentiation of LVNC from other cardiomyopathies and normal hearts is possible. • The best diagnostic performance can be achieved if combined MRI criteria for the diagnosis are used.…”
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    Value of cardiovascular MR in diagnosing left ventricular non-compaction cardiomyopathy and in discriminating between other cardiomyopathies
  6. 766
    “…Results show that LV/RV APD and APD adaptation heterogeneities promote unidirectional block following rate acceleration, albeit being insufficient for establishment of reentry in normal hearts. However, in the presence of an ischemic region at the LV/RV junction, LV/RV heterogeneity in APD and APD rate adaptation promotes reentrant activity and its degeneration into fibrillatory activity. …”
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    In Vivo Human Left-to-Right Ventricular Differences in Rate Adaptation Transiently Increase Pro-Arrhythmic Risk following Rate Acceleration
  7. 767
    “…Although studies show alternans in normal hearts, it occurs only in the OVVR model, and only for a narrow range of cycle lengths. …”
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    A Quantitative Comparison of the Behavior of Human Ventricular Cardiac Electrophysiology Models in Tissue
  8. 768
    “…The Brugada syndrome (BrS) is a malignant, genetically-determined, arrhythmic syndrome manifesting as syncope or sudden cardiac death (SCD) in individuals with structurally normal hearts. The diagnosis of the BrS is mainly based on the presence of a spontaneous or Na + channel blocker induced characteristic, electrocardiographic (ECG) pattern (type 1 or coved Brugada ECG pattern) typically seen in leads V1 and V2 recorded from the 4th to 2nd intercostal (i.c.) spaces. …”
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    Electrocardiographic methods for diagnosis and risk stratification in the Brugada syndrome
  9. 769
    “…To further investigate the regulation of those factors, we performed single cell analysis to dissect the gene expression profile of MSCs at single cell level in infarcted and normal hearts, respectively. Consistent with the in vivo observation, a similar regulation pattern of those factors was detected in cultured MSCs under hypoxia. …”
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    Paracrine Action of Mesenchymal Stem Cells Revealed by Single Cell Gene Profiling in Infarcted Murine Hearts
  10. 770
    “…METHODS: Patients with normal hearts and supraventricular tachycardia underwent electrophysiological studies avoiding sedation. …”
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    Effect of mental stress on dynamic electrophysiological properties of the endocardium and epicardium in humans
  11. 771
    “…ABSTRACT: Catecholaminergic polymorphic ventricular tachycardia (CPVT) is an inherited arrhythmia syndrome characterized by fatal ventricular arrhythmias in structurally normal hearts during β‐adrenergic stimulation. Current treatment strategies include β‐blockade, flecainide and ICD implementation – none of which is fully effective and each comes with associated risk. …”
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    In silico prediction of drug therapy in catecholaminergic polymorphic ventricular tachycardia
  12. 772
    “…NEW & NOTEWORTHY In ventricular myocytes from normal hearts, regulation of the L-type Ca(2+) current by β(2)-adrenoceptors and the constitutive regulation by caveolin-3 is localized to the t-tubules. …”
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    Loss of caveolin-3-dependent regulation of I(Ca) in rat ventricular myocytes in heart failure
  13. 773
    “…CONCLUSION: Quantitative SPECT/CT can measure HDP myocardial uptake in individuals with normal hearts and those with cardiac ATTR without recourse to comparison with bone. …”
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    Tc-HDP quantitative SPECT/CT in transthyretin cardiac amyloid and the development of a reference interval for myocardial uptake in the non-affected population
  14. 774
    “…It should be kept in mind that children with structurally normal hearts may present with an earlier concealed phase and can be diagnosed with ARVD.…”
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    Clinical features and arrhythmic complications of patients with pediatric-onset arrhythmogenic right ventricular dysplasia
  15. 775
    “…CONCLUSION: T3 intracoronary perfusion did not alter cardiodynamics and hemodynamics nor influence the activation of Akt and ERK of normal hearts. Accordingly, the rapid non-genomic effects of T3 were not evident. …”
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    Left ventricular phosphorylation patterns of Akt and ERK1/2 after triiodothyronine intracoronary perfusion in isolated hearts and short-term in vivo treatment in Wistar rats
  16. 776
    “…HFD increased body weight, heart weight, total cholesterol, and triglyceride levels, whereas Q normalized heart weight and triglycerides. LV ejection fraction was lower in HFD vs. control mice (56.20 ± 15.8% vs. 73.38 ± 5.04%, respectively, P < 0.05), but improved in HFD + Q mice (67.42 ± 7.50%, P < 0.05, vs. …”
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    Quercetin Reverses Cardiac Systolic Dysfunction in Mice Fed with a High-Fat Diet: Role of Angiogenesis
  17. 777
    “…Cardiac muscle myosin regulatory light chain (RLC) is constitutively phosphorylated at ∼0.4 mol phosphate/mol RLC in normal hearts, and phosphorylation is maintained by balanced activities of dedicated cardiac muscle–specific myosin light chain kinase and myosin light chain phosphatase (MLCP). …”
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    Myosin light chain phosphatase catalytic subunit dephosphorylates cardiac myosin via mechanisms dependent and independent of the MYPT regulatory subunits
  18. 778
    “…Because LV function in patients with type 2 diabetes mellitus (T2DM) may be subtly altered long before the onset of ALVDD, quantitative assessment of the risk of progression to early-stage DCM in T2DM patients with normal hearts is critical for delaying or even reversing DCM. …”
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    Nomogram based on multimodal echocardiography for assessing the evolution of diabetic cardiomyopathy in diabetic patients with normal cardiac function
  19. 779
    “…The Thoracic Inlet Heart Size (TIHS) normalizes heart size to body size using the thoracic inlet length. …”
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    The Thoracic Inlet Heart Size, a New Approach to Radiographic Cardiac Measurement
  20. 780
    “…A systematic comparison between detailed vs smooth anatomies, male vs female normal hearts was carried out. The heart with a myocardial infarction was subjected to a programmed stimulus protocol to identify the effects of sex and anatomical detail on ventricular tachycardia inducibility. …”
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    Ventricular anatomical complexity and sex differences impact predictions from electrophysiological computational models
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